Physiology Of Training Flashcards

1
Q

Overload

A

Increased capacity of a system in response to training above the level to which is accustomed (intensity, duration and frequency)

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2
Q

Reversibility

A

When training is stopped, the training effect is lost quickly

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3
Q

Training effect is specific to (3)

A
  1. Muscle fiber recruited during exercise
  2. Type of contraction (eccentric, concentric, iso)
  3. Energy system involved (aerobic v.s. Anaerobic)
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4
Q

Who is training improvement always greater for

A

Individuals with lower initial fitness

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5
Q

Which genotype shows higher untrained vo2 max and which shows lower

A

Genotype E shows higher and genotype A shows lower untrained VO2 Mac and exhibit limited exercise training response

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6
Q

Why is anaerobic capacity more genetically determined than aerobic capacity

A

Training can only improve anaerobic performance to a certain degree. Depends on fast IIx fibers that are determined early in development

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7
Q

What changes happen to the muscle from repeated excitation and contraction of muscle fibers in endurance training

A

Changes to their structure and function

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8
Q

What 4 changes to muscle fibers occur during endurance training to structure and function

A
  1. Muscle fiber type
  2. Capillary density
  3. Mitochondrial function
  4. Myoglobin content
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9
Q

How does endurance training change muscle fiber type

A

Reduction in amount of fast fibers and increase in slow fibers

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10
Q

How does capillary density change in endurance training

A

Increased number of capillaries surrounding muscle fibers allowing for enhanced diffusion of oxygen and removal of wastes

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11
Q

How does endurance training change muscle myoglobin content?

A

Increases content by 75-80% increasing capacity for oxidative metabolism in the muscle after training

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12
Q

How does muscle mitochondria change from endurance changing

A

It adapts quickly to training and can double within 5 weeks of training

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13
Q

What type of mitochondria do we have in our muscles

A

Subsarcolemmal and intermyofibrillar mitochondria

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14
Q

Why is improving mitochondrial capacity important

A

It improves oxidative capacity and ability to utilize fat as fuel; improves turnover and breakdown of damaged mitochondrial to replace with healthier mitochondria.

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15
Q

Significance of increased mitochondrial volume

A

-greater capacity for oxidative phosphorylation
-increased volume will decrease ADP leading to less lactate and H+ formation and less PC depletion

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16
Q

What is the result in faster rise in oxygen uptake (from training)?

A

Less lactate formation and less PC depletion

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17
Q

What is the important exercise-induced secondary signaling molecule?

A

PGC-1alpha

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18
Q

How does low muscle glycogen benefit endurance training induced adaptations?

A

Promotes increased protein synthesis and mitochondrial formation due to higher activation of PGC-1alpha

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19
Q

What are the two approaches to achieving low muscle glycogen?

A
  1. Restrict dietary carbohydrates
  2. Train twice per day every other day
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20
Q

What is the effect of increasing capillary density?

A

Slower blood flow in the muscle and increased FFA transporters

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21
Q

Why are increased FFA transporters important

A

Increase the uptake of FFA and FFA utilization to spare plasma glucose

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22
Q

Why is increasing the quantity of mitochondria important

A

Increases beta oxidation enzymes to increase FFA utilization and spare plasma glucose

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23
Q

What is the benefits of increasing FFA oxidation from increasing mitochondria

A

Decreases pyruvate formation to decrease Lactate and H+ formation

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24
Q

Benefits of delaying lactate accumulation in the blood

A

One can exercise at a higher percentage of their VO2 max before the lactate will accumulate

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25
What exercise intensity will show the greatest improvement in VO2 max
80-100% VO2 max and Intensity over duration
26
Based on Ficks equation what adaptation must change in order to increase VO2 max
Adaptations in SV max and a-vO2 difference. HR max cannot increase
27
How does endurance training change total blood volume?
Increases plasma volume Increase volume of red blood cells Decrease hematocrit
28
What 2 factors increase a-vO2 difference
1. Increase in muscle blood flow 2. Improve ability of the muscle to extract oxygen from the blood
29
How do you increase muscle blood flow
Decrease SNS vasoconstriction
30
How do you improve ability of the muscle to extract oxygen from the blood
By increasing capillary density and # of mitochondrial
31
Which training adaptation is a potential mechanism that would explain and increase in VO2 max
Increasing stroke volume through increasing end diastolic volume
32
Why does respiratory system function not limit performance?
Ventilation can be increased to a much greater extent than cardiovascular function
33
What adaptations can you make to the respiratory system during endurance training to maximize efficiency
Blood volume expansion; modulation to the skeletal muscles; neural adaptations
34
What happens to pulmonary ventilation at rest
no effect on lung structure or function
35
How does ventilation act during submaximal exercise following training
Ventilation is lower
36
What is training effect on maximal pulmonary ventilation
It substantially increases
37
What causes VO2 max to dip in first 12 days of not training
Decrease in SV max
38
What decreases VO2 max after 12 days of not training
Decrease in mitochondria with no change in capillary density
39
Why does stroke volume go down first?
It is typically the easiest to regulate
40
How many weeks does it change of retraining to regain mitochondrial adaptations?
3-4 weeks and a months to regain
41
What are the responses of cardiovascular, pulmonary and sympathetic nervous systems dependent on?
The trained state of the muscle involved in the activity. More so than specific adaptation in those systems
42
Can you transfer training to another muscle that is untrained in endurance training
No but it will take less time for that muscle to build because of adaptations to the blood; you can transfer a portion of the training in strength training
43
Why is the transfer effect present in weight training
Neuromodulation. Strengthening the neuro signal in the muscle; recruit more motor units
44
What are the two phases responsible for muscular adaptations to resistance training?
Neural modulation followed by hypertrophy
45
Hyperplasia
Increase in muscle fiber muscle (unclear if it happens in humans)
46
Hypertrophy
Increase in the cross-sectional area of muscle due to increase in muscle proteins (actin and myosin)
47
What is hyperplasia caused from
Natural mutation in myostatin gene
48
What is the predominant muscle fiber in resistance training?
Fast to slow shift in fiber type from type IIx to type IIa (5-11% change)
49
Why would muscle fiber types shift from type IIx to IIa during strength training
Oxidative capacity; supplying more ATP to muscles; multiple efforts
50
What is the signaling effect leading to resistance training induced hypertrophy?
mTOR protein activation which are very involved in protein synthesis
51
How does resistance training-induced hypertrophy effect myonuclei?
It results in parallel increases in muscle fiber cross-sectional area and increased number of myonuclei
52
What is the genetic influence on magnitude of resistance training-induced hypertrophy?
Responsible for 80% of the differences in muscle mass. Hormones are also a large factor
53
How does atrophy happen?
Inactivity causes atrophy due to decreased protein synthesis and increased protein breakdown
54
Training cessation effects
Muscle atrophy and loss of strength; strength training takes longer than endurance;
55
How long do re-training effects take?
6 weeks
56
What 3 factors impact concurrent changes in strength and endurance changing
1. Neural factors 2. Overtraining 3. Depressed protein synthesis
57
How do neural factors limit strength gains in congruent training?
Impaired motor unit recruitment
58
How does depressed protein synthesis limit strength gains in congruent training?
Endurance training cell signaling can interfere with protein synthesis
59
Why does Endurance training cell signaling interfere with protein synthesis
Inhibition of mTOR by activation of AMPK
60
What is the 10 % rule
Increase intensity or duration < 10 % per week