Lymphatics/Wound Care Flashcards

1
Q

Primary lymphedema

A

abnormal development of lymphatic system - from birth

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2
Q

Secondary lymphedema

A

Trauma
Surgery
Breast cancer
Radiation
Filariasis

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3
Q

Mechanical debridement

A

nonselective removal of necrotic or infected tissue (along with good viable tissue) with irrigation, wet to dry dressing or debridement pads

nonselective

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4
Q

Autolytic debridement

A

Auto = self

moisture retaining dressing - uses body natural fluids and enzymes
best for noninfected wounds

selective

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5
Q

Enzymatic debridement

A

using chemicals or enzymes such as an ointment or gel to soften bad tissue

selective

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6
Q

Biological debridement

A

maggots

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7
Q

Serous fluid

A

clear to yellow thin fluid,
no blood

this is a normal exudate for a healing wound - seen in inflammatory and proliferative stages

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8
Q

Serosanguinous

A

thin and watery, pink in color

this is a normal exudate for a healing wound - seen in inflammatory and proliferative stages

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9
Q

Sanguineous

A

fresh blood that is typically produced from deep wounds during the inflammatory stage of wound healing - may be brown if dried

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10
Q

Purulent

A

yellow, thick pus, odor
indicates infection

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11
Q

Sarcoidosis

A

african americans>caucasion

skin or eye lesions present
upon XRAY - diffuse pulmonary infiltration along with bilateral hilar adenopathy

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12
Q

Rule of nines adult

A

ANTERIOR ONLY
Head 4.5%
Trunk 18%
- 9% for upper half
- 9% for lower
UE 4.5% each
LE 9% each
Genitals 1% total

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13
Q

Rule of nines pediatric

A

ANTERIOR ONLY
Head 9%
Trunk 18%
UE 4.5% each
Legs 7% each

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14
Q

Wagner Ulcer Classification scale

A

Specific to diabetic foot ulcers

Grade 0 - no open lesion or healed ulcer
Grade 1 - superficial ulcer
Grade 2 - deep ulcer involving subcutaneous tissue and may expose bone or tendon
Grade 3 - deep ulcer with osteomyelitis
Grade 4 - gangrene of digit
Grade 5 - gangrene of entire foot

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15
Q

How is Wagner ulcer system different than pressure ulcer staging?

A

Wagner is specific for diabetic foot ulcers

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16
Q

Pressure ulcer staging

A

Stage 1 - intact skin but nonblanchable erythema
pain and sensation intact
Stage 2 - partial thickness affecting epidermis and dermis
presents with blistering
Stage 3- full thickness with loss of subcutaneous tissue
Stage 4 - full thickness with exposed tendon, bone, muscle

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17
Q

Pre albumin vs albumin

A

pre albumin = short term nutrition (2 days)
albumin = long term (20 days)

