Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

NBDE Part II > ORAL PATH REVIEW POWERPOINT 4 > Flashcards

ORAL PATH REVIEW POWERPOINT 4 Flashcards

1
Q

Cemento-osseus dysplasias are typically found in what age range and gender?

A

middle-aged females

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What are the three types of Cemento-Osseus Dysplasias?

A
  1. Periapical COD 2. Focal COD3. Florid COD
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Name that lesion: middle aged, AA female. Mand anterior teeth. No tx necessary, RL, RO and Mixed

A

Periapical Cemento-osseus dysplasia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Name that lesion: middle aged, AA female. in ONE area and NOT the anterior mandible. No tx necessary, RL, RO and Mixed

A

Focal COD

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Name that lesion: middle aged, AA female. in MULTIPLE QUADRANTS. No tx necessary, RL, RO and Mixed

A

Florid COD

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is the etiology of Fibrous Dysplasia?

A

genetics

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Do you see swelling in fibrous dysplasia? Is it unilateral or bilateral?

A

Unilateral swelling present

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Is fibrous dysplasia painful? What age demographic does it affect? What age does it STOP growing?

A

Painless, EARLY onset that ends at age 20

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is the cardinal sign of Fibrous dysplasia on a radiograph? Are the borders well defined or poorly defined?

A

GROUND GLASS appearance (kids dont PLAY (DIS-PLAY) on ground glass). ill defined borders

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is the treatment for fibrous dysplasia?

A

wait till it stops (age 20) and cosmetically remodel the bone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is the condition where there is abnormal resorption and deposition of bone, weakened or deformed bone and happens in males over 40yrs (vs fibrous dysplasia in young)

A

Paget’s Disease of Bone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Name that condition: enlargment of jaws/ridges, dentures wont fit, spaces between teeth, generalized hypercementosis and COTTON WOOL RO areas

A

Paget’s Disease of Bone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Name that lesion: COTTON WOOL radiopaque areas

A

Paget’s Disease of Bone (old men make COTTON WOOL dresses for the beauty PAGET)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Paget’s Disease of Bone has an INCREASED RISK of what condition?

A

OSTEOSARCOMA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

WTF is this?? Rapid onset, increase in VANILL-YL-MAN-DELIC ACID (VMA), anterior maxilla, radiolucent and destructive

A

Melanotic NERURO-ECTODERMAL Tumor of Infancy (WTF?)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

In a Melanotic NeuroEctodermal Tumor of Infancy (long ass name, I Know), what molecule is overproduced?

A

VANILL-YL-MAN-DELIC ACID (VMA)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Name that condition: defect in OSTEOCLASTS leading to INCREASED DENSITY of bone (skull and jaws) and osteomyelitis?

A

Ostropetrosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Osteopetrosis can cause what in older folks and what in younger folks?

A

older: cranial nerve compression; younger: bone fractures

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

What are the two Central Neural lesions and what two types of structures can they affect?

A
  1. Neurofibroma and 2. Schwannoma…they can ENLARGE 1. canals and 2. foramina
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

What RL lesion can look like anything (multilocular, unilocular, in any location) and is extremely important to ASPIRATE before biopsy?

A

Hemangioma (aspirate any RL lesion before biopsy to rule out vascular lesions)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

What is the most common primary bone cancer?

A

osteosarcoma

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

What is the most common cancer in bone?

A

Metastasis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Since you already know metastasis is the most common cancer found in bone, what are the 6 most common cancers to metastasize to the bone?

A

1.breast 2.lung 3.thryoid 4. kidney 5.prostate 6.colorectal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

What is associated with numb chin syndrome?

A

malignant bone tumor growth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

What age range is most commonly affected by Osteosarc?

A

20’s-30’s

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

Name that condition: age 20’s-30’s, swelling of bone, loose/displaced teeth, PARESTHESIA

A

Osteosarc

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

What condition is associated with “SUN RAY” appearance on an x-ray?

A

Osteosarcoma

28
Q

Name that lesion: poorly defined margins, sun ray appearance, irregular widening of PDL in early stages

A

Osteosarcoma

29
Q

What is the oral maifestation of Cleidocranial Dysplasia?

A

multiple, unerupted supernumerary teeth

30
Q

What is the oral manifestation of ECTODERMAL DYSPLASIA?

A
  1. Hypodontia 2.Conical/Cone shaped teeth (that midwestern guy was freaking spot on!)
31
Q

The _______ type of Ectodermal Dysplasia is a partial or complete absecnce of eccrine sweat glands and hair follicles leading to fine, sparse hair and decreased heat tolerance

A

A-NY-DRO-TIC type

32
Q

WTF? Name that condition: oral mucosa neuromas (hamatomas), medullary carcinoma of the thyroid gland, and pheochromocytoma of the adrenal gland

A

MULTIPLE ENDOCRINE NEOPLASIA SYNDROME (hey! the name makes sense!) TYPE IIB

33
Q

Name that condition (2 names PLEZ): Multiple neurofibromas, CAFE AU LAIT pigmentations (also seen in FIBROUS DYSPLASIA), axillary freckling and LISCH nodules in the eye

