JVD 2018 deck Flashcards
According to “Congenital feline hypothyroidism w partially erupted adult dentition in a 10mo MN DSH case report” by Jacobson, Rochette, what are some clinical signs of congenital hypothyroidism in cats?
shorter stature, rounded body, enlarged broad heads/face, short neck, delayed eruption of dentition, delayed closure of ossification centers, lethargy, mental dullness, constipation/difficulty passing feces, palpable goiter
According to “Congenital feline hypothyroidism w partially erupted adult dentition in a 10mo MN DSH case report” by Jacobson, Rochette, this kitten had thickened fibrous gingiva covering partially erupted adult dentition (all deciduous had exfoliated). What other findings were seen on dental rads and other radiographs taken?
open apices of 309/409 (should be closed by 7mo), epiphyseal dysgenesis (delayed closure of ossification centers), scalloped ventral borders of vertebral bodies
According to “Congenital feline hypothyroidism w partially erupted adult dentition in a 10mo MN DSH case report” by Jacobson, Rochette, what blood work tests were performed to diagnose hypothyroidism? what was the treatment and outcome?
total T4, FT4, and TSH to help r/o euthyroid sick syndrome (T4 low, FT4 low, TSH high); tx w supplemental thyroid hormone and ginvivectomy/plasty to allow continued eruption; kitten became clinically normal by 22mo; side note: there is no feline TSH assay so canine was run
According to “Congenital feline hypothyroidism w partially erupted adult dentition in a 10mo MN DSH case report” by Jacobson, Rochette, in this case, what was thought to be preventing eruption of adult teeth?
mechanical barrier of fibrous overgrown gingiva (no pocketing, tightly adhered to teeth); thyroid supplementation allowed for continued development of teeth but may not have fixed lack of eruption due to mechanical barrier; thyroid hormones potentiate effect of growth hormones and are necessary for skeletal bone and tooth development
According to “Crown reduction and vital pulp therapy in a dog w malocclusion” by Blanchard, Koehm, what malocclusion did the dog have? What previous tx had been performed and why?
Class II with MAL I/LV 304, 404 with palatal trauma and trauma from mand incisors; interceptive orthodontics: previous XSS 704, 804 to allow for continued jaw growth and stop adverse/unfavorable dental interlock that was impeding jaw growth
According to “Crown reduction and vital pulp therapy in a dog w malocclusion” by Blanchard, Koehm, what were possible tx options discussed for this malocclusion? what type of cement was used? What are its benefits?
Extraction 304/404, orthodontic mvmt 304/404 (inclined plane), CR/XP and VPT 304/404 w odontoplasty of mand incisors; used MTA (mineral trioxide aggregate) bc antibacterial, biocompatible, induces dentinal bridge, promotes bone and PDL growth formation
According to “Crown reduction and vital pulp therapy in a dog w malocclusion” by Blanchard, Koehm, what % phosphoric acid is acid etch and what is its purpose? Why seal odontoplasty teeth?
37% (usually), removes smear layer, dissolves hydroxyapatite crystals and exposes dentinal tubules to allow for micro mechanical interlock with dentinal bonding agents; odontoplasty teeth removes enamel and exposes dentin, sealant allows time for tertiary dentin to form and prevent bacterial leaching into tubules
According to “Dental pathology of Iberian Lynx part I: congenital, developmental, and traumatic abnormalities” by Collados, Garcia, Rice, 88 post mortem skulls were evaluated grossly and with dental rads. What is the dental formula of the Iberian lynx? Is this similar to other wild cats? What is the deciduous formula?
28 teeth= I3/3, C1/1, P2/2, M1/1; no second premolar as in domestic cats shift towards reduced dentition similar to caracal, leopard and Canadian lynx; I3/3, C1/1, P2/2= 24 teeth
According to “Dental pathology of Iberian Lynx part I: congenital, developmental, and traumatic abnormalities” by Collados, Garcia, Rice, what are some noteable changes to the mandibular first molar present in 83% of cases? How was age estimated?
poorly developed cingulum and no metaconid (more distal of the lingual cusps of mammalian molar tooth); gross exam, radiographs, and cementum annuli analysis with first complete cementum annuli around 18mo of age
According to “Dental pathology of Iberian Lynx part I: congenital, developmental, and traumatic abnormalities” by Collados, Garcia, Rice, what was the most common dental pathology noted? what was the second most common dental lesion?
attrition/abrasion 90.9% of specimens affected (43.7% of teeth examined) split into 3 types based on severity; tooth fractures (68% of specimens, 11.3% of teeth), affecting primarily canines and most often CCF or CCRF
According to “Dental pathology of Iberian Lynx part I: congenital, developmental, and traumatic abnormalities” by Collados, Garcia, Rice, what was endodontic dz typically caused by? How frequent was intrinsic staining of teeth?
CCF/CCRF or attrition/abrasion type 3 (pulp exp) mostly affecting canines; 8.5% of teeth in primarily canines
According to “Dental pathology of Iberian Lynx part I: congenital, developmental, and traumatic abnormalities” by Collados, Garcia, Rice, what percent of teeth were suspected to have artifactual defects?