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18
Q

Normal pre albumin levels

A

20-40

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19
Q

What pre-albumin level would indicate malnutrition

A

less than 15

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20
Q

Normal albumin level

A

3.5-5.5

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21
Q

What Albumin level would indicate malnutrition

A

less than 3.5

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22
Q

Primary intention

A

Surgical closure via sutures, staples, glue, etc

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23
Q

Secondary intention

A

secondary = self

would closure through natural healing

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24
Q

Tertiary intention

A

delayed primary union

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25
Indolent ulcer
ulcer that is slow to heal, non painful
26
How do first degree wounds heal?
through regeneration/epithelialization
27
What is the purpose of providing a moist wound environment?
a moist wound bed is healthier and epithelizes better than a dry wound
28
Factors that delay wound healing
increased age - decreased metabolism and needed growth factors for healing impaired oxygenation/circulation poor nutrition comorbidities infection
29
SInus tracts
tunneling wounds that communicate to deeper structures such as fat or muscle
30
Dermatitis is also known as
ezcema
31
Dermatitis
inflammation of the skin (can be caused by allergic reaction to chemicals/soaps, poison ivy, adhesives, etc) causes itching, redness, scaling
32
Advice for dermatiits
apply lotions within 5 minutes of showering know triggers
33
impetigo
superficial skin infection producing small pus filled vesicles MRSA, staph
34
Cellulitis
inflammation of the skin and underlying tissue skin is warm, red, signs of infection
35
Treatment for cellulitis
antibiotics elevation cool and wet dressings
36
Herpes simplex (1)
itching and soreness vesicular eruption on face or mouth ex. cold sore
37
Herpes 2
genital herpes sexual contact
38
Tinea corporis
ringworm fungal infection of hair skin nails spread by direct contact
39
Tinea pedis
atheletes foot treated with antifungal meds/creams can progress to bacterial infection or cellulitis if untreated
40
Psoriasis most commonly affects which joints
small distal joints
41
Psoriasis
chronic autoimmune disorder where scaly silver plaques cover the skin
42
Most common area for psoriasis
ears, elbows, scalps, knees, genitals
43
Precipitating factors of psoriasis
trauma infection pregnancy smoking cold weather
44
Discoid lupus
affects only the skin flare-ups with sun exposure
45
Systemic lupus
affects multiple body systems skin heart kidneys nervous system
46
Long term side effects/conditions of corticosteroids
weight gain, acne, HTN, bruising, osteoporosis, diabetes, myopathy
47
Scleroderma
autoimmune disease of connective tissues causing fibrosis of skin, joints, blood vessels, internal organs (lungs heart)
48
Scleroderma is usually associated with what symptoms
arthralgias (joint pain) malaise/fatigue raynauds phenomenon HA weight loss anemia photosensitivity
49
Population: lupus
15-40 year old women
50
Most common cause of death with lupus
kidney failure
51
Stages of scar healing
bright pink (6-12 weeks) lavender (12-15 months) white, flat, soft (2 years)
52
What should a scar look like at 6-12 weeks
bright pink
53
What should a scar look like at 12-15 months
lavender
54
What should a scar look like at 2 years
white, soft, flat
55
What should PTs ensure to assess with patients with scleroderma
regularly assess vital signs and blood pressure at risk for acute HTN
56
Scleroderma - CREST
Calcification Raynauds Espohageal dysfunction Sclerodactyl (skin damage of fingers) Telangiectasia (Spider veins)
57
Zone of coagulation
most cell damage, irreverible damage
58
Zone of stasis
cells injured but can be "revived" with specialized tx CSH coag Stasis Hypermedia
59
Zone of hyperemia
minimal cell injury, recovery
60
1st degree burn
redness and tenderness, painful only epidermis is damaged heals in 3-7 days
61
1st degree burn healing times
heals in 3-7 days
62
2nd degree burn (superficial partial thickness) healing times
7-21 days
63
2nd degree burns (superficial partial thickness)
all of epidermis, partial dermis painful - light touch intact bright red or pink blanching with brisk refill fluid loss and blistering minimal scarring
64
2nd degree burns (deep partial thickness)
red or white waxy mod pain severe damage to dermis and epidermis blanching with slow refill sensitive to pressure but lacks light touch or pin prick 21-28 days
65
2nd degree (deep partial thickness) burns healing times
21-28 days
66
3rd degree burns
damaged all the way to subcutaneous tissue, muscle and tendons - risk for contracture no blanching painless, dry, leathery severe fluid loss and infection risk grey, white or eschar appearance will heal with a keloid scar
67
Hypertrophic scar vs keloid scar
Hypertrophic Scar: raised scar that stays within boundaries of burn wound Keloid Scar: raised scar that extends beyond boundaries of original burn wound
