motor recovery Flashcards

1
Q

mm synergies

A

firing of several mm groups simultaneously

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2
Q

normally we move in numerous __ movement patterns

A

variable

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3
Q

after stroke we move in __ ways

A

stereotypical

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4
Q

stereotypical snergies

A

initial: flaccid
followed by: spasticity, hyperreflexia , stereotypical movements
-inflexible, invarient

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5
Q
UE flexor synergy pattern 
scapula
shoulder
elbow
wrist
flexrors thumb
A
elevates/retracts
abd/ER
Flex/sup
flex
flex/add
flex/add
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6
Q

LE extensor synergy

Hip
knee
ankle
great toes

A

adducts/ IR
extends
PF/Inversin
extend

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7
Q

strong components of flexor synergy

A

elbow flexion

hip flexion

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8
Q

strong components of extensor synergy

A

shoulder adducts/ IR

knee extends

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9
Q

Brunnstrom stages of motor recovery

A

1-6

flaccid to spastic to normal

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10
Q

chedoke stroke assesemtn

A

1-7

flaccid to spastic to normal

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11
Q

brunstrom stages with no voluntary movement

A

stage 1 /2

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12
Q

brunstrom spastic movement levels

A

3-6

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13
Q

Motor recovery stage 1

UE

A

flaccid
hyporeflexic
no active reflex / mm

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14
Q

motor recovery 2

UE

A

spasticity
reflex response to facilaition
no voluntary mm

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15
Q

motor recovery stage 3 UE

A

spasticity marked
synergistic movement can be elicited volunitariy
pattern of movement within limited range

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16
Q

motor recovery stage 4 UE

A

spasticity decrease
less resistance to passive
synergy pattern can be reversed
movement possible within greater range

17
Q

motor recovery stage 5

A

spasticity wanes but evident w rapid movement
able to reverse patterns
isolated movement possible
specific movements

18
Q

motor recovery stage 6

A

no resistance to passive movemnt
controlled isolated movement
lacks normal speed

19
Q

chedoke stage 7

A

normal movement, normal speed

no evidence of functional impairment

20
Q

which one has stage 7 chedoke or fugl

A

chedoke

21
Q

3 purposes of chedoke

A

stage the recovery
predict outcome
measure clinical important change

22
Q

chedoke 2 inventories

A

physical (shoulder posture arm, hand leg foot)

and activity (gross and walking)

23
Q

associated reactions

A

unintentional involuntary movements caused by lack of inhibition from CNS

can become conditioned

can occur with voluntary action of another limb

24
Q

post stroke shoulder pain from

A
snap malignment
IR humerus
neglect
sensory loss
UE low stage in recover CMSA 123
25
Q

is shoulder sublaxation common? preventable

A

yes n yes

26
Q

relationship between shoulder subluxation and pain direct or not

A

not direct

27
Q

can should sublax if increased mm tone

A

yes

28
Q

how to prevent shoulder sublax

A

support it
education
joint approximation
maintain ROM (safe below 90)

29
Q

contributing factors to shoulder sublaxation

A

lack of support in upright / standing
pulling on hemiplegic during transfers
improper positioning in bed / wc

30
Q

joint approximation

A

mm stimulation
strengthening
braces
taping

31
Q

complication of spastic UE

A
adhesive capsulitis 
decrease functional movement 
loss of scapular mobility
loss of humeral alignment
contracture/ loss ROM
32
Q

best practice for shoulder PROM Arm

A

do not move shoulder past 90 of shoulder flex / abd UNELSS scapular upwardly rotated and humerus laterally rotated