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2 Weeks > NEW AFTER 1 WEEK-14 > Flashcards

NEW AFTER 1 WEEK-14 Flashcards

1
Q

DUAL RULE RRNA

A

CATALYTIC AND STRUCTURAL

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2
Q

STRCTURES OF TRNA

A

ACCEPTOR ARM,D ARM, TPSI ARM,V LOOP AND ANTICODON

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3
Q

TPSI ARM CONTAIN ?

A

RICH IN THYMINE AND PSEUDOURACIL

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4
Q

ARM D CONTAIN ?

A

DIHYDROOURADINE

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5
Q

rRNA PROCESSING PRO

A

1- RNASE 3 CLEAVE COLMPLEMENTERY AROUND 1 AND 23
2- RNASE E : 5S REMOVE

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6
Q

rRNA PROCESSING EUK

A

1- 45S TO 41 S REMOVE 5 PRIME
2- 41S SPLIT TO 20S AND 32 S
3-3 PRIME OF THE 20 S REMOVE FROM 18S
SEPERATE THE 28 ADN 5.8

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7
Q

AMINOACYL TRNA SYNHTHESASE

A

TRANSFER AA TO TRNA

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8
Q

ISOACCEPTOR

A

DIFFERNT ANTOCODON SAME AA
SAMEE maniacyl trns synthesase

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9
Q

nucleotidyl traSNFERASE wnyzme vs aminoacyl trna synthesase

A

1- transfer cca to the 3 prime of the trna in eyk
2- transfer aa to trna

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10
Q

ALPHA SUBUNIT OF THE RNA POL

A

CTD BIND TO DISTAL ELEMENTS
NTD PUT THE COMPONENTS OF THE RNA POL THOGHTER

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11
Q

RNASE

A

REMOVE 5S FROM PRECURSORE rRNA

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12
Q

RNASEP

A

REMOVE THE 5 PRIME IN THE TRNA IN BOTH EUKARYOTES AND PROKARYOTES

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13
Q

….S IS PRE-rRNA IN EUK

A

45

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14
Q

C/D BOX SNORNA

A

SMALL NUCLEOLAR
METHYLATION

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15
Q

H/ACA BOX SNORNA

A

SMALL NUCLEO
PSEUDOURIDILATION

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16
Q

WHAT WE ADD IN THE CAPPING PROCESS

A

WE ADD M7G FORMING THE 5 PRIME 5 PRIME TRIPHOSPHATE BOND

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17
Q

ADIIDTION OF THE CAP ENZYMES

A

1- RNASE TRIPHOASPHATASE : GMAMMPHOPHATE THE 5 PRIME

2- GUANYLYL TRANSEFERASE : ADD GMP

3- METHYLTRANSFERASE : ADD METHYL FROMT THE S ADENOSYL METHONINE TO 7 OF TH N IN G

4- ANOTHER : ADD METHL TO 2 PRIME OH OF THE NUCLEOTIDE OF THE TRANSCRIPT

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18
Q

WHEN OCCUR THE CAPPING

A

ERALT TRANSCRIPTION
20-40 NT

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19
Q

WHEN POLY A OCCUR

A

POLY A RICH SIGNAL SEQUENCE POLY ADNYLATIONSIGNAL
CPSF + CSTF + CF1 +CF2+ POLY A POLYMERASE

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20
Q

2 COMPLEX IN THE POLY A

A

CPSF AND CSTF

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21
Q

CPSF

A

CLEAVAGE AND POLY A SPECIFICITY FACTOR
BIND TO AAUAAA POL A SIGNAL

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22
Q

CSTF

A

CLEAVAGE STIMIULATION FACTOR
BIND TO GU OR U RICH SITE INCREASE EFFCIENCY OF THE POLY A

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23
Q

SHORT / LONGTHENEING THE POLY A

A

RNASE , CYTOSOLIC POLY A

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24
Q

Bloom syndrome

A

Helicase BLM in a mutated form is the cause of the
Bloom syndrome (recessive inheritance)
Tipically, in patient’s cells there is an excess of
homologous recombination between sister chromatids

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25
Q

CONJUGATION

A

SEX BACTERIA CROSS OVER YES RECOMBINATION YES

26
Q

TRASNFORMATION

A

FROM THE MEDIUM
RECOMBINATION YES

27
Q

TRASNDUCTION

A

VIRUS
RECOMBIANTION YES

28
Q

intergenic suppression IN EU

A

second mutation is in a
different geneoften due to gene interactions)

