Lifespan A: Antisocial behaviour in Childhood, WEEK 10 Flashcards

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1
Q

Antisocial behaviour

A
  • ASB are normative in development but differ from each other so some are more harmful than others (arguing isn’t as harmful as physically fighting someone) + are more/less frequent (e.g. fight once or fight often for a long time)
  • ASB include excessive verbal and physical aggression, lying, stealing, disobedience, rule breaking and violence.
  • Suggests ASB in on a continuum (from not much ASB to high amounts of ASB)
  • ASB can be overt (explicit) or covert (secretive), can be reactive (impulsive) or proactive (premeditated) and can be relational (relation to other people like arguing) or physical like destroying property
  • Conduct problems is a preclinical manifestation > only becomes clinically important when it affects people around them + ed > child only gets classified w/ conduct problems if engage in harmful beh which affects different contexts in their lives
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2
Q

Conduct disorder

A
  • Angry, irritable mood, argumentative and defiant behaviour, vindictiveness for at least 6 months
  • Persistent pattern of behaviour in which basic rights of others or major age-appropriate norms or rules are violated over at least 6 months.
  • Aggression to people or animals (e.g., bullying, fighting, threatening, using weapons, cruelty).
  • Destruction of property (e.g., vandalism, fire)
  • Deceitfulness or theft
  • Serious rule violations (e.g., running away, truancy)
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3
Q

Callous-Unemotional Traits

A
  • 2 sub-types of conduct problems w/ children w/ conduct problems vary in callous + unemotional traits
  • Callous + unemotional traits include lack of guilt or remorse, a lack of concern for others, shallow emotions and a lack of concern for performance
  • There are people w/ conduct problems w/ no callousness + unemotional traits > these are reactive, explosive + emotional people
  • Other type have a calm demeanour + don’t appear emotional > callousness + unemotional traits
  • CU is characterised by instrumental aggression > aggression designed to satisfy the need of the child
  • CU is characterised by premeditated way of thinking + not reactive > can be found as young as 3yrs + has stability so can continue into adulthood > has a genetic component
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4
Q

Epidemiology of Conduct Disorder

A
  • Not all ASB is problematic + research looking at trajectories of aggression show normative increase in ABS in young children
  • CD occurs in 5-8% of pop > more common in boys
  • 50% of parents report ABS in 12mo + rises to 70% when they are 2-3
  • Typically levels of ABS decrease + trail off around 4yrs > but subgroup of children whose ABS continues
  • Onset of ABS means it may have different cause:
    1. Early onset life course persistent > child shows extreme conduct problems early on + are more likely to persist over time
    2. Adolescent onset > only happens in adolescence + trails off after
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5
Q

Social information processing

A
  • Dodge argues people w/ high ASB + conduct problems think differently about social world
    1. Knowledge Structures: Hostile world view, Defensive goals, Aggressive repertoire > this underlying schema means you will encode info differently about the world > leads to inaccurate processing + inappropriate reactions to situations > scheme are affected by biological + EV factors
    2. Encoding of cues from environment: incomplete + hypervigilant
    3. Representation of Encoded cues: have difficulty understanding intentions of others + likely to assume -ve intent + struggle taking others perspectives
    4. Response Selection and Evaluation: Poor judgment of outcomes > due to above problems, likely to respond poorly
    5. This manifests as aggression or ASB
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6
Q

Social information processing: encoding differences

A
  • Evidence supports that children w/ conduct disorders perceive things differently
  • Viding et Al compared 3 groups of children (high cu, low cu + TD) while they were doing a task in FMRI > task was to sit in FMRI scanner + were shown faces + asked if it was male or female > the faces shown had fearful or neutral exp
  • Results show amygdala activated differently in ppt when they saw the faces > when shown fearful faces, people w/ conduct problems + high callous + unemotional traits showed under activation of amygdala > under-active response to fear
  • Those w/ low CU traits showed over active response to fear > may see fearful face as threatening
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7
Q

Social information processing: Representation difference

A
  • Not much evidence that there is a difference in understanding others beliefs + thoughts (ToM)
  • If you show emotional faces + ask them to label it, they get confused between sad + fearful faces > people w/ conduct problems likely to think they had malicious intentions when typical people would see it as an accident > perceive hostility when there isn’t any
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8
Q

