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Praxis > Motor speech and dysphagia > Flashcards

Motor speech and dysphagia Flashcards

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1
Q

what are the characteristics of AOS

A
  1. slower rate of speech
  2. distorted speech sound substitutions
  3. syllable segmentation
  4. articulatory groping, false starts and restarts
  5. prosodic impairment
  6. more errors on longer utterances
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2
Q

what are the characteristics of AOS in adults

A

unimpaired reflex and automatic acts. The difficulty is with voluntary movement

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3
Q

what does AOS typically associate with

A
  1. aphasia, especially Brocas

2. less frequently with unilateral uppermotor neuron dysarthria

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4
Q

what is primary progressive apraxia of speech

A
  1. when AOS is the only dominant symptom in primary progressive aphasia or dysarthria
  2. insidious onset and slow progress as compared to stroke induced AOS which is sudden with some improvement then stabilized
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5
Q

what is the neuropathology of AOS

A
  1. most often from a single left hemisphere stroke
  2. progressive AOS may be caused by degenerative diseases that cause primary progressive aphasia
  3. can be caused by left hemisphere trauma
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6
Q

what are the general symptoms of AOS

A
  1. impaired oral sensation in some
  2. language disorders may coexist if aphasia present
  3. if dysarthria coexists, facial and lingual weakness
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7
Q

what are the communication deficits in AOS

A
  1. auditory processing deficits
  2. have general awareness of speech problem which causes frustration
  3. slow or delayed initiation of speech
  4. may use compensatory strategy of reduced rate
  5. speech production errors
  6. prosodic problems
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8
Q

what are the speech production errors seen in AOS

A
  1. problems in volitional or spontaneous sequencing of movements with unaffected automatic speech
  2. high variability of speech errors that change on repeated attempts
  3. speech sound substitutions more common that distortions and omissions
  4. voicing errors (voiceless for voiced)
  5. more difficulty with consonants than vowels
  6. regressive assimilation
  7. progressive assimilatio
  8. metathetic errors ( switched positions of phonemes)
  9. insertion of schwa into consonant clusters
  10. increased frequency of errors on longer words
  11. trial and error groping
  12. greater difficulty with word initial sounds
  13. attempts at self repair
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9
Q

what prosodic problems are associated with AOS

A
  1. slower rate with difficulty changing the rate when requested
  2. impaired intonation becuse of increased duration of consonants
  3. even stress and restricted range of loudness, limited pitch
  4. fluency problems (silent pauses especially at initiation
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10
Q

How is AOS assessed

A
  1. check for aphasia when the patient shows coexisting signs
  2. tape record speech sample and transcribe. Note groping, self correction, repetition, dysfluencies, facial grimacing etc.
  3. evoke imitation of speech sounds
  4. diadochokinesis
  5. imitate progressively longer words, phrases, sentences
  6. evoke counting
  7. picture description
  8. oral reading
  9. check for oral apraxia or dysarthri
  10. assess limb apraxia
  11. standardized test: Apraxia Battery for Adults
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11
Q

How is AOS treated

A

behavioral treatment

  1. treatment should include instructions, demonstration, modeling, shaping, phonetic placement, frequent cueing, use of rhythm, immediate positive or corrective feedback
  2. treatment targets include artic accuracy, slower rate, systematic practice, gradual increase in rate, and normal prosody
  3. more difficult words promote better generalization
  4. include practice with a variety of sounds and sound combinatins
  5. repeated trials on same target response necessary to start
  6. cueing includes tactile cues for artic placement, simultaneous production, modeling, delayed imitation
  7. contrastive stress, phonetic cotrasts, carrier phrases, and singing may be useful
  8. push on abdomen to get vocal fold closure
  9. metronome or pacing board may be useful
  10. emphasize total communication
  11. teach self monitoring
  12. teach family members to speak slower when client is focused and use total communication
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12
Q

