objective 2.2 (pt.2) (3) Flashcards

1
Q

when the heart is unable to pump blood in sufficient amounts from the ventricles to meet the bodys metabolic needs

A

HF

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2
Q

what are the causes of HF?

A

A heart defect
A defect outside the heart

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3
Q

what does myocardial deficient cause?

A

Inadequate contractility caused by an MI, coronary artery disease, cardiomyopathy, or valvular insufficiency
Inadequate filling caused by atrial filribillation (AF), infection, tamponade, or ischemia

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4
Q

Pulmonary edema
Coughing
SOB
Dyspnea

A

left sided HF

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5
Q

Systemic venous congestion
Pedal edema
Jugular veins distention
Ascites
Hepatic congestion

A

right sided HF

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6
Q

what are drug therapies for HF?

A

Positive inotropic drugs:
Positive chronotropic drugs:
Positive dromotropic drugs:

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7
Q

increase the force of myocardial contraction

A

positive inotropic drugs

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8
Q

increase HR

A

positive chronotropic drugs

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9
Q

accelerate cardiac conduction

A

positive dromotropic drugs

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10
Q

prevent sodium and wtr resorption by inhibiting aldolsterone secretion
Diuresis results, which decreases preload and the work of the heart
Lisinopril is used in HTN, HF, and acute MI causes hyperkalemia, cry cough and decreased renal function

A

ACE inhibitors

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11
Q

are potent vasodilators and decrease systemic vascular resistance
Used alone or incombo with other drugs such as diuretics in the treatment of HTN or HF

A

ARBs

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12
Q

similar AE as lisinopril, not as likely to cause the cough like ACE inhibitors, not as likely to cause hyperkalemia

A

valsartan

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13
Q

prevent catecholamine-mediated actions on the heart by reducing or blocking sympathetic nervous system stimulation to the heart and the hearts condition system

A

cardioprotective quality of B blockers

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14
Q

Blocks aldosterone therefore lessens retention of sodium and water, edema, and HF symptoms
Useful in severe stages of HF

A

aldosterone antagonists

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15
Q

potassium -sparing diuretic (keep potassium in the body) but helps body excrete excess fluid

A

spironolactone

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16
Q

Digoxin is the prototype- very narrow therapeutic window
Used in HF and to control ventricular response to atrial fibrillation
Improved symptom control, quality of life, and exercise tolerance, with no apparent reduction in mortality- so no longer a first line treatment
Increase in myocardial contractility
Decrease rate of electrical conduction- decreases HR

A

cardiac glycosides

17
Q

Very narrow therapeutic window; drug levels must be monitored
Low potassium levels increase its toxicity; electrolyte levels must be monitored
AE: dysrhythmias, including bradycardia or tachycardia, headaches, fatigue, malaise, confusion, convulsions, colored vision, halo vision, anorexia, nausea, vomiting, diarrhea

A

digoxin