Musculoskeletal and Infectious Disease Flashcards
How many bones at birth
300
Birth/childhood bone qualities
- Large cartilage component
- More flexible
- Dense periosteum
- Open epiphyseal plate
- Spine c-shaped
How many bones in adulthood
206
Adulthood bone qualities
- Bones are ossified
- Bones more dense and rigid
- Spine s- shaped
Factors in Bone and muscle development
- Genetics
- Gender
- Mechanical force
- Nutrition
- Environment/context
- Sleep
Assessment of musculoskeletal system
- history
- size and symmetry of body
- joint range of motion
- gait
- muscle strength
- muscle tone
- developmental milestones
- nutrition
- pain
Diagnostic and Lab test
- X-ray
- bone scan
- CT
- electromyelogram
- MRI
- ultrasound
- bone marrow biopsy
- muscle biopsy
- serum enzymes
Physical complications of musculoskeletal conditions
- pain
- cardiovascular
- respiratory
- GI/GU
- skin
- nutrition/metabolic
- safety
psychosocial complications of musculoskeletal conditions
- Depression
- anxiety
- fear
- altered development
- altered body image
- family disruption/dysfunction
Talipes equinovarus (congenital club foot) etiology
unknown, abnormal intrauterine positioning, family hx
Talipes equinovarus treatment
- non-operative serial casting ASAP after birth
- casting every 1-2 weeks
- surgery may be required, followed by a long cast and bracing
Talipes equinovarus nursing care
- long range planning for casting and appointments
- promotion of normal developmental needs
- cast care
- monitoring neuro function
Developmental dysplasia of the hip etiology
- Physiologic factors (maternal hormones that lead to laxity of the joints, intrauterine positioning)
- Mechanical factors (delivery methods, postnatal positioning)
- Genetic factors
Developmental dysplasia of the hip treatment
Pavlik harness (newborn-6 mo)
Traction (6 mo-18 mo)
Closed surgical reduction with casting (spica cast)
Developmental dysplasia of the hip nursing care
- Monitoring for skin breakdown or neurovascular compromise
- Teaching for parents and caregivers
- Promotion of normal developmental activities
- Safety and home modifications
Genu varum
bow-leg
Fenu valgum
Knock-knee
Legg-Calve-perthes etiology
Coagulation Disorder
Genetic and family history
Mild trauma to site
Legg-Calve-perthes progression
Prenecrosis –insult to blood supply
Necrosis/asymptomatic – 3-6 months (stage I)
Revascularization – 1-4 years (stage II)
Bone Healing - reossification (stage III)
Remodeling – improved joint function (stage IV)
Legg-Calve-perthes treatment
- Bracing or traction
- Surgery to release muscles, treat acetabulum or femur, and restore range of motion
Legg-Calve-perthes complications of untreated disease
- Osteoarthritis
- Hip dysfunction
- Shorten height
- Leg length discrepancy
Slipped Capital Femoral Epiphysis etiology
Obesity Recent growth spurt Hypothyroidism Hypogonadism Can be acute or gradual
Slipped Capital Femoral Epiphysis clinical manifestations
Pain in affected hip or referred to knee
Change in hip range of motion
Slipped Capital Femoral Epiphysis treatment
- Surgical fixation of hip and bone graft followed by
- Spica cast or russell traction
Slipped Capital Femoral Epiphysis nursing care
Pain management Monitor for complications of immobility Traction or cast care Body image Weight control (proper nutrition and fluid intake)
Scoliosis etiology
- congenital
- idiopathic
- acquired
Scoliosis manifestations
Truncal asymmetry Uneven shoulders Uneven hips On-sided rib hump Prominent scapula Hip and back pain Extreme fatigue Dyspnea with exertion
Scoliosis treatment- 10-20 degrees
Physical Therapy to improve posture and muscle strength and tone
Scoliosis treatment- 20-40 degrees
Bracing with Boston brace in order to maintain current level of curve. Does not reduce the degree of curve.
Scoliosis treatment- over 40 degrees
spinal fusion with segmental instrumentation
Muscular Dystrophy – Duchenne’s etiology
Genetic- autosomal recessive (x-linked)
Muscular Dystrophy – Duchenne’s clinical manifestations
Lower extremities weakness Gower Sign Toe-walking Tripping Enlarged calves Progresses to complete immobility, respiratory difficulty and failure, cardiomyopathy, fractures, chronic disability