what is SPT or sound production treatment

A

minimal contrast therapy

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13
Q

what is the pathology of ataxic dysarthria

A
  1. appears only during movement
  2. will be manifested in other motor movements
  3. neuropathology is bilateral or generalized cerebellar lesions
  4. Friedreich’s ataxia is a degenerative form
  5. ataxic gait, nystagmus, and dysmetria may be present
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14
Q

what is dysmetria

A

undershooting and overshooting a target

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15
Q

what are the characteristics of ataxic dysarthria

A
  1. gait disturbance
  2. movement disorder (overshoot, undershoot, jerky, etc)
  3. respiratory disorder: exaggerated and paradoxical movement during speech
  4. artic disorders:imprecise consonants, irregular artic breakdown, distorted vowels
  5. prosodic disorder: excessive and even stress, prolonged phonemes, intervals between wors or syllables, slow rate
  6. phonatory disorder: monopitch, monoloudness, harshness
  7. speech quality: sounds drunk
  8. resonance disorder: intermittent hyponasality on some
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16
Q

what is flaccid dysarthria

A
  1. damage to LMN for volitional and nonvolitional movement
  2. characterized by hypotonia
  3. can be caused by degenerative disease (ALS),motor neuron disease, progressive bulbar disease, MS
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17
Q

what are the major characteristics of flaccid dysarthria

A
  1. muscular disorders: weakness, hypotonia, atrophy, diminished reflexes, fasiculations, fibrillations, rapid and progressive weakness with use
  2. respiratory weakness: reduced subglottic air pressure, weak inhalation
  3. phonatory disorder: breathy voice, audible inspiration
  4. resonance disorder: hypernasality, nasal emission
  5. artic disorder: imprecise consonants, weak pressure consonants
  6. cluster: phonatory incompetence, resonatory incompetence, phonatory-prosodic insufficiency
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18
Q

what is the etiology of hyperkinetic dysarthria

A
  1. results from damage to the basal ganglia
  2. produces variable muscle tone and involuntary movement
  3. commonly caused by degenerative disease (Huntington’s), vascular disease (brainstem stroke), trauma, toxic condition,
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19
Q

what are the major characteristics of hyperkinetic dysarthria

A
  1. orofacial dyskinesia: abnormal, involuntary, movement of orofacial muscles
  2. myoclonus: involuntary jerks of body
  3. tics: patterned, rapid, commonly of face and shoulders
  4. chorea: purposeless, random, involuntary
  5. athetosis: slow, writing,
  6. Ballism: bilateral, involutary movement of extremities
  7. hemiballism: unilateral ballism
  8. dystonia: contractions of antagonistic muscles that cause abnormal postures
  9. respiratory problems: audible inspiration, forced sudden inspiration not typical of other dysarthrias
  10. phonatory disorder: voice tremor, strained voice, voice stoppage, vocal noise
  11. resonance disorders: hypernasality
  12. artic problems: impreise consonant production with distorted vowels and slow rate
  13. prosodic problems: prolonged interword intervals, inappropriate silent periods, phoneme prolongations, excess and equal stress
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20
Q

what are the characteristics of hypokinetic dysarthria

A
  1. resting tremors
  2. mask face with infrequent blinking, no smiling
  3. micrographic writing
  4. walking disorder
  5. postural disturbances
  6. decreased swallowing: drooling
  7. respiratory problems: reduced vital capacity, irregular breathing
  8. phonatory disorders: monopitch, low pitch, harsh and breathy voice
  9. prosodic disorder: reduced stress, inappropriate silent intervals, short rushes of speech
  10. artic disorders: imprecise or distorted consonants, stops sound like fricatives
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21
Q

what is the etiology of spastic dysarthria

A

bilateral damage to upper motor neuron

  1. most commonly caused by multiple strokes that damage both the pyramidal and extrapyramidal systems
  2. Cerebral palsy in children
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22
Q

what are the characteristics of spastic dysarthria

A
  1. spasticity and weakness
  2. movement disorder: reduced range, force, and speed, loss of fine and skilled movement, increased muscle tone
  3. artic: imprecise consonants and distorted vowels
  4. prosody: excess and equal stress, slow rate, reduced stress, short phrases
  5. phonatory: hyperadduction of folds,
  6. resonance disorder: predominant hypernasality due to inadequate closure of velopharyngeal port
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23
Q

