interactionist approach Flashcards

1
Q

What is the interactionist approach in explaining schizophrenia?

A

The interactionist approach acknowledges that both biological and psychological factors contribute to the development of schizophrenia.

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2
Q

What does the diathesis-stress model propose?

A

The diathesis-stress model proposes that schizophrenia develops due to an interaction between a genetic vulnerability (diathesis) and environmental stressors.

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3
Q

What is meant by ‘diathesis’ in the context of the diathesis-stress model?

A

‘Diathesis’ refers to a pre-existing vulnerability to schizophrenia often genetic in nature.

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4
Q

What role do environmental stressors play in the diathesis-stress model?

A

Environmental stressors act as triggers that can activate the underlying vulnerability leading to the onset of schizophrenia.

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5
Q

Give an example of a potential environmental stressor for schizophrenia.

A

Examples include childhood trauma significant life events

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6
Q

What is the modern understanding of diathesis in the diathesis-stress model?

A

Modern research suggests diathesis may include factors beyond genetics such as early neurological damage.

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7
Q

How does the diathesis-stress model inform treatment approaches for schizophrenia?

A

It supports the use of a combination of treatments such as antipsychotic medication to address biological factors and cognitive behavioural therapy (CBT) to manage psychological stressors.

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8
Q

Why is the interactionist approach considered beneficial for schizophrenia treatment?

A

It allows for more comprehensive and personalised treatment by addressing both biological and psychological aspects of the disorder.

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9
Q

What evidence supports the diathesis-stress model?

A

Research shows that individuals with a genetic predisposition to schizophrenia are more likely to develop the disorder if exposed to significant stressors.

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10
Q

How does the diathesis-stress model explain why some people with genetic vulnerability do not develop schizophrenia?

A

It suggests that without the presence of significant environmental stressors the disorder may not manifest despite the genetic vulnerability.

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