Executive Dysfunction

A
  • ASB in children may be due to executive dysfunction
  • Resemblance between conduct disorder and antisocial symptoms of patients with frontal lobe injuries > not as severe
  • ED can be characterised as difficulty inhibiting emotions + controlling behaviour, impaired ability to anticipate consequences + difficulty generating alternative responses
  • Frontal lobe dysfunction may lead to ED
  • Problem w/ this theory is that it isn’t specific to ASB > children w/ autism have ED so why do they not all have ASB?
  • Meta analysis on different between TD and CD kids on EF measures show consistent difference
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9
Q

Genes & Environment

A
  • Longitudinal study on people w/ ASB on 17,000 twins > looked at ASB overtime + compared MZ twins to DZ twins to see how strongly associated they were in terms of their ASB
  • 43%-50% of variance in ASB is explained by genes > stable genetic affect (doesn’t change)
  • In children, the common EV effect is just as strong as genetic effect but impact of family EV reduced in adolescence + adulthood while non shared EV impact increases + explains half of variance in ASB
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10
Q

Role of parenting in ASB

A
  • Schemas (knowledge structures) are shaped by early experiences that nurture hostile attributions:
    1. Harsh control (i.e., physical or verbal punishment) 2.Psychological control (i.e., attempts to manipulate child through guilt, shame and conditional love)
  • Exposure to harsh parenting might cause children to develop these schema which give a hostile world view + see world as threatening + hostile place
  • Harsh parenting (r= .21) + psychological control (r= .22) is associated w/ conduct problems but these are weak to modest correlations
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11
Q

Adoption studies: passive gene-environment correlation

A
  • It is assumed harsh parenting is an EV factor but it is possible that if child + parent are biologically related, it could be due to shared genetic factors
  • Harsh parenting may be a form of conduct problems > perhaps the same genes that cause harsh parenting cause ASB > passive gene-environment correlation > not a environmental impact but a genetic transmission of ASB between generations
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12
Q

Evidence for passive gene-environment correlation

A
  • Can test if this is EV or genetic by comparing adopted + non-adopted samples
  • If there is an association between harsh parenting + conduct problems in the related + unrelated sample > suggests an EV effect as adopted sample aren’t biologically related
  • But if there is only an association between harsh parenting + CD in related samples > this suggests a passive rGE > due to genes
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13
Q

Adoption studies: evocative gene-environment

A
  • Harsh parenting doesn’t necessarily cause conduct problems in children > conduct problems in children may evoke harsh parenting > child-driven effect > Child can elicit behaviour in their parents
  • Child may have an underlying genetic propensity towards aggression or ASB > this underlying genetic factor causes harsh parenting in parent
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14
Q

Evidence for evocative gene-environment

A
  • Looked at adopted children + adoptive parents > shared EV > shared EV
  • But also found biological parent who didn’t know adoptive parents + only spent a few weeks/months w/ child at beginning of life
  • Interviewed biological parent + assessed them > found if biological parent had history of mental health problems or ASB > their child was more likely to have it
  • Equally, adoptive parents were more likely to be harsh if biological parent had history (having never met them)
  • Shows an association between biological parent MH + ASB + adoptive parent’s harshness > there is an evocative rGE
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15
Q

Nature and Nurture in conduct problems

A
  • Nature + nurture work together to cause conduct problems in a coercive cycle of parenting
  • Child is born w/ genetic risk towards ASB but negative interactions w/ parent evoke ASB causing conduct problem or reinforces it
  • Got families w/ children diagnosed w/ conduct problems > didn’t treat conduct problem but trained parent’s response to be less harsh
  • When harsh parenting reduced so did the level of conduct problems
  • Even if there is a genetic propensity, modification of EV reduced conduct problems
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16
Q

Causal model of developmental disorders

A
  • Two way direction so biological factors may influence children’s thinking which causes ASD but these behaviours could evoke certain environmental responses which in turn reinforce the behaviours at a cognitive level > some kind of cycle rather than a directional association