Module 11 (Final) Flashcards

1
Q

Rh Incompatibility

A

Rh - mother carrying a Rh + fetus

Mother’s body considers Rh+ blood an antigen and an antibody response is triggered.

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2
Q

When does Rh incompatibility take place?

A

Pregnancy: small amount of fetal blood may enter maternal circulation VIA microtears in placenta

Placental separation (greatest risk): Larger amounts of fetal blood enter maternal circulation

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3
Q

Fetal/Newborn Effect of Rh incompatibility

A

Antibodies from mother attack Rh+ blood cells. Hemolysis of fetal/newborn RBCs takes place.

Result: hemolytic anemia, pathological jaundice

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4
Q

How is Rh incompatibility prevented?

A

Rhogam

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5
Q

What does Rhogam do?

A

Suppresses mother’s normal immune response. The mother’s cells no longer recognize fetal cells as foreign and no antibody formation occurs.

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6
Q

How long does Rhogam last?

A

12 weeks

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7
Q

Coombs Test

A

Identifies antibodies to Rh+ blood. It is interpreted as positive or negative.

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8
Q

Direct Coombs

A

Done on baby. Identifies antibodies to Rh+ blood in the neonate’s serum.

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9
Q

Indirect Coombs

A

Done on momma. Identifies antibodies to Rh+ blood in maternal serum.

Negative: no antibodies. given rhogam.

Positive: antibodies. No rhogam (too late). The fetus is at risk for hemolytic anemia and pathological jaundice.

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10
Q

When is indirect coombs testing performed?

A

Indirect coombs testing is performed on all Rh- women at about 28 weeks’ gestation

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11
Q

When is direct coombs testing performed?

A

postpartum, on a neonate with a Rh + blood sample.

Negative: mother recieves Rhogam within 72 hours to prevent antibody formation to protect future pregnancies.

Positive: Infant has been exposed to maternal antibodies. Monitor infant for jaundice. Treat infant for hemolytic anemia (transfusion)

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12
Q

When does ABO incompatibility occur?

A

Type O mother, Type A, B or AB fetus

Mother may have naturally occurring antibodies to blood type A or B, so sensitation does not necessarily have to be from fetal/maternal blood crossing.

Antibodies hemolyze type A and/or type B RBCs. Typically mild.

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13
Q

Effects of ABO incompatibility on the newborn and fetus

A

Fetus: No effects

Newborn: Potential hemolytic anemia, potential jaundice

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14
Q

How would you manage ABO incompatibility postpartum?

A

If mother is blood type O: Determine newborn blood type and perform direct coombs to detect antibodies to both blood type and Rh.

Coombs Positive: Monitor infant for jaundice, anticipate treatment for possible pathologic jaundice and/or hemolytic anemia (HA is rare)

Negative: Infant not at risk. No interventions necessary.

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15
Q

PP Hemorrhage Vaginal Birth

A

greater that 500mL blood loss

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16
Q

PP Hemorrhage Cesarean Birth

A

greater that 1000mL blood loss

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17
Q

Other criterion for PP Hemorrhage

A

Decrease in HCT of 10% or more from admission

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18
Q

Early postpartum hemorrhage

A

Criteria are met within 24 hours after birth

19
Q

Late postpartum hemorrhage

A

Criteria are met after the first 24 hours

20
Q

Cause of early postpartum hemorrhage

A

Uterine atony associated with “boggy” fundus

Predisposing factors: uterine overdistention, multiparity, rapid labor, prolonged labor, macrosomia, rapid labor, assisted birth

Other causes: trauma, lacerations (perineum, vagina, cervix), hematoma (vagina, perineum)

21
Q

Prevention and Treatment of early postpartum hemorrhage

A

Prevention: Identification of risk factors

Treatment: Fundal massage, IV fluid replacement, oxytocic medications, foley catheter (inserted for pressure from inside), treat underlying cause, monitor blood loss (weigh pads)

22
Q

Causes of late postpartum hemorrhage

A

Uterine subinvolution

Other: retained placental fragments

23
Q

Management of late postpartum hemorrhage

A

Outpatient: surgical removal of placental fragments

24
Q

What is a puerperal infection?