2.3%; most missing teeth were lost through artifact (81%) with only 0.4% congenitally absent
According to “Dental pathology of Iberian Lynx part II: perio, TR, oral neoplasia” by Collados, Garcia, Rice, what was the limiting factor to staging of perio? How common was perio in this study pop? IN domestic cats?
post mortem bony examination only w rads (no gingivitis or perio pocketing for AL); 81.3% of teeth with 64.8% of specimens having at least 1 tooth affected by stage 3/4 dz, 63.1% affected by stage 2 with most common tooth affected by stage 3/4 maxillary and mandibular incisors (59.4%); in domestic cats >80% over age of 2y
According to “Dental pathology of Iberian Lynx part II: perio, TR, oral neoplasia” by Collados, Garcia, Rice, how prevalent was TR? was this finding consistent w domestic cats? how many cats had neoplasia?
uncommon, affecting 0.63% of teeth in 6.8% of skulls; lower than that of domestic cats (by far) and that of other wild felids; only 1 case of suspect neoplasia (no biopsy obtained)
According to “Management and outcome of maxillofacial trauma in a 9 week old dog” by Castejon-Gonzalez, Buelow, Reiter, what are the most common causes of maxillofacial trauma in dogs? what groups of dogs are over represented?
surprisingly did not reference Soukup/Snyder articles: HBC, dog fight; young dogs <1y, small breeds, and males;
According to “Management and outcome of maxillofacial trauma in a 9 week old dog” by Castejon-Gonzalez, Buelow, Reiter, what factors should be considered when determining a type of jaw fracture management? what are some types of fixation?
patient age, fracture type and location, presence and condition of teeth for anchorage, jaw bone quality, available methods for stabilization, operator skill; Noninvasive: tape muzzles, labial reverse suture through buttons, BEARD, inter arch splinting, interdental wiring and splinting; Invasive: intraosseous wiring, ex-fix, bone plating
According to “Management and outcome of maxillofacial trauma in a 9 week old dog” by Castejon-Gonzalez, Buelow, Reiter, what are complications of maxillofacial trauma even following fixation? what were some limitations in the type of fixation chosen given the patient’s signalment?
dental injuries/loss of vitality, malocclusion, palatal defects, osteomyelitis, bone sequestrum, delayed or malunion bone fx, facial deformities, delayed or abnormal dental eruption; patient age (9wk) jaws still growing, incomplete permanent dentition, location of developing tooth buds (interfere w wire/screws), plates inhibit jaw growth, splints not possible due to presence of deciduous teeth therefore, tape muzzle was most appropriate choice with monitoring for permanent dentition
According to “Management and outcome of maxillofacial trauma in a 9 week old dog” by Castejon-Gonzalez, Buelow, Reiter, what were specific long-term complications told to the owner given the left mandibular non favorable fx, maxillary fractures including TMJ fracture?
abnormal development of upper and lower jaws causing malocclusion, malunion fx, abnormal tooth development including EH, abnormal crown/root shape, infection, pulpitis, pulp necrosis, abnormal eruption, TMJ pain, ankylosis, DJD of TMJ
According to “Management and outcome of maxillofacial trauma in a 9 week old dog” by Castejon-Gonzalez, Buelow, Reiter, at 2 weeks a bony callous was forming. Where does bony deposition occur up to 50d? >50d? an asymmetric malocclusion (side-to-side and rostrocaudal) was noted. Is there additional fixation that could have been used to help avoid this?
rostral bone deposition of the mandible during first 50d after birth, continued growth via bone formation at caudal aspect of mandibles; in addition to tape muzzle could have added inter fragmentary stabilization with absorbable suture, will dissolve in 3 weeks and should not interfere w mandibular jaw growth
According to “Management and outcome of maxillofacial trauma in a 9 week old dog” by Castejon-Gonzalez, Buelow, Reiter, what are the goals of management of maxillofacial fx? why was TMJ surgical correction not recommended?
restore function, non painful occlusion, avoidance of dental, vascular, and nerve damage during repair, long-term establish healthy dentition with normal eruption; minimally displaced fx, more damage to correct via condylectomy, should be reserved if TMJ ankylosis or DJD develops
According to “Validation of quantitative light-induced fluorescent for quantifying calculus on dogs’ teeth” by Wallis, Allsopp, Colyer, what are the two main methods to quantify plaque and calculus on dogs’ teeth? which historically has the best results?
plaque indices (humans brush off plaque then do total scoring by hand) and planimetry (for plaque, dye then measured by computer analysis software w QLF; For plaque QLF repeatable, reproducible, and accurate, first study for calculus
According to “Validation of quantitative light-induced fluorescent for quantifying calculus on dogs’ teeth” by Wallis, Allsopp, Colyer, what two plaque quantification models were used for this calculus study? how was the study performeD?
QLF and modified Logan and Boyce using VOHC teeth: buccal maxillary third incisor, canine, third/fourth premolar, first molar, mandibular buccal third/fourth premolar and first molar; randomized crossover study with 26 schnauzers with 7d brushing following prophy, then 1 test group w dental chew, one w just standard RC hard kibble diet, then test, then switch groups.
According to “Validation of quantitative light-induced fluorescent for quantifying calculus on dogs’ teeth” by Wallis, Allsopp, Colyer, what was measured to test calculus quantity?
% coverage, thickness, coverage of 3 areas of tooth summed within each tooth as “coverage” and each of 3 areas scored per tooth coveragexthickness (Warrick-Gorrel) for overall tooth score for Logan Boyce; % coverage via avg percent across 18 teeth or “average mouth” and total calculus of teeth divided by total area of all teeth multiplied by 100 for overall percentage of calculus or “weighted mouth”
According to “Validation of quantitative light-induced fluorescent for quantifying calculus on dogs’ teeth” by Wallis, Allsopp, Colyer, what were the results of each test method?
Dental chews led to 43.8% reduction of calculus using Warrick-Gorrel method (coveragexthickness from Logan, Boyce) and 65.8% reduction of calculus via QLF (average mouth)