JVD 2010 deck Flashcards
According to “Building a telescoping inclined plane” by Legendre, what does is added to the traditional acrylic IP?
An 18g and 21g needle with the hubs cut off as a cross bar across the palate covered in acrylic/protemp, then an orthodontic wire around the maxillary canine and third incisor for a base of the IP
According to “Surgical extractions for PD in a western lowland gorilla” by Huff, what teeth were extracted from the 30y male gorilla? what may have been a contributing factor to his PD?
28, 29, 30, 31, 32 all PD 4 with buccoversion of 30; low pH from chronic regurge.
According to “Modified distal wedge excision for access and Tx of an infra bony pocket in a dog” by Klima, Goldstein, how was the infra bony pocket of 309 treated in a 6y Dachshund?
Staged procedure to extract 310 with PD3-4 then return for perio surgery of 309 in 6 weeks; surgery entailed a modified wedge resection along the alveolar crest, exposure to the site, and placement of a bone graft with osteoconductive, synthetic bioactive ceramic, then sutured closed and recheck radiographs in 6mo
According to “Modified distal wedge excision for access and Tx of an infra bony pocket in a dog” by Klima, Goldstein, why was this called bone augmentation not GTR? what # of walled defect was this? what time of bone graft was used? what suture? could things have been done better per the author?
no membrane was placed; 3 walled; osteoconductive biogloss (alloplast); 4-0 chromic gut; could have used membrane for GTR, autograft is best, and could have used 4/5-0 monocryl as chromic gut only lasts 14d and is extremely inflammatory/irritating as it is absorbed by phagocytosis not hydrolysis
According to “Modified distal wedge excision for access and Tx of an infra bony pocket in a dog” by Klima, Goldstein, what did the wedge resection look like?
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According to “Modified distal wedge excision for access and Tx of an infra bony pocket in a dog” by Klima, Goldstein, what was the outcome? what is circumferential bone loss around a tooth called? what is the “col”?
complete resolution of clinical signs… no histo so unsure if regeneration or repair but on rads normalizing PDL space; cupping defect, osseous crater; co is the interdental valley of non-keratinized tissue between two closely positioned teeth (site of inflammation and bacterial accumulation)
According to “Modified distal wedge excision for access and Tx of an infra bony pocket in a dog” by Klima, Goldstein, define allograft and alloplast. what is the purpose of a membrane?
allograft from same species, mineralized freeze-dried bone allograft (FDBA) and decalcified freeze-dried bone graft (DFBDA), osteoconductive, osteoinductive; alloplast osteoconductive only (no inductive or genic properties), synthetic material including plaster of Paris, calcium carbonates tricalcium phosphate, hydroxyapatite, and bioglass (used here), promotes repair, not necessarily regeneration; prevent downgrowht of epithelium and healing w long junctional epithelium and CT, goal to get regeneration not repair
According to “Open root planing for a PD pocket of a maxillary canine tooth” by Greenfield, a 10y MN Dachshund presents for a COHAT. Define this term. What did they find on oral exam? What was the tx recommendation?
comprehensive oral health assessment and treatment; Stage 3 PD palatal 104, Stage 2 304, 404, TR 205; recommended X 205, RPO and placement of bone graft (alloplast) palatal 104, RPC and doxirobe of 304, 404, followed by daily tooth brushing and COHAT q6mo w rads to monitor
According to “Open root planing for a PD pocket of a maxillary canine tooth” by Greenfield, what # walled defect was palatal 104? what are most common sites for this type of defect in the dog? what percentage of doxycycline gel was used? what was the tx outcome in 6mo?
3 walled; btwn mandibular first and second molars and palatal maxillary canine; standard 8.5%; complete healing of 104 pocket (3mm) and 304/404 (2mm)
According to “Open root planing for a PD pocket of a maxillary canine tooth” by Greenfield, what are some of the mechanisms in which periodontal bacteria evade host defenses and cause tissue destruction? what are some mediators produced that are part of the host response to tissue destruction? what mediator is partly responsible for bone loss? bone loss in PD is not necrosis, explain this.