what are the most common mixed forms of dysarthria

A
  1. flaccid-spastic: associated with ALS

2. ataxic-spastic: associated with MS

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24
Q

what is the etiology of UUMN dysarthria

A
  1. purely anatomical

2. most common cause is stroke

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25
Q

what are the characteristics of UUMN dysarthria

A
  1. neurologic impairmet: unilateral lower face, tongue, palate weakness, and hemiplegia
  2. artic disorder: imprecise production of consonants, irregular artic breakdown, some vowel distortion
  3. phatory disorder: harsh voice, reduced loudness, strained, hoarse etc
  4. prosodic disorder: slow rate, equal and excess stress
  5. resonance : hypernasality or nasal emission
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26
Q

how do you assess the dysarthrias

A
  1. conversational speech sample and reading sample
  2. speech tasks including imitation of syllables, words, etc
  3. diadochokinetic rate AMR’s, and SMR
  4. oral mech exam
  5. observe patient posture and breath
  6. assess phonation: ahs
  7. assess artic, prosody, resonance, intelligibility,
  8. assess muscle strength, speed, range, accuracy, tone, and steadiness of movement
  9. use a standardized test
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27
Q

what are some standardized tests for dysarthria

A
  1. Assesment of Intelligibility of Dysarthric Speech
  2. Quick assessment for dysarthria
  3. Frenchay dysarthria assessment
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28
Q

how do you treat respiration in dysarthria

A
  1. train maximum vowel prolongation
  2. production of longer phrases
  3. controlled exhalation
  4. have patient push, pull, or bear down during speech tasks
  5. use resistive breathing devices to increase breath support
  6. use a manual push on the clients abdomen
  7. teach client to inhale more deeply and exhale slowly with greater force
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29
Q

how do you treat resonance

A
  1. provide feedback on nasal airflow by mirror, nasal flow transducer, or nasendoscope
  2. train the client to open mouth wider to increase oral resonance and vocal intensity
  3. use a nose clip
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30
Q

how do you modify articulation in dysarthria

A
  1. use a bite block to improve jaw control
  2. model, shape, immediate feedback, phonetic placement, slow rate
  3. minimal constrast pairs
  4. relaxation techniques such as jaw shaking exercise to loosen up muscles
  5. teach compensatory artic movements
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31
Q

what is the most common causes of dysphagia

A
  1. brainstem strokes
  2. tumors and neurologic disease
  3. surgery or radiation treatment
  4. TBI
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32
Q

what are disorders of the oral prep phase

A
  1. chewing problems because of reduced movement, buccal tension, and poor alignment of mandible and maxilla
  2. difficulty forming and holding bolus
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33
Q

what are disorders of the oral phase of swallowing

A
  1. begins with anterior to posterior tongue action.
  2. ends when food passes through the faucial arches
  3. anterior instead of posterior tongue movement, weak tongue movement, reduced range of movement and elevation
  4. tongue thrust
  5. decreased tongue sensation may cause premature loss of bolus
  6. residue due to incomplete swallow
  7. premature swallow before swallow
  8. piecemeal swallow
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34
Q

what are disorders of the pharyngeal phase of swallowing

A
  1. involves velopharyngeal closure, laryngeal elevation, opening of UES
  2. delayed or absent swallow reflex
  3. pharyngeal stripping problems
  4. residue in valleculae due to reduced base of tongue strength
  5. aspiration before and after swallow
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35
Q

what are disorders of the esophageal phase of swallowing

A
  1. not under voluntary control
  2. food propelled through peristaltic action and gravity
  3. difficulty passing the bolus through the cricopharyngeal muscle
  4. backflow from esophagus to pharynx
  5. diverticulum (pouch that collects food)
  6. tracheoesophageal fistula
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36
Q

what is achalasia

A

esophageal swallowing disorderdue to esophageal motility impairment. Food remains in the esophagus