A

Bacterial infection during the six weeks following birth

25
Q

What are the most common puerperal infections?

A
  1. Endometritis
  2. Urinary Tract
  3. Mastitis
26
Q

What is endometritis?

A

Infection of the endometrium

27
Q

What are the risk factors for endometritis?

A
  • Prolonged rupture of membranes
  • Prolonged labor
  • Multiple vaginal exams
  • Trauma of the perineum, vagina, or cervix
  • Catheterization
  • Retained placental fragments
  • Cesarean birth
  • Concurrent problems (ie. diabetes)
28
Q

What assessment data would you find consistent with endometritis?

A
  • Uterine tenderness
  • Chills and malaise
  • Abdominal cramping
  • Fever of 38 degrees or higher after the first 24 hours
  • Foul smelling lochia
  • Uterine subinvolution
  • Elevated WBCs
29
Q

What would you do for endometritis?

A
  • Place the woman in fowler’s position
  • Analgesics
  • IV antibiotics
  • Non-pharmacological comfort measures
  • Assistance with infant care
30
Q

What are the risk factors for urinary tract infection?

A
  • Trauma (pressure on urethra from presenting part)
  • Catheterization
  • Stasis/reflux (regional anesthesia, pressure on bladder from presenting part)
31
Q

What assessment data would you find for urinary tract infection?

A
  • Dysuria
  • Frequency
  • Suprapubic pain
  • Low grade temperature
  • Signs of ascending infection
    • Flank pain
    • Elevated temperature
32
Q

How would you manage a urinary tract infection?

A
  • Antibiotics
  • Analgesics
  • Encourage frequent voiding
  • Reinforce perineal hygiene
  • Encourage increased fluid intake
  • Discharge teaching
    • Signs/symptoms of worsening urinary infection
33
Q

What are the risk factors for mastitis?

A
  • Poor hand washing
  • Newborn oral contact (staph aureus)
  • Milk stasis
    • Infrequent breastfeeding
    • Engorgement
    • Clogged duct
34
Q

What assessment data would you find with mastitis?

A
  • Flu like symptoms
  • Temperature of 38 degrees or greater
  • Localized pain over effected breast area
  • Warmth and erythema of affected breast
35
Q

How would you manage mastitis?

A
  • Frequent breast feeding
  • Monitor breast feeding technique
  • Antibiotics
  • Analgesics
  • Non pharmacologic comfort measures
  • Assistance with newborn care
36
Q

What are the risk factors from thromboembolitic disorders?

A
  • Compression of large veins of lower extemities during pregnancy
  • Venous stasis
  • Postpartum hypercoagulation
37
Q

What assessment data would you find with superficial thrombophlebitis?

A
  • Localized tenderness
  • Swelling
  • Warmth
  • Erythema
  • Homan’s negative
38
Q

How would you manage superficial thrombophlebitis?

A
  • Ambulation as tolerated
  • Analgesics
  • Moist heat
  • Elevation of limb
  • Anticoagulation not indicated
39
Q

What assessment data would you find with deep vein thrombosis?

A
  • Positive Homan’s sign (pain)
  • Extemity cool to palpation
  • Edema of effected extremity
  • Diminished peripheral pulses in affected area
40
Q

How would you manage deep vein thrombosis?

A
  • Doppler flow studies
  • Bedrest
  • Analgesics
  • IV anticoagulation
  • Monitor for pulmonary embolism
    • Dyspnea
    • Shortness of breath
    • Decreased oxygen status
41
Q

Define puerperal infection

A

Infection of the reproductive tract up to six weeks postpartum

42
Q

Define subinvolution

A

Failure of a part to return to its normal size after functional enlargement

43
Q

Define mastitis

A

Inflammation of the breast ocurring primarily in lactating women

44
Q

Describe the rationale for weighing perineal pads postpartum

A

1 gram = one mL of blood loss