Lecture 7 - stress Flashcards

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1
Q

reaction-focused models

A

based on reaction of the human body to unspecific situations
- Cannon (1914)
- Selye (1956)

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2
Q

Cannon 1914

A

the body is in a state of homeostasis
- stimulus –> short-term effect (fight or flight)

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3
Q

Selye, 1956

A

general adaptation syndrome (long-term consequences)
o Alarm phase: immediate response to stressor –> sympathetic nervous system
o Resistance phase: effort to manage the stressor –> glucocorticoids
o Exhausting phase: exhaustion of physiological resources –> impairment of health

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4
Q

stimulus-focused models

A

stress is provoked by external stimuli

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5
Q

cognitive-transactional models

A

stress is a cognitive transaction between internal and external demands. It explains individual differenced and is based on a evaluative (appraisal) process.

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6
Q

CTM primary appraisal

A

nterpretation of stressor (what is at stake?)

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7
Q

CTM secondary appraisal

A

analysis of resources ( do I have the capacities to successfully deal with the stressor

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8
Q

CTM coping

A

overcoming stress
o Problem-focused: change the situation
o Emotion-focused: change your relationship to the situation

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9
Q

CTM re-appraisal

A

pacing and learning (are my attempts to reduce stress successful?)

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10
Q

physiological stress response

A
  • sympathetic activation
  • HPA activation
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11
Q

sympathetic activation

A

Cerebral cortex: classification of event as harmful
- Hypothalamus: early response of sympathetic nervous system arousal (fight or flight) –> stimulates medulla of adrenal glands –> secretes catecholamines, epinephrine, norepinephrine (cranked-up feelings)
- Leads to: increased heartrate, increased sweating, contraction of peripheral blood vessels, enhanced blood flow to muscles

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12
Q

HPA activation

A

Hypothalamus releases CRF –> stimulates pituitary gland –> ACTH –> stimulates adrenal cortex to release glucocorticoids –> cortisol
 Conserves stores of carbohydrates
 Reduces inflammation
 Heps the body to return to steady state
 Chronic high cortisol –> chronic stress –> higher risk for civilization diseases (CHD, mental disorders)
- Elevates growth hormone (prolactin) levels and beta-endorphin and enkephalin (responsible for immune-related disorders

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13
Q

stress and negative health outcomes

A

Russ et al (2012): analysis of relationship between stress and mortality
- Mortality risk twice as high for people with high stress scores
- Stress elicits strong physiological responses –> negative consequences for health

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14
Q

relationships between pa and stress

A
  • physical activity reduces stress
  • stress reduces physical activity
  • physical activity <–> stress
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15
Q

3 types of studies

A
  • Cross-sectional studies: high levels of pa are associated with lower perceived stress
  • Prospective studies: baseline measures of physical activity can predict stress at follow-up
  • Longitudinal studies: reciprocal (<–>) relationship between pa and stress
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16
Q

Aldana 1996

A

-Relation between leisure time pa and perceived stress
-N= 32.229 (big sample)
-Low amount of pa –> 0.78-0.62 times less likely to have less stress (1.0 at no pa)
-Moderate pa –>0.5 –> half the rate of perceived stress
-Causality: what influences what? Stress < – >pa
- Other studies: stress leads to reduction in pa

17
Q

crossed-lagged panel design

A

a study of the relationships between two or more variables across time in which one variable measured at an earlier point in time is examined with regard to a second variable measured at a later point in time, and vice versa. (to look at causality)

18
Q

Luz 2007

A

Perceived stress predicted pa negatively, not the other way around

19
Q

stress buffer hypothesis

A

pa buffers the negative effects of stress on health
-Stress has bad negative consequences on health, pa reduces these negative effect
-Pa reduces reactivity towards stressors (HR, BP, cortisol)
-Pa gives more rapid recovery of stress parameters (cardiovascular and endocrine)
- cross-stressor response

20
Q

cross stressor response

A

Pa functions as a stressor and trains the neurophysiological stress activity system, the stress systems adapts and therefor responds less strong to that and other stressors

21
Q

Sothmann 1996

A
  • cross-stressor response
  • Conclusion: trained men reacted less strong (physiological and self-reports) to psychological stress compared to untrained men
22
Q

O’Dougherty 2012

A

negative effects of critical life events on depressive symptoms are buffered by aerobic training

23
Q

TSST

A
  • trier social stress test
  • naturalistic exposure to a socio-evaluative stressful situation
  • 5 min public speaking task
  • 5 min calculations in front of others
  • 3 min preparation
24
Q

EMA

A

ecological momentary assessment
- Method of assessing intense and chronic stressors in everyday settings
- Assessment of self-report questionnaires (stress and well-being) and physiological parameters (ECG, accelerometer) at fixed timepoints or at certain events using a smartphone

25
Q

AA

A

ambulatory assessment
- Monitors movement, physiological functions, contextual information (GPS) and EMA in realtime

26
Q

causes of stress during pa

A
  • Physiological factors (intense training)
  • Environmental factors (training conditions)
  • Social factors (social relationships)
  • Psychological factors (expectations)
27
Q

Fletcher and Fletcher 2005

A

most important stressors in sport
- Stressors during or right before competition (fear, nervousness, competition density, previous failure)
- Organizational stressors (communication, media, travel)
- Chronic stress (constant high-performance pressure, financial uncertainty)
- Social stress (problems at home)
- Transition phases (junior –> senior, end of career)