PSYC 276 UNIT 3 Study Guide Flashcards

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1
Q

Why is the idea that we only use “10% of our brain” a neuromyth?

A

We use our entire brain. There is no evidence for unused capacity in the brain, though we do not use all of the brain at the same time (e.g., stoplight)

Extra info:
if you think about development, any neurons that are not used die off. That said, we do not only use all of the brain at the same time (like a stoplight, only some portions are “on” at a time).

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2
Q

circuit of brain regions active when we are mentally inactive

A

Default Mode Network

Extra: This is NOT active when we are intentionally doing something or thinking.

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3
Q

surgery that physically removes on hemipshere (primarily the cortex)

A

hemispherectomy

Extra: Some tissue is still left intact. If young enough, brain plasticity is better able to rewire itself to perform functions of removed hemisphere.

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4
Q

new connections are made between active neurons to create alternate neural pathways

different part of brain takes over task that damaged part usually does

A

Rerouting

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5
Q

new axon and dendrite extensions “sprout” and allow existing neurons to form new connections.

A

Sprouting

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6
Q

When would collateral sprouting be considered good, neutral, and negative?

A

good = replaces broken pathways
neutral = might not do anything
negative = collateral path may result in chronic pain

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7
Q

What are two ways the brain rewires itself after injury?

A

sprouting and rerouting

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8
Q

What therapies support the process of the brain rewiring itself after damage?

A

Physical and cognitive therapies (e.g., video games, exercise, language therapy)

Medication (drugs, esp. stimulants, may accelerate activity in healthy brain regions after stroke)

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9
Q

sharp blow to the head that does not puncture the brain

most common brain injury in young adults

A

closed head TBI

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10
Q

Why are closed head brain injuries difficult to treat?

A

(Coup/contre-coup) Bruising occurs at the site of impact and site of reverberation (where brain hits other side of skull)

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11
Q

temporary loss of blood flow of the brain

A

stroke

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12
Q

most common type of stroke resulting from blood clot or obstruction of an artery

nutrient supply is stopped and cannot get to areas of the brain

A

ischemic stroke

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13
Q

less common type of stroke resulting from ruptured artery

neurons at site of rupture are flooded with excess blood, calcium, oxygen, and other chemicals AND neurons at the end of the blood’s intended pathway are deprived of necesary nutrients

A

hemorrhagic stroke

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14
Q

Two effects of a stroke

A

edema and disruption of NA/K pumps

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15
Q

fluid accumulation, increased pressure on brain, can cause cell death

A

edema

effect of stroke

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16
Q

accumulation of NA and K ions cause hypopolarization (reduced polarization, inside of cell becomes less negatively charged and leads to more action potentials) leading to NA build up and cell death

A

Disruption of NA/K pumps

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17
Q

List the main symptoms of stroke.

A

BEFAST
- Balance (loss of balance)
- Eyes (loss of vision)
- Face (facial drooping)
- Arm (weakness in arm)
- Speech (difficulty with speech, slurred)
- Time (Call 911 ASAP)

18
Q

3 treatments for stroke

A
  • Cooling the brain to minimize damage – theoretically protects brain after ischemia by reducing overstimulation, apoptosis, and inflammation; not a ton of evidence for this but it doesn’t cause harm
  • Tissue plasminogen activator (tPA) – given within 3 hours (in some cases up to 4.5 hours) to break up blood clots
  • Stent – for prevention, done very rarely in high risk people
19
Q

subjective mental state

A

emotion (scientific definition)

20
Q

Behaviors associated with certain emotions have a(n) ———- basis

A

evolutionary

21
Q

Main purposes of emotion

A
  • aid in quick decision making
  • help communicate needs to others (and understand others’ needs)
22
Q

Theory of Emotion

We experience an emotion in response to a stimulus, then our body responds. (body’s reaction is determined by emotion)

A

Folk Psychology

23
Q

We experience a physiological pattern of arousal in response to a stimulus BEFORE experiencing the emotion. (emotion is interpreted based on our body’s reaction). This is similar to the facial feedback hypothesis.

A

James-Lange Theory

24
Q

In response to a stimulus, we experience a particular emotion and general pattern of autonomic arousal simultaneously, BUT these processes happen independently.

A

Cannon-Bard Theory

25
Q

We use context to cognitively attribute specific emotions to arousal. (the experience of emotion is still attributed to physical arousal, but context of the situation decides which emotion we experience. NEED TO REVISIT VIDEO

A

Schachter-Singer Theory

26
Q

Why might botox injections impact emotion?

A

Facial feedback hypothesis;
- By paralyzing facial muscles, we have reduced facial expression. This has been reported to cause some people to experience emotions less intensely.
- This is consistent with the James-Lange Theory or facial feedback hypothesis, which implies that sensory feedback from facial expressions can affect mood.
- Physiological and psychological effects can be bidirectional.

27
Q

When extreme facial expressions were photoshopped onto a body getting piercing versus body winning tennis match, participants’ interpretation of which emotion the photographed person was experiencing depended on:

A

the context (body positioning, situation)

28
Q

When multiple brain areas contribute to different patterns of behavior

A

degeneracy

29
Q

the ability to recognize facial expressions even when blind

(person can look at a picture and even though they cannot see it, they can gell what emotion the person is experiencing)

A

affective blindsight

(person is unable to tell WHO is in the picture still)

30
Q

How is degeneracy different than redundancy?

A

Degeneracy – different factors and different mechanisms lead to the same outcome (way that system does it is different)
Redundancy – different factors lead to ONE mechanism to the same outcome

31
Q

surgical disconneciton of the prefrontal cortex

A

prefrontal lobotomy

32
Q

Why were prefrontal lobotomies done?

A

Initially used in treating schizophrenia and depression with self-harm to minimize impulsivity, aggressive behavior, etc. Later was used on people with less severe mental illnesses.

33
Q

What changes were caused by prefrontal lobotomies?

A
  • While it removed undesirable symptoms, it often also removed emotion entirely (flat affect).
  • Apathy
  • Lack of ability to plan
  • Memory disorders
  • Lack of emotional expression
34
Q

behavior intent on causing pain or harm to others

A

aggression

35
Q

Based on studies in monkeys and humans, we know that aggressive behavior is linked to —————-, but are unsure of the direction of this effect.

A

low serotonin turnover

36
Q

Post-castration, monkeys show a ——— in aggression. What does this suggest?

A

decrease; suggests that androgens play an important role in aggression

37
Q

When monkeys were administered testosterone, —————– increased. Further, ——————— until the subjections stopped receiving testosterone.

A

aggression; aggression behaviors continued

38
Q

In humans, what does increased testosterone at puberty case and what does this suggest?

A

Puberty and the accompanying increase in testosterone does not affect aggression, indicating that other factors are at play in the testosterone/aggression relationship.

39
Q

For people who scored low on ———– traits, testosterone had no affect on aggression.

A

dominance

40
Q

People who ———– and ————–, were significantly more likely to be aggressive.

A

scored high on dominance traits; had high testosterone

41
Q
A