29
Q

ntergenic suppression IN PRO

A

change results in a tRNA anticodon capable of pairing with
the stop codon

30
Q

Mutation frequency

A

proportion of individuals in a population t

31
Q

Mutation rate

A

probability that a particular mutation will o

32
Q

(STRs

A

short
tandem repeats,

33
Q

RFLP

A

restriction
fragment length
polymorphism

34
Q

mutation models

A

inifinte allel= each mutation 1 allel
infinite site allel= each mutation just 1 site no chance for back mutation or recurrecnt mutation
stepwise mutation = add / delete the same rate by 1 unit
indel=insretion./deeletion

finit sit mutation = mtation same site diffent times
cpg

35
Q

controle theinteraction and associaton of the ribosome

A

nitiiaion factors

36
Q

formyl trna block… formation

A

peptie bond

37
Q

MOST COMPACT DISTRIBUTION OF THE EXON

A

VERTEBRATESS

38
Q

REPLICATOR

A

ORIC/ORGIN OF THE EPLICATION

39
Q

COTROL ELEMNETS IN THE REPLICATION

A

REPLICATOR/ORIGIN \ND TERMINATION SITE

40
Q

EXTRA CHROMOSOMAL REPLICON

A

ORGANELLAR
PLASMID
PHAE/VIRAL DNA

41
Q

REPRESSOR OF THE LAC OPERON

A

TETRAMER
BIND TO 2 SIDE FORM THE LOOP
BIND TO THE OPERON AS DIMER
N TERMINAL BIND TO DNS AND THE ALLOLACTOSE
C TERMINAL BIND TO ALPHA HELIX
WHEN THE INDUCER BIND WE CAN SEE ALLOSTERIC TRANSITION

42
Q

INDUCER /COREPRESSOR IN LAC OPERON

A

ALLOLACTOSE - INDUCER

43
Q

LAC A CODE FOR ?

A

TRANSACETYLASE (ACETYL TO BETA GALACTOSIDASE 0

44
Q

beggining and donwstream of introns

A

UPSTREAM/ 5 PRIME /BEGINNING: GU
DOSNTREM/3 PRIME/THE END : AG

45
Q

RLFP

A

RESTRICTION FRAGMENT LENGTH POLYMORPHISM

46
Q

LONGEVITY

A

e BALANCE between
the advantage of having an appropriate immune
response (↑ inflammation) and the disadvantage of
chronic inflammation during ageing.

47
Q

Resilience

A

s at the core of life and is interpreted as the harmonic
assemblage of the biochemical processes that are aimed at
maintaining the identity, integrity, and autonomy of individual
organisms against the perturbations induced by both internal and
external environments.

48
Q

apoe

A

related to anatgonstic pleitropy
fertility high but the alzihmer high as well

49
Q

brca

A

feterility high but risk of breast cancer

50
Q

AOUNT OF ENERGY BY ATP HYDROLYSATION

A

8 KCAL/MOL

51
Q

PUROMYCIN

A

mimic aminoacyl trna

52
Q

mimic aminoacyl trna

A

PUROMYCIN

53
Q

each release factor in euk corrospond to what release factor in bacteria

A

erf1 = rf1 and rf2
erf3=rf3

54
Q

kirromycin

A

not release of the EF-TU

55
Q

CHLORAMPHENICOL

A

INHIBITOR OF PEPTIDE BOND FORMATION - PROK

56
Q

CYCLOEXIMIDE

A

INHIBITOR OF PEPTIDE FORMATION
EUK

57
Q

FUSIDIC ACID

A

RELEASE OF EF-G AND GDP

58
Q

RIBOSWITCH TPP

A

THUAMINE MONOPHOSPHATE VIT B1 SYNTHESIS

59
Q

RELA PR

A

STRINGENCY FACTOR

60
Q

TERMINATION IN THE RNA POL 1

A

TTF1 BIND AND BLOCK TRNASCIPTION

61
Q

STRINGENT FACTOR

A

REL A

62
Q

ppGpp
PPPGPP

A

GUANOSINE TETRAPHOPHATE
GUONOSINE PENTAPHOPSHPHATE

2 Weeks (10 decks)

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