A

Neurofibromatosis TYPE I, von Ricklinghausen’s disease of skin)

34
Q

NAME THAT CONDITION: numerous melanotic macules intraorally and periorally. Intestinal polyps that do NOT become malignant, but at high risk for GI cancer…

A

PEUTZ-JEGHER Syndrome

35
Q

Name that condition: Dental anomalies or OSTEOMAS (may block the eruption of teeth), polyps of the colon that become MALIGNANT…Epirdermoid cysts on the skin

A

Gardner syndrome

36
Q

Name that condition (2 names please):: Deformed ears/hearing loss, Coloboma (fissure of lower eyelid), CONVEX facial profile (downward sloping palpebral fissures, HYPOPLASIA of the mandible and malar bones, “bird face”

A

Treacher Collins Syndrome (MandibuloFacial DysOstosis)

37
Q

Name that condition (2 names please):: multiple basal cell carcinomas, multiple OKCs, calcified falx cerebri, rib anomalies, palmar plantar pits, ocular hypertelorism, enarged head, spina bifida

A

GORLIN syndrome (Nevoid basal cell carcinoma syndrome)

38
Q

What condition am I??? Trauma or periapical inflammatory disease of primary tooth leads to enamel defect of developing permanent teeth

A

TURNER TOOTH

39
Q

Name that condition: too much cementum, well-defined PDL space; looks like turkey drum

A

Hypercementosis

40
Q

GENERALIZED HYPERCEMENTOSIS can be a sign of _______

A

pagets disease

41
Q

What am I??? large mass of cementum with a radiolucent rim around it; mass appears fused to the root with root resorption

A

cementoblastoma

42
Q

What situation is this?? shortened root fused with bone; PDL space lost; occlusal surface apical to other teeth

A

ankylosis

43
Q

What is this describing? Cusp-like elevation of enamel

Most commonly on MANDIBULAR PREMOLARS, May lead to occlusal interference

A

Dens EVAGENATUS

44
Q

What is this describing? Most commonly in permanent maxillary LATERAL INCISORS; RARE on mandibular or primary teeth Various degrees of severity

A

Dens INVAGINATUS

45
Q

physiologic wear due to tooth to tooth contact

A

attrition

46
Q

pathologic wear due to mechanical action of external agent, ie, habits, occupations

A

abrasion

47
Q

pathologic wear due to chemicals not involving bacteria (ie, caries)…wtf? Chlorinated pools

A

Erosion

48
Q

Huh, new term eh?! erosion from gastric regurgitation and GERD

A

Peri-Mo-lysis

49
Q

loss of tooth structure from occlusal stresses that create repeated tooth flexure with failure of enamel/dentin

A

abfraction

50
Q

The 3 sizes (types) of Amelogenesis Imperfecta are small medium and what?!

A
  1. hypoplastic 2.hypocalcified 3.hypomature
51
Q

“picket fence teeth” are indicative of what condition

A

Amelogenesis Imperfecta

52
Q

What am I talkin’ bout? Translucence of teeth: blue to brown Bulbous crowns, cervical constriction
Thin, tapered roots; OBLITERATION of root canals and pulp chamber

A

Dentinogenetis Imperfecta

53
Q

AI vs DI: little to no enamel

A

Amelogenesis Imperfecta

54
Q

AI vs DI: picket fence appearance

A

AI

55
Q

AI vs DI: cervical constriction

A

DI

56
Q

AI vs DI: pulpal obliteration

A

DI

57
Q

AI vs DI: “golf ball on tee” lol

A

DI

58
Q

Genetic disease that leads to defective collagen

A

Osteogenesis imperfecta (w opalescent teeth)

59
Q

What other condition looks like dentogenesis imperfecta clinically?

A

osteogenesis imperfecta

60
Q

What condition? Bones are brittle/fragile: multiple fractures, bowing of legs, Blue sclera, hearing loss
Opalescent teeth: diffuse blue-brown discoloration

A

Osteogenesis Imperfecta

61
Q

Rootless teeth
Pulpal obliteration
Tooth mobility
Rarefying osteitis without cause

A

Dentin Dysplasia

62
Q

Enlarged thistle-tube-shaped pulp chambers

A

dentin dysplasia

63
Q

2 names: Autoimmune disease, Hardening of the skin, Trismus/Limited jaw opening, Generalized widening of PDL space

A

Progressive Systemic Sclerosis (Scleroderma)

64
Q

What is the mechanism for H&N radiation therapy causing caries? Does pulp necrosis usually occur?

A

radiation causes xerostomia which causes the caries..NO pulp necrosis

65
Q

Whats your differential for DIFFUSE, Ill-defined RL areas? (5 please)

A
  1. Osteomyelitis 2.Radiation induced osteonecrosis 3.MRONJ 4.metastatic disease 5.osteosarc
66
Q

In CONDYLAR HYPERPLASIA, the CHIN will deviate to which side?

A

AWAY from affected side upon closure

67
Q

“Tennis racket” / “honeycomb” appearance on a radiograph =

A

Odontogenic Myxoma

NBDE Part II (14 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions