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OBGYN microbiology > name that skin disease > Flashcards

name that skin disease Flashcards

1
Q

90+% seropositive rates that vary with socioeconomic status

A

HSV-1 (oral herpes)

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2
Q

dermatophytes that are NOT fluorescent

A

trichophyton

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3
Q

these organisms do not live on humans, they prefer hairier and warmer environments like pets

A

flea

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4
Q

a risk factor for this type of necrotizing fasciitis is crush wound

A

type 2 monomicrobic GAS necrotizing fasciitis

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5
Q

fever, malaise, HA, neuralgia, pruritic lesions

A

VZV- chickenpox

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6
Q

infestations common in houses with pets

A

flea

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7
Q

noninfectious skin condition that is not related to skin cleansing- causative agent

A

propionibacterium acnes

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8
Q

sometimes its difficult to dx this without sx intervention

A

necrotizing fasciitis

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9
Q

fever, GI issues, HA, back pain, myalgia, cough, increased sweating, anemia, hypotension

A

malaria- plasmodium

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10
Q

thick discolored nails that may be mistaken for psoriasis

A

tinea unguium from dermatophytes (trichophyton, epidermophyton, microsporum)

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11
Q

Linked to Burkett’s lymphoma

A

EBV

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12
Q

this big blood sucking parasite causes a bite irritation is a easily spread in crowded conditions

A

pediculus humanus

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13
Q

cough, red gooey eyes, coryza, high fever

A

measles (prodrome)

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14
Q

Asymmetric attacks of swelling and pain at large joints (arthritis) + flu sxs

A

(Lyme) stage 2- subacute disseminated disease

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15
Q

avoid NSAIDS

A

cellulitis (can mask the pain if its something worse like necrotizing fasciitis)

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16
Q

a tunnel or burrow rash beneath the skin, anywhere on the body

A

scabies

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17
Q

mixed integumentary infection of aerobes and anaerobes

A

necrotizing fasciitis

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18
Q

macular patch of depigmented or hyperpigmented skin

A

tinea versicolor from malassezia furfur

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19
Q

infected hair breaks off leading to alopeica and black dots

A

tinea capitis aka ringworm of the scalp, from dermatophytes (trichophyton, epidermophyton, microsporum)

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20
Q

darkened and thickened skin from years of infestation

A

“vagabonds disease” from pediculus humanus (lice)

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21
Q

“this area is red-purple-blue-gray, hot, SHINY, and swollen AND THE WORST PAIN I HAVE EVER FELT”, but it doesn’t look like a big deal

A

streptococcal gangrene necrotizing fasciitis

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22
Q

this parasite needs blood to survive, and during its blood meal, it injects its saliva into your blood, which contains 15 substances known to initiate an allergic response

A

pulex irritans (human flea)

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23
Q

itching, peeling, scratching of skin on feet, usually toe webs and soles

A

tinea pedis from dermatophytes (trichophyton, epidermophyton, microsporum)

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24
Q

rash that’s borders blend in elevation and color to surrounding tissue

A

cellulitis

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25
Q

“my leg is hot, red, swollen, and painful”

A

cellulitis hallmarks (HEET) ** also seen in necrotizing fasciitis**

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26
Q

B cell infection that induces a polyclonal T cell reaction

A

EBV

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27
Q

mild sxs for several days (fever, HA, myalgia, N/V) followed by lacy rash on limbs and trunk

A

parvovirus B19

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28
Q

Facial palsy, follicular conjunctivitis, hepatitis, meningitis

A

random sxs (of Lyme) stage 2- subacute disseminated disease

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29
Q

what is the causative agent of bullous impetigo

A

SPECIFIC STRAINS of s. aureus

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30
Q

belt or stripe over dermatome

A

VZV- shingles

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31
Q

brownish lesion on hands and feet on someone living in the tropics

A

tinea nigra aka horaea werneckii

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32
Q

Agglutination of horse antibodies shows heterophile antibodies

A

EBV *** age specific, not in young kids

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33
Q

Residual tissue damage and autoimmune reactions like Reiters syndrome and guillain-barre

A

Post treatment Lyme disease syndrome

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34
Q

bite irritation and visualization are the clinical findings

A

phthirus pubs (pubic lice)

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35
Q

Lyme disease causative agents

A

Borreliela burgdorferi (N. America), b. Garinii and b. Afzelii (Europe)

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36
Q

I already had a skin infection (and it was probably necrotizing fasciitis) and then I got

A

streptococcal toxic shock syndrome

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37
Q

romanas sign

A

acute phase of chagas disease from trypansoma cruzi

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38
Q

this organism has short spikes on its legs that allow it to attach to its host

A

pulex irritans (human flea)

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39
Q

infestations common in kids, elderly, and people in crowded living situations

A

scabies

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40
Q

large spots on buccal mucosa

A

measles (kopliks spots)

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41
Q

“rash that looks like a sunburn on my right leg… now that you mention it, I fell down and scratched it a few weeks ago on some rocks but the wound is gone”

A

cellulitis

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42
Q

groups of 3 or 4 bites in a linear pattern, often on the lower extremities

A

flea

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43
Q

sterile bullae

A

ritters

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44
Q

superficial skin infection with crusting or bullae- causative agent

A

staphylococci and or streptococci (impetigo)

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45
Q

mild sxs for several days (fever, HA, myalgia, N/V) followed by slapped cheek rash

A

parvovirus B19

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46
Q

highly contagious respiratory droplet infection

A

measles

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47
Q

destroyed fascia but muscles are ok

A

necrotizing fasciitis

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48
Q

sudden pain, bronze skin, can hear popping under skin, blisters

A

gas gangrene

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49
Q

superficial skin infection with crusting or bullae and ulcers

A

ecthyma

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50
Q

Persistent arthritis attacks

A

(Lyme) stage 3 chronic disease

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51
Q

sudden onset of arthritis or arthralgia (in a parent)

A

parvovirus B19 (adult presentation)

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52
Q

androgen hormone triggers inflammation and folliculitis

A

acne vulgaris

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53
Q

sxs begin within 2-14 days after a bite and are nondescript GI sxs

A

RMSF- rickettsia rickettsii

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54
Q

3-4 days after prodrome comes a rash that goes from ears down + high fever

55
Q

when you perform sx, theres no pus, just a brown exudate and gray fascia

A

streotococcal gangrene necrotizing fasciitis

56
Q

Peak incidence between 17 and 25

A

EBV *** but earlier in africa***

57
Q

“abscess on my neck/ thigh/ butt/ face around a hair”- causative agent

58
Q

mild pain, itching, irritation with pustules or nodules surrounding a hair

A

folliculitis

59
Q

a risk factor for this type of necrotizing fasciitis is DM

A

type 1 polymicrobic

60
Q

HSV infection occur later and correlate with sexual activity

A

HSV-2 (genital herpes)

61
Q

shallow vesicles on an erythematous base, ballooning, vesicles crust over

62
Q

organism invades endothelial cells and is fatal if not treated within the first few days

A

RMSF- rickettsia rickettsii

63
Q

flesh eating bacteria

A

type 2 monomicrobic GAS necrotizing fasciitis

64
Q

2-5 days after infection, pt may develop small pink non itchy macules on wrists, forearms and ankles that spread to trunk, palms and sole

A

RMSF- rickettsia rickettsii

65
Q

fever, malaise, HA, myalgia, anorexia prodrome

A

VZV- chickenpox (only in older kids and adults!)

66
Q

acute infection of skin and deeper SQ tissues

A

cellulitis

67
Q

crawl very slowly, unable to jump or fly

68
Q

raised bump or halo surrounding the bite site

69
Q

vector for epidemic typhus

A

pediculus humanus

70
Q

bullae, thick pink/ purple fluid, cutaneous anesthesia, gangrene

A

streptococcal gangrene necrotizing fasciitis

71
Q

teratogens

A

VZV, rubella

72
Q

most commonly encountered opportunistic fungal infection that is in the normal flora of the skin and mucuous membranes

A

candidiasis

73
Q

looks like a mild, less infectious measles

74
Q

rash that is unilateral and does not cross the center line (sharp limits)

A

VZV- shingles

75
Q

Erythema migrans + flu sxs

A

(Lyme)Stage 1- acute localized disease of

76
Q

cervical dysplasia and cancer

A

HPV 16 and 18

77
Q

causative agent of gas gangrene

A

clostridium perfringens type A

78
Q

capable of jumping long distances

79
Q

ASA not recommended

A

VZV (incl vaccine)

80
Q

rubella aka

A

german measles (aka little red)

81
Q

asymmetrical vesicular rash that sometimes follows a dermatomal pattern

82
Q

exanthem subitum aka

A

HHV-6 aka roseola infantum aka 6th disease

83
Q

6+ days after infection, pt may develop red to purple petechial rash signifying a blood borne infection

A

RMSF- rickettsia rickettsii **** warning, at this point it may be too late***

84
Q

many co-infections with lyme disease

A

nantucket island fever from babesia microti

85
Q

itching and allergic response can take weeks to develop the first time, while during reinfections, the itching occurs within 24 hours

