Ischaemia, Aneurysm , Varicose Veins Flashcards

1
Q

What is intermittent claudification?

A

Lack of O2 on exercise to muscle leading to cramp like pain

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2
Q

Clinical Signs of intermittent claudification?

A

Pain upon walking further steeper faster, but pain is quickly relived upon rest
Ankle brachial pressure under between 0.4-0.85

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3
Q

Treatment of intermittent claudification?

A

No cure for peripheral arterial disease
Slow progression on prevention
Stop smoking, more exercise, lipid lowering
Bypass need healthy inflow and outflow
Endovascular Stent treats only the symptoms

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4
Q

Why does walking more despite the pain improve intermittent claudification?

A

Because more pain means more anastomoses form increasing blood flow to the area

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5
Q

What is critical limb ischaemia

A

Pain occurs in the foot even at rest, particularly bad lying down and sleeping

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6
Q

Signs of severe ischaemia?

A

Cool to touch, absence of peripheral pulses, hairless, thick nails, shiny skin, venous guttering

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7
Q

How does critical limb ischaemia lead to limb loss?

A

Injury, ulcer gangrene amputation

Life or Limb

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8
Q

What is Varicose Veins

A

Dilated tortuous superficial veins due to abnormal transmission of pressure within deep veins

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9
Q

What three thing result in VV?

A

Increased venous pressure, damaged valves, deep vein obstruction

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10
Q

Causes of VV

A

Pregnancy , old age , obesity, females,

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11
Q

Risks related to VV

A

Bleeding- significant and recurrent elevation and pressure
thrombophlebitis - inflammation due to clot sore bruising scaring
Haemosidium deposits- Black dots under the skin red cell leakage out of vessel iron broken down
Liperdermatosclerosis- intense inflammatory response leads to scaring fibrosis- like cellulitis
Venous ulceration

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12
Q

Management of VV

A

Compression bandages, low APBI no bandages as blood completely prevented from getting to foot

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13
Q

Foam Sclerotherapay

A

Chemical treatment of VV fuses veins together

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14
Q

Endogenous ablation

A

Physical thermal radiowaves all damage endothelium of vein closing it off

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15
Q

Why is surgery not really used for VV

A

Are risks to patient and was more for cosmetic reasons , higher success rate endovenous

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16
Q

What is an Abdominal Aortic Aneurysm

A

Aorta has been dilated by more than 50%

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17
Q

Two types of Aneurysm?

A

True- vessel walls are intact

False- Blood contained by the surrounding soft tissue

18
Q

Three shapes of aneurysm

A

Sacular- simple sack on aorta
Fusiform- bulges out on both sides
Mycotic- due to infection

19
Q

What causes an AAA

A

Medial degeneration- elastin collagen decrease, increased pressure, increased wall stress

20
Q

Risk factors for an AAA

A

Old male Smoker Hypertension

21
Q

Are most AAA symptomatic

A

No 75% are asymptomatic

22
Q

Symptoms of an AAA

A

Pain can mimic renal colic, trashing

23
Q

What symptoms follow a rupture

A

tearing sudden epigastric pain radiating to the back, sudden collapse

24
Q

Is an free intraperitoneal worse than a retroeritoneal rupture

A

Yes as its not contained by anything rapidly bleed out and die

25
Q

When is an aneurysm deemed serious enough to deal with

A

If over 5.5cm or rapidly growing

26
Q

Two options for treating an AAA

A

Open surgery- more risky iliac dacron graft used as stent

Endovascular- required good seal before and after AAA

27
Q

Acute limb ischaemia

A

Sudden loss of blood to the foot

28
Q

Causes of Acute limb ischaemia

A

Trauma embolism arterial dissection, compression

29
Q

Signs of acute limb ischaemia

A
6 P's
Pallor 
Pulsless
Perishingly cold
Paraesthesia- pins and needle
Pain
Paralysis
30
Q

0-4 hours in ALI

A

White foot painful sesorimotor effect- reversible

31
Q

4-12 hours in ALI

A

White with mottled blanches on pressure- partly reversible

32
Q

12h+ hours in ALI

A

Fixed mottling non blanching, improving perfusion is dangerous non reversible could lead to harmful systemic effects due to chemical released by necrosis

33
Q

Management of ALI

A

ABCDE, Blood tests, ECG CXR

Anticoagulants, prevent thrombus formation, increase perfusion

34
Q

Diabetic Foot Sepsis

A

Pressure as a result of infection results in lack of perfusion to the foot

35
Q

Three things required for diabetic foot sepsis to occur?

A

Diabetic neuropathy, peripheral vascular disease, infection

36
Q

Common sources that lead to DFS

A

Pressure ulcers- don’t feel them forming due to neuropathy
Ingrown toe nail
puncture wound

37
Q

Why does pressure build due to infection?

A

Muscles are confined by fascia and bones into rigid compartments, inflammation due to infection results in obstruction of blood vessels

38
Q

Clinical signs of DFS

A

Pyrexia, tachypnoea , tachycardia, confused , kussmauls breathing- laboured
Swollen and Boggy, tender to touch, ulcers with puss, erythema tracking up leg, woody necrosis patches, crepitus from microbes releasing gas, pedal pulse is lacking

39
Q

Is the infection usually mononclonal?

A

No its usually multicultural gram -ve and +ve

40
Q

Treatment for DFS

A

Remove all infected tissue leave open to encourage drainage