IgA and IgG degrading proteases degrade specific antibodies, leukotoxins that inhibit PMNS, heat sensitive surface proteins that lead to apoptosis of PMNs and inhibit IL-8 production of epithelial cells causing impairment of PMN response to bacteria; proteinases (MMPs, elastase, cathepsin G, neutrophil serine proteinase), cytokines (IL1, TNFa) prostaglandins; PGE2 (remember orthodontic mvmt too); not necrosis but activity of living cells along bone, as soft tissue destruction occurs, exposes root and subsequent bone resorption occurs, tissue necrosis and purulent d/c is from the soft tissues, not bone, there is a normal turnover of periodontium by bacetierla proteinases and mediators and inhibition of host cells, in diseased state it is no longer in balance
According to “Open root planing for a PD pocket of a maxillary canine tooth” by Greenfield, what are the 2 types of pocket? what are some mentioned root surface applications mentioned? why not use them? what is the goal of this type of perio surgery with bone augmentation?
gingival/pseudopocket and periodontal pocket; root conditioners, etc to improve attachment/CT: citric acid (low pH causes damage to soft tissue), EDTA (not approved in vet texts), fibronectin and tetracycline (as well as citric acid) have no literature proving improved attachment over root planing alone; new attachment with periodontal regeneration
According to “Open root planing for a PD pocket of a maxillary canine tooth” by Greenfield, what happens if gingiva populates root surface first? bone? what is the difference btwn osteogenesis and osteoinductive? what are some examples of osteoconductive only grafts? what was used in this case report?
gingiva first leads to root resorption; bone first leads to root resorption or ankylosis; goal is to have periodontal tissues (PDL, cementum) repopulate first); osteogenesis has blasts within the graft, only autografts can do this, while osteoinductive means the product will aid in generation of new bone via BMPs converting neighboring cells into blasts to make new bone (DFBDA); FDBA (not decalcified), hydroxyapatite, tricalcium phosphate, bioactive glass; SBGP: synthetic bioactive graft particulate
According to “Open root planing for a PD pocket of a maxillary canine tooth” by Greenfield, how long does doxirobe last? what are its properties?
2-4 wks to aid in healing of perio tissues; antimicrobial, anti-collagenase, stimulates fibroblast activity
According to “A survey of equine oral pathology” by Anthony, Laycock, et al, 556 cadavers were examined. What were the most common findings? where did the majority of pathology occur? were there associations between the noted pathology?
sharp edges 48%, buccal abrasions, calculus 30%, lingual ulcers, gingival recession, periodontal pockets, ramps 15%, and waves 13% (in order); cheek teeth (only 28% did not had cheek tooth pathology); horses w sharp edges were 100x more likely to have buccal abrasions, 3.6x more likely to have lingual ulcers, and 2.3x more likely to have calculus than horses w/o sharp edges, lingual ulcers were 3.2x more likely to occur in horses w buccal abrasions, PP were 21x more likely to occur w gingival recession, etc.
According to “A survey of equine oral pathology” by Anthony, Laycock, et al, in regards to canine teeth and wolf teeth, were they typically present or absent? in all horses?
60% were missing all four canine teeth, more common in geldings and stallions; most horses did not have wolf teeth, those that did were maxillary (26%)
According to “A survey of equine oral pathology” by Anthony, Laycock, et al, were older or younger horses more likely to have normal cheek teeth? sharp enamel points were most associated with what 2 pathologies? what were PP associated with?
younger; buccal abrasions and lingual ulcers; most commonly seen in cheek teeth and closely associated w diastema, missing teeth and gingival recession
According to “Scanning electron microscopy of pulp cavity dentin in dogs” by Hernandez, Saccomanno et al, 36 teeth from 12 adult cadaver dogs 2.5-13y were taken from 104, third premolar, 409 and looked at radicular and coronal dentin to determine tubule density and diameter. They were split in 2 groups I (<7y), 2 (>7y). Was there significant difference between the 2 groups? what shape were most of the dentinal tubules?
no. round or oval.