A

sarcoptes scabiei

86
Q

superficial cellulitis with focal dermal lymphatic involvement- causative agent

A

Group A Streptococcus (erysipelas)

87
Q

cemented to a fiber in clothing or hair

A

pediculus humanus

88
Q

this cellulitis has not responded to abx in the last 48 hours

A

because it’s necrotizing fasciitis

89
Q

“been hanging out in rainforests, not cleaning myself, and snorting cocaine, and now have this crusty rash”

90
Q

this tinea also usually has a fungal (candida) co-infection

A

tinea unguium from dermatophytes (trichophyton, epidermophyton, microsporum)

91
Q

HA fever malaise fatigue followed by sore throat and palatal enanthem at hard and soft palate junction with petechial lesions

92
Q

10-20% of PT’s who take abx after treatment of this disease develop Post Treatment ——-

A

Post Treatment Lyme Disease Syndrome

93
Q

anogenital warts

A

HPV 6 and 11

94
Q

muscle necrosis

A

gas gangrene

95
Q

chagoma

A

acute phase of chagas disease from trypansoma cruzi

96
Q

roseola infantum aka

A

HHV-6 aka exanthem subitum aka 6th disease

97
Q

observed most frequently in HIV pts, these crusted scaling lesions are intensively pruritic and teaming hundreds of thousands of the organisms

A

crusted Norwegian scabies

98
Q

causative agents in 90+% cases of cellulitis

A

s. aureus and s. pyogenes

99
Q

pain 1st, then + redness, then + papules and vesicles

A

VZV- shingles

100
Q

spread by direct contact with vesicular fluid, saliva, secretions

101
Q

often confused for scabies

A

pulex irritans (human flea)

102
Q

a pimple like rash that leads to intense itching at nightime

103
Q

big blisters that formed b/c of the exfoliative action of a toxin

A

bullous impetigo

104
Q

usual causative agent of folliculitis

105
Q

high fever (for 2-5 dyas) followed by rose colored rash

106
Q

parvovirus B19 aka

A

fifth disease aka erythema infectiosum

107
Q

cemented to hair

A

phthirus pubis (pubic lice)

108
Q

staphylcoccal scalded skin syndrome

109
Q

fever + boils that are deep into dermis and SQ on neck/ back/ thighs

110
Q

Hepatomegaly increased liver enzymes jaundice

111
Q

“went in a hot tub 8-48 hours ago and now I have painful, itchy bumps”- causative agent

A

pseudomonas aeruginosa (superficial “hot tub” folliculitis)

112
Q

with RMSF, every attempt should be made to begin tx before ____

A

petechiae develop

113
Q

humans are the only reservoir and asymptomatic shedding is possible

114
Q

“my leg is hot, red, swollen, painful, yellowish white in the center, has a central point (head), has pus”

115
Q

superficial acute infection of skin with focal dermal lymphatic involvement

A

erysipelas

116
Q

I had surgery WHILE on my period and wearing a tampon and I got

A

staphylococcal toxic shock syndrome

117
Q

fat, sweaty, diabetic person transmits this by towels, clothing and bed linens

A

tinea corporis and tinea cruris from dermatophytes (trichophyton, epidermophyton, microsporum)

118
Q

Downy cells in circulation

A

Atypical lymphocytes (EBV)

119
Q

clostridial myonecrosis aka

A

gas gangrene

120
Q

muscle and nerve degeneration and necrosis, enlarged heart, esophagus, and colon, chronic inflammation

A

chronic phase of chagas disease from trypansoma cruzi

121
Q

vesicles that are ruptured and crusty- causative agent

A

s. aureus (+ maybe strep pyogenes co-infection)

122
Q

toxins are superantigens that over stimulate immune response

A

toxic shock syndrome

123
Q

An under appreciated reservoir is small children

124
Q

kissing bugs

A

triatomine bugs- trypanosoma cruzi

125
Q

Symmetrical LAD

126
Q

rubeola aka

127
Q

small pinpoint lesions

N/V/D

cough

hypotension

anemia

A

nantucket island fever from babesia microti

(the non bolded sxs are the malaria like sxs)

128
Q

What’s the dif b/w EBV and CMV

A

CMV HAS NO HETEROPHILE ANTIBODIES

129
Q

skin across entire body is split off within 2 days, and body looks red and burnt

130
Q

accounts for most measles deaths

131
Q

mimics gastroenteritis, PNA, meningitis, encephalitis, hepatitis

A

malaria- plasmodium

132
Q

blood-filled lesions that resemble Norwegian scabies (crusted and scaly)

A

pediatric scabies

133
Q

HSV infection that occurs early in life

A

HSV-1 (oral herpes)

OBGYN microbiology (13 decks)

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