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37
Q

how are swallowing disorders assessed

A
  1. assess ethnocultural background, food habits, restrictions
  2. screen speech, voice, language, and writing skills during clinical interview. Note voice quality, hypo and hypernasality
  3. screen concrete and abstract language comprehension through verbal commands and patient defining proverbs and phrases
  4. oral mech for strength and range of motion
  5. bedside swallow test
  6. examine laryngeal elevation by placing fingers on chin, hyoid, and larynx
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38
Q

how should you position the patient for test swallows

A
  1. tilt head downward as food is placed in mouth, and tilt head back when swallow is initiated in the case of tongue weakness
  2. for hemilaryngectomy, delayed swallow, inadequate laryngeal closure, tilt head down to hold food in valleculae until the reflex is triggered
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39
Q

what are direct treatments of swallowing disorders

A
  1. food or liquid is placed in mouth to shape appropriate swallowing
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40
Q

how do you treat disorders of the oral prep phase of swallow

A

teach the patient to

  1. press tongue against hard palate
  2. keep food on more mobile side of the tongue or mouth
  3. apply pressure to damaged cheek to increase tension
  4. tilt head to stronger side
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41
Q

how do you treat disorders of the pharyngeal phase of swallowing

A
  1. chin tuck to compensate for delayed or absent swallow reflex. Widens the valleculae so the bolus can collect there before the swallow
  2. alternate liquid and semisolid swallows to help clear the pharynx with reduced peristalsis
  3. multiple swallows
  4. tilt head to strong side for unilateral paralysis, decreased laryngeal elevation, poor epiglottic inversion, or decreased PES opening
  5. tilt head to weak side if there is weak pharyngeal contraction on one side. It forces the bolus down the intact side
  6. tilt head forward while placing pressure on thyroid cartilatge on damaged side to improve laryngeal closure
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42
Q

how do you treat disorders of the esophageal phase of swallowing

A
  1. avoid foods
  2. small portions, 2-3 hours before bed, elevate head
  3. lose weight, stop smoking
  4. remain upright for 30 minutes after eating
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43
Q

what are indirect treatment of swallowing disorders

A

does not involve food

exercises and skill training designed to improve muscle strength

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44
Q

how do you increase the range of tongue movement

A

raise tongue, hold as high as possible, alternate raising and lowering

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45
Q

how do you increase buccal tension

A

stretch lips tightly and say “ee”
round lips tightly and say “oh”
alternate

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46
Q

how do you increase range of lateral movement of the jaw

A

wide opening and sideways movement of jaw

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47
Q

how do you increase tongue resistance

A

push tongue against a tongue depressor

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48
Q

how do you stimulate the swallow reflex

A
  1. thermal stimulation of anterior faucial arch with laryngeal mirror dipped in ice water
  2. ask patient to swallow after stiulation without food
  3. practice liquid swallow after stimulation
  4. increase consistency
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49
Q

how do you improve adduction of folds

A
  1. holding breath, patient pushes down on chair

2. lift or push with simultaneous voicing

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50
Q

how do you increase base of tongue strength

A
  1. Masako maneuver: patient holds anterior tongue between teeth and dry swallows
  2. can also be strengthened by sucking a thick consistency through a straw
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51
Q

what is the supraglottic swallow maneuver

A
  1. helps close airway at folds
  2. patient holds food in the mouth and takes a deep breath, exhales, then holds breath
  3. swallow while holding breath then cough
52
Q

what is the super-supraglottic swallow

A
  1. promotes false vocal fold closure
  2. patient inhales and holds breath by bearing down
  3. patient swallows while doing this, then coughs
53
Q

what is the effortful swallow

A
  1. increases posterior motion of tongue and increases pharyngeal pressure
  2. patient squeezes hard as possible while swallowing
  3. may be more effective with electrical stimulation
54
Q

what is the Mendelsohn maneuver

A
  1. helps elevate the larynx and widen the cricopharyngeal opening
  2. educate patient first about laryngeal elevate by having them feel the larynx while they swallow salive
  3. patient holds the laryngeal elevation during swallowing for progressively longer durations
55
Q