According to “Scanning electron microscopy of pulp cavity dentin in dogs” by Hernandez, Saccomanno et al, what type of media was used to store teeth and clean teeth to limit shrinkage and dehydration? was there a decrease in # of tubules within the tooth, if so where? was tubule density influenced by age or occlusal function?
first bleach (2.5 -5%), then 100% acetone, best to maintain hardness and have least volume reduction; yes, decrease in # of tubules per mmsq moving from the pulp cavity towards enamel (90K at pulp, 24K at DEJ); also a decrease in diameter and area of tubules in a corona-ical direction and decreased as age increased; No.
Abstract: According to “Clinical periodontal and microbiologic parameters in patents with acute myocardial infarction (AMI)” by Stein, Conrads, et al, what were the find gins in this study?
104 patients (54AMI, 50 healthy control) sub gingival plaque analyzed for red pathogens and others, p w AMI had singiciantly higher frequency of probing depths than controls, all pathogens overrepresented by AMI p and positively correlated w increased PD AL; Porphyromonas gingivalis was an indicator for AMI. association btwn perio and AMI in PD correlated to presence of PD pathogens.
According to “Effect of kibble size, shape, and additives on plaque in cats” by Clarke, Biouge et al, 40 mixed breed cats 1.5-7y were placed into 4 groups following 14d dry kibble only diet A for acclimation and dental charting and blood work. then a dental prophylaxis was performed following by 7d acclimation period on diet A. Then, cats were evaluated under GA, plaque scored and teeth were cleaned again, at that time, they were split into 4 groups. What were the 4 groups of kibble diet? Once diet was initiated 7d later plaque score was performed and again at 28d post diet change.
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According to “Effect of kibble size, shape, and additives on plaque in cats” by Clarke, Biouge et al, which diets contained STPP? PRN? Which were larger/rectangular?
Diet B and C had STPP (sodium triphosphate); Diet D had PRN; diet C/D were larger
According to “Effect of kibble size, shape, and additives on plaque in cats” by Clarke, Biouge et al, what was measured? was there any significant difference between cats fed diet A and B on day 7 or 28? what was the theory as to why cats fed diet with STPP did not change their plaque score?
individual coverage and thickness scores from gingival half of tooth (gingival half score) and a total mouth plaque score, mean gingival score, and coronal half plaque score; no; bc sodium triphosphate do not reduce plaque accumulation but are mineral chelators and mineralization inhibitors that bind salivary calcium helping to reduce the formation of calculus
According to “Effect of kibble size, shape, and additives on plaque in cats” by Clarke, Biouge et al, between diet C and D, which had better results? why was C better than A/B? Why was D better than A, B, and C at reducing plaque?
Diet D had a more significant reduction in plaque (43% less than A/B at day 28); C was a larger, harder kibble with greater SA and thickness providing more mechanical debridement; D contained not only STPP to prevent calculus formation, but was larger (like C) and contained PRN
According to “Effect of kibble size, shape, and additives on plaque in cats” by Clarke, Biouge et al, what is PRN? how does it work? Was Diet D larger or smaller than C? what is most important for reduction of plaque?
sodium ascorbic phosphate group (ascorbic acid- Vit C), water soluble vitamin promotes wound healing, helps maintain normal CT, and aids in promotion of healthy teeth and gums; ascorbic acid promotes collagen synthetic CT protein at the level of hydroxylation of propyl and lysol residues of pro collagen, has a direct effect on gingival inflammation, bleeding, risk of PD, and amount of visible plaque; D had slightly less SA and volume than C, but contained PRN which improved its efficacy (D had 12% less plaque than C at day 28); size and texture of kibble for mechanical debridement and addition of PRN may be useful