68
blanching with slow refill =
2nd degree (deep partial thickness)
69
blanching with quick refill =
2nd degree (superficial partial thickness)
70
sensitive to pressure but lacks light touch or pin prick =
2nd degree (deep partial thickness)
71
Location of arterial ulcers
lateral malleolus lower 1/3 of leg dorsal foot and toes
72
Appearance/symptoms arterial ulcers
smooth, deep, with punched-out edges minimal exudate absent pulses no edema thin and shiny skin with hair loss
73
Leg elevation increases pain
arterial ulcer
74
Venous ulcers location
proximal to medial malleolus
75
Appearance/symptoms venous ulcers
irregular, shallow wound normal pulses moderate to heavy exudate mild pain edema hemosiderin staining
76
Pulse scale
0- absent 1+ thready, weak 2+ normal 3+ increased 4+ full, bounding
77
Braden scale
used for determining pressure sore risk 15-18 mild 13-15 mod 10-12 high 9 or less severe
78
High risk Braden Scale
Rated out of 23 12 or less is serious risk
79
Hydrogel
increases moisture in dry wound bed
80
Most occlusive moisture retention dressing
hydrocolloid
81
Best used for infected wounds with exudate
alginates
82
Indications for autolytic debridement
natural debridement under semi-occlusive or occlusive dressing use for all necrotic wounds in medically stable individuals
83
Contraindications for autolytic debridement
infected wounds - do not want to trap in infection dry gangrenous wounds
84
Enzymatic debridement indications
Topical application of enzymes to promote liquefication of necrotic tissue Moist necrotic wounds people who are homebound
85
Enzymatic debridement contraindications
clean granulated wounds ischemic wounds
86
Indications for mechanical debridement
ex. wet to dry, pulsed lavage, wound vac wounds with foreign material
87
Contraindications for mechanical debridement
clean granulated wounds
88
Indications for sharps debridement
scoring or excision of necrotic tissue
89
Contraindications for sharps debridement
clean wounds people on anticoagulant therapy
90
Surgical debridement indications
when life is threatened with infection or sepsis
91
Minimally absorbent dressings
hydrogels transparent films gauze
92
Maximally absorbent dressings
alginates foams hydrocolloids (mod to max)
93
Minimally occlusive dressings
Gauze Alginates Hydrogels
94
Maximally occlusive dressings
hydrocolloids transparent films foams
95
Wound care options for arterial ulcers
hydrogel
96
Wound care options for arterial ulcers
hydrogel transparent fild
97
Advantages of transparent films
clear to visualize wounds promote autolytic debridement impermeable to water and bacteria
98
Disadvantages of transparent films
nonabsorptive wrinkling, hard to apply not for infected wounds can rip fragile skin
99
Indications for transparent films
autolytic debridement secondary dressing stage 1-2 pressure ulcers w/ no infection
100
Advantages of hydrocolloid
maintain moist wound environment supports autolytic debridement mod absorption waterproof/occlusive
101
Disadvantages of hydrocolloid
nontransparent not good for infections not good for lots of drainage
102
Indications hydrocolloids
protection of partial thickness wounds moderate exudating wound autolytic debridement
103
Advantages of hydrogel
Rehydrate dry wounds promote autolytic debridement nonadherent can be used on infected wounds
104
Disadvantages of hydrogel
require secondary dressing not for exudating wounds if dry out, could adhere to wound bed
105
Indications for foams
partial to full thickness wounds min to mod exudate protection/insualtion
106
Disadvantages foams
not used for dry eschar or no exudate wounds require secondary dressing and tape
107
Anteiror neck burn contracture
into flexion position in hyperextension with cervical collar
108
Shoulder burn contracture
adduction and IR position into abduction and ER (airplane splint)
109
Elbow burn contracture
flexion and pronation position into extension and supination
110
Hand burn contracture
flexion and adduction position into wrist extension, MCP flexion, IP extension (resting pan splint)
111
Knee burn contracture
flexion position into extension with posterior knee brace
112
Foot burn contracture
plantar flexion position into DF
113
Treatment compression for CVI
20-55 mmHg
114
CDT Phase 1
intensive, MLD + skin care + bandaging (23 hours) bandaging basically gets the limb as small as possible to prepare for compression garment at end of phase (20-40 mmHg) - worn 24 hours/day
115
Bandaging for lymph
short stretch bandage (Comprilan, rosidal) high working pressure low resting pressure
116
Exercise in phase 1 CDT
low impact, waling/cycling/swimming, Tai Chi
117
Phase 2 CDT
self management phase skin care + compression garment day, bandage at night
118
Phase 2 CDT you wear what during the day
compression garment
119
Phase 2 CDT you wear what during the night
bandage
120
Pressures more than ... are CONTRAINDICATED for lymphedema
45
121
Describe phases of CDT
Phase 1 - intensive Phase 2 - self management
122
Centrifugal vs centripetal
fugal = toward feet petal - toward heart