what are some medical treatments for swallowing disorders

A
  1. cricopharyngeal myotomy: cricopharyngeal muscle is split to create an open sphincter
    recommended for Parkinsons ALS
56
Q

what is an esophagostomy

A

designed for those who cant take oral food

feeding tube inserted into esopharus

57
Q

what is nasogastric feeding

A

tube inserted through the nose into the stomach

58
Q

what is a pharyngostomy

A

tube inserted into esophagus and stomach through a hole in the pharynx

59
Q

what are the muscles of mastication and their innervation

A
  1. temporalis: trigeminal
  2. masseter: trigeminal
  3. lateral and medial pterygoid: trigeminal
60
Q

which face muscles are used for swallowing and their innervation

A

1.orbicularis oris: labial seal
2.buccinator : cheeks
3depressor labii inferioris: depress lower lip
4levator labii superioris, zygomaticul minor and major: elevators
all facial nerve VII

61
Q

what are the supra hyoid muscles and innervation

A
  1. geniohyoid: cervical nerve
  2. digastric and mylohyoid: trigeminal
  3. stylohyid: facial nerve
62
Q

what happens in the oral phase of the swallow

A
  1. masticatory mucles form bolus and place on tongue
  2. tip of tongue eevated and moves bolus back into the oro-pharynx
  3. sensory receptors in posterior guid initiation of pharyngeal swallow
63
Q

what happens during the pharyngeal phase

A
  1. velopharyngeal closure
    contraction of posterior tongue and forward movement of the LPW and PPW
  2. bolus moves from high pressure to low contributing to pharyngeal stripping
  3. suprahyoid muscles move the hyoid up and forward
  4. larynx is closed through epiglottic inversion and laryngeal squeeze
  5. food reaches UES
64
Q

what does the trigeminal nerve innervate

A

maxilary branch: sensory: upper lip, maxillary teeth and palate
Mandibular branch: sensory: anterior 2/3rd tongue, mucous membranes of mouth, cheeks, gums
motor: masticatory muscles, tensor veli palatine, mylohyoid and digastric

65
Q

what does the facial nerve innervate

A

sensory: chorda tympani: taste anterior 2/3rd tongue
motor: lip sphincter

66
Q

what does the glossopharyngeal nerve innervate

A

sensory: visceral afferent: taste from posterior 1/3rd tongue
motor: stylopharyngeus, pharyngeal constrictors

67
Q

what does the vagus nerve innervate

A

sensory: hypopharynx, larynx
motor: velum, pharynx, larynx, esophagus

68
Q

what does the hypoglossal nerve innervate

A

all tongue muscles except palatoglossus

69
Q

how is the brainstem involved

A

sensory info terminate in nucleus of tractus solitarius

motor info goes through the nucleus ambiguous

70
Q

what are the 2 pathways for swallowing control

A
  1. sensory info to cortex via thalamus, motor commands through cortico-bulbar pathways
  2. sensory info reaches nucleus tractus solitarius, is relayed to reticular formation and commands modulated by nucleus ambiguous
71
Q

what do lesions in the insula cause

A

reduced magnitude of sensory input resulting in delayed swallow, decreased taste, volume, and temperature

72
Q

what does a high volume bolus create

A
  1. more hyoid displacement

2. prolonged UES opening

73
Q

what does a viscous bolus create

A
  1. increased oral pressure
  2. high chance of aspiration for thin consistency
  3. more residue with thick
74
Q

what are symptoms of impaired oral bolus prep

A
  1. prolonged mastication
  2. prolonged oral transit time
  3. premature spillage of bolus anteriorlyy or posteriorlly
  4. oral residue
75
Q

what are symptoms of delayed swallow initiation

A
  1. pre swallow pooling before swallow

2. laryngeal penetraion before swallow

76
Q

what is the symtom of impaired tongue base retraction

A

residue in valleculae

77
Q

what is the symptom of impaired epiglottic inversion, laryngeal vestibule squeeze

