Quiz 7 Flashcards
transmissible gastroenteritis
Most deadly and most feared of baby pig diarrhea
Lose most pigs farrowed over 2-3 week period
Etiology: coronavirus- appears like petals under microscope
Infection occurs via ingestion
Virus easily destroyed by detergents and inactivated by warm attempts, drying and sunlight
Virus resistant to acid and survives well in cold, dark environments, survives indefinitely when frozen
Has affinity for S.I cells, virus multiples and cells explode resulting in severe villus atrophy which decreases absorption and digestion
Tge clinical signs
Newborn pigs:
-severe, diarrhea starts 18-30 hours after exposure. Born healthy and see signs the next day. Immediate diarrhea is not GE
-first clinical sign is always vomiting followed by thin and watery diarrhea (yellowish puddles). Pigs become thirsty and dehydrated, they drink water
-highly sensitive to chilling and are difficult to save <2 weeks of age
> 3 weeks
-mild disease, vomiting, fluids diarrhea 1-3 days
Sows
-fever, vomiting, anorexia
TGE diagnosis
History, histopathology, fluoresecent Ab test
TGE prevention
Vaccines not effective, use natural planned infection
Sanitation concerns are extremely important
Often seen in winter and spread via swine, man, starlings
TGE treatment
Fluids (H20, electrolytes)
Increase temperature > 90 F
Planned infection
Swine coccidiosis
Contributes to 10-15% of all baby pig scours
8/9 species are genus eimeria
Microscopic, species specific, single called Protozoa that invades intestinal lining
Infects pigs 7-10 days of age- yellow to light green fluid feces
Pigs scour for 4-10 days and severity depends on degree of infection
Severe infection results in dehydration and mortality around 30-70%
Swine coccidiosis diffential
E. Coli (happens earlier on)
TGE (vomiting)
Rotavirus (3 week old diarrhea, white)
Swine coccidiosis diagnosis
Discovery of oocysts in feces, necropsy/ histopathology and history
Swine coccidiosis treatment
No federally licensed or approved for swine, several sulfa drugs that have been of value, prevention more effective then treatment
Swine coccidiosis prevention
Good sanitation- oocysts are shed in feces
Clean sows prior to entering farrowing
All in all out farrowing
Steam cleaning and 5-10% solution of soapy ammonia will remove and decrease numbers of infective oocysts
Rotavirus diarrhea
Very resistant to decreased pH and disinfectants enabling survival for long periods of time under normal conditions
Affects pigs 1-6 weeks old
“Milk scours”/ white scours/ 3 week old scour
More pigs get this diseases at 3 weeks due to decrease in maternal ab and dilution of Ab due to pigs ingesting creep feed and water
Sows not sick with this disease
Early weaned pigs more susceptible to severe infection
Initial or reaper infectious disrupt intestinal lining, 3-7 days post weaning. Concurrent E. Coli infection increases mortality
Continuous farrowing, buildup or organisms, results in infection of younger animals
Rotavirus diarrhea clinical signs
Initially liquid white stool, then turns creamy/ pasty
Undigested milk in the intestine, milk curd is in stomach
Transient diarrhea usually self correcting, mild disease characterized by short term diarrhea
Increased mortality when simultaneous infectious with E.Coli, TGE, clostridia, coccidiosis, decrease intake of immune milk or stresses such as chilling
Rotavirus diarrhea diagnosis
Lab, submit intestinal sections
Rotavirus diarrhea prevention
Optimal management conditions
Not blood Ab levels but continual milk Ab present
Toxoplasmosis
Etiology: toxoplasma gondii- Protozoa parasite
ZOONOTIC
Caused by consumption of undercooked pork
Cats are definitive hosts for parasite
Infection during pregnancy results in infection of fetus
Easily killed by cooking, freezing, or irradiation
Toxoplasmosis clinical signs
Most signs are minor and disease is sub clinical
Pigs born sick/ dead or become sick 3 hours after birth
#1 sign is labored breathing, fever, general weakness, diarrhea, nervous signs, and occasionally loss of vision
Mummified or stillborn pigs also result
Toxoplasmosis diagnosis
Serum antibody test; dead pig necropsy and histopathology
Toxoplasmosis prevention
Keep cats out of swine barns, feed and water
Remove dead pigs to prevent cannibalism
Rodents should be controlled
Never feed uncooked garbage to pigs
Salmonella choleraesuis
Gram negative bacteria that lives with or without oxygen
S. Choleraesuis causes 70-90% of salmonella cases in pigs
S. Typhimurium causes 10-30% of other cases
ZOONOTIC
Live vaccine on the way, MLV effectively utilized
Salmonella choleraesuis outbreak causes
Poor sanitation
Commingling
Overcrowding
Transportation
Inclement weather
Nutritional changes
Parasitism
Aflatoxins in feed
Concurrent disease
Salmonella choleraesuis clinical signs
Acute septicemia, diarrhea, death without for warning signs. Anorexic, lethargic, shallow cough, Pyrexia, and neurologic involvement
Salmonella choleraesuis diagnosis
Lab isolation and identification of organism
Salmonella choleraesuis treatment
Proper systemic antibiotics, remove and isolate all sick pigs, thorough cleaning of pens, water bowls, etc. Decrease stress, ample and clean water supply, dry clean and well-ventilated pens
Swine arthritis
Major factor in swine lameness
Disease results from injection of joints
Leading cause of loss at slaughter
Low mortality, slower and less efficient gains, lowers adult performance
Etiology: streptococcus suits- acute or chronic
“Joint ill” found in baby pigs with navel infection
Rough flooring/ bedding material causes abrasions of legs and increased invasion by organisms
Morbidity