A

laryngeal penetration during the swallow

78
Q

what is the symptom of impaired UES opening, hyoid excursion

A
  1. residue in pyriform sinuses

2. penetraion after the swallow

79
Q

what is the difference between non nutritive and nutritive sucking in an infant

A

infant breathes with non nutritive

nutritive suck swallow ration is 1 : 1

80
Q

what is the difference between suckle and sucking

A

suckle is backward-forward, sucking is up and down

81
Q

what is the tongue thrust reflex

A

when lips are touched, tongue moves out of the mouth to allow for feeding from breast or bottle

82
Q

what are swallowing issues associated with aging

A 
1. muscle atrophy
decreased range of motion, speed, strength
diminished sensory
respiratory compromise
2. prolonged mastication, poor dentition
3. delayed swallow initiation
4. inadequate hyolaryngeal excursion
5 increased laryngeal penetration
6. sensory and motor impairments leading to residue
83
Q

what are the side effects of radiation therapy

A
  1. reduced blood supply which damages nerve endings
  2. nausea
  3. fibrosis
  4. skin irritation
  5. xerostomia
  6. peripheral neuropathy
  7. necrosis of tissue
  8. reduced range of motion, flexibility of structures, speed
  9. reduced sensation of bolus
  10. reduced synchrony
84
Q

what are characteristics of lip cancer

A
  1. present as non healing ulcers
  2. pain in advanced stage
  3. treated with radiation
  4. mostly oral stage deficits
  5. radiation may affect other structures
85
Q

what are characteristics of floor of mouth cancer

A
  1. can spread to bone and tongue causig glossectomy
86
Q

what are characteristicsof tongue cancer

A
  1. mostly oral stage
  2. partial or total glossectomy
  3. cant create bolus or create enough pressure to push it back
87
Q

what is retro moar trigone

A

cancer in triangular space behind last molar

affects mastication, oral control, posterior propulsion

88
Q

what is achalasia

A

insufficient LES relaxation and loss of esophageal peristalsis
feels like something stuck in throat
symptoms: dysphagia, regurgiotatin, chest pain

89
Q

what is diffuse esophageal spasm

A

contractions of smooth muscle portiaon of esophagus
striated portion and LES relax normally
corkscrew esophagus
peristaltic action compromised

90
Q

what are strictures

A

loss of lumen area in esophagus
normal esophagus is 20 mm
strictures is when lumenal diameter is less than 15 mm

91
Q

what are schatzki rings

A

narrowing of lower esophagus by rings of mucosal tissue

92
Q

what is Zenkers diverticulum

A

small pouch close to UES

associated with cough, bad breath, regurgitation of undigested food

93
Q

what is scleroderma

A

connective tissue disorder that weakens LES causing GERD

affects mostly smooth mucle (lower 2.3)

94
Q

what is necrotizing enterocolitis

A

food reacts with bacteria in mucosa causing throwing up and diarrhea
reflux impacts swallowing

95
Q

what is infant respiratory distress syndrome IRDS

A

when infant first egins to breathe, alveoli open, collapse then stick together after each breath
surfactant deficiency

96
Q

what is transient tachypenea

A

poor clearance of lung fluid during birth

97
Q

what is bronhopulmonary dysplasia

A

chronic lung disease due to prematurity
most common cause is mechanical ventilation
primary problem is poor separation of oxygenated and deoxygenated blood

98
Q

what is atrioventricular septal defect

A

malformation of heart from failure of normal separation of ventricular inlet
characteristic feature of Down syndrome

99
Q

what is included in a bedside swallow screening

A
  1. history
  2. cranial nerve exm
  3. trial swallows
100
Q

what should you observe during screening

A
  1. mental status, nutritional status, respiratory status
101
Q

what is included in the history

A
  1. sympotms such as signs of choking, weight loss, complaints of food going down wrong pipe or getting stuck
  2. past and current medical history
  3. any previous swallowing assessments
  4. socio cultural status
102
Q

how do you examine the trigeminal nerve

A
  1. facial sensation using cotton wisp or sharp object
  2. corneal reflex: touching cornea with cotton wip to look for asymmetries in blinking
  3. jaw jerk reflex: tap on jaw with mouth slightly open
103
Q

what do you examine the facial nerve

A
  1. look for asymmetry
  2. ask patient to smile, puff cheeks, wrinkle brow
  3. check taste with sugar, salt, or lemon juice to lateral aspect of each side of tongue
  4. UMN lesions: contralateral lower face weakness
  5. LMN weakness whole ipsilateral face
104
Q

how do you examine the glossopharyngeal nerve

A
  1. palatal elevation and gag reflex

2. taste in posterior tongue

105
Q

how do you examine the vagus nerve

A
  1. vocal functions

2. ability to cough voluntarily

106
Q

how do you examine the hypoglossal nerve

A
  1. note atrophy or fasiculations
  2. patient sticks out tongue and see if it curves to one side
  3. patient moves tongue side to side and pushes against cheek
  4. listen for artic errors
107
Q

what is a dye test

A

blue dye determines if there is aspiration in a trach patient
2. dye placed in mouth, trach cuff deflated and suctioned for secretions resting on or above the cuff

108
Q

what are compensatory strategies for swallowing

A
  1. chin tuck: initiation and airway protection
  2. head back: oral stage deficits: uses gravity
  3. small sips and bites
  4. repeat swallows
  5. thickening viscosity
  6. head tilt to strong side
  7. head turn to weak side
  8. supra glottic maneuver
109
Q

what exercises target delayed initiatin of swallow

A
  1. thermal tactile stimulation

2. air pulse application

110
Q

what exercises target BOT to PPW

A
  1. Tongue back/ Masako
  2. Effortful Swallow
  3. Tongue pull back maneuver
111
Q

which exercises increase hyolaryngeal excursion

A
  1. mendelsohn maneuver
  2. chin tuck against resistance
  3. Shaker
112
Q

what exercises strengthens the pharynx

A

supraglottic swallow

113
Q

what is FEEST

A

fiberoptic endoscopy evaluation with sensory testing

  1. adds device that sends air pulses
  2. laryngeal adductor reflex in response to air
114
Q

what is a J tube

A

inserts predigested food into intestines

115
Q

what is a PG tube

A

feeds directly into stomach

116
Q

what is the Shaker exercise

A

1 minute sustained head raise for isometric
30 consecutive head raises for isokinetic
improves premature bolus loss, residue in valleculae, laryngeal elevation, epiglottic closure, residue in pyriform sinuses, pharyngeal transit time, and aspiration

117
Q

which exercises aid pharyngeal stripping

A

mendelsohn and Shaker

118
Q

what is the Masako maneuver

A

aids base of tongue to posterior wall

tip of tongue between teeth while patient dry swallows

119
Q

what is effortful swallow

A

imagine you have ping pong ball in mouth that you have to swallow
10 reps
helps BOT-PPW

120
Q

what is supraglottic swallow

A

for airway protection

  1. hold breath
  2. swallow hard
  3. cough
  4. swallow again 10-12 times
121
Q

what is mendelsohn maneuver

A

for hyolaryngeal extension-UES

at peak of swallow patient squeezes and holds as long as they can

122
Q

how does pitch gliding help

A

increases length and tension of folds to improve airway protection

123
Q

what does chin tuck and head tilt help

A

residue in vallecuae

124
Q

what does head rotation help

A

residue in pyriform sinuses

125
Q

what affect does a trach have on swallowing

A

increased risk of aspiration due to

  1. poor sub glottic pressure
  2. poor laryngeal elevation
  3. reduced airway sensitivity
  4. general muscle weakness
126
Q

what consistencies are used for trial swallows

A

thin: 5 ml 10 ml 20 ml continuous
puree or pudding: 5 ml 10 ml 20 ml continuous
solid

127
Q

what should you observe during a bedside swallow

A
  1. laryngeal palpation: laryngeal elevation
  2. timing/ complete swallow/ number of swallows
    3pre-post voice quality
  3. coughing/clearing
  4. oral residue

Praxis (20 decks)

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