Hiv Aids Flashcards

1
Q

Who is hiv found among

A
Iv drug users
Hemophiliac 
Blood transfusion receipients 
Infants born to infected moms
High risk heterosexuals
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Hiv causes

A

Aids

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is hiv positive

A

Virus is present but few if any clinical signs are present

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What is aids

A

Active infection with marked and multiple clinical manifestations

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Does HIV do

A

Replicates within living cells and uses the cells replication enzymes to make copies of itself
Enzyme then converts RNA into DNA before cell begins viral replication

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What does hiv primarily effect

A

Cd4 cells which is an immune system cell

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What does hiv do in early stages

A

Virus invades and multiplies in lymphoid tissue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What does Dna do

A

Long term sore age and transmission of genetic info

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

How does it enter the body

A

Through blood system (sexual contact, blood to blood, perinatal lay)
Through body fluids

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

How to diagnose it

A

Blood tests

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is seroconversion

A

Process of moving from negative hiv antibodies in blood to positive hiv antibodies

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What are seroconvertors

A

Those that test hiv positive

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is phase 1

A

Primary infection phase
Happens weeks after exposure
Many asymptomatic
Symptoms: low fever, fatigue, sore throat, malagia, night sweats, go problem, maculopapular rash, headache
Tx: decrease number of cells through antiretroviral therapy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Pahse two

A

Latency phase
No symptoms or sign of illness
Median time is 10 years
Lymphadenopathy develops in some (chronically swollen lymph gland)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Phase 3

A

Without antiretroviral therapy death occurs
Determined by number of cd4 cells
Development of opportunistic infections due to weakened immune system

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What are typical progressions

A

60-70% with Hiv develop aids in 10-11 years after infection

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What are rapid progressors

A

10-20% develops it in less than 5 years

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

What are slow progressors

A

5-15 don’t develop for 15 years or more

19
Q

What is pcp

A

Pneumocystis carinii pneumonia

Mild cough, fever, sob, weight loss, tachypenea,

20
Q

What is tb

A

Lungs most common site for infection

Present with fever, night sweats, cough, weight loss

21
Q

Thrush

A

Typically complain of painful swallowing or retrosternal pain
White velvety lesion from Yeast infection and

22
Q

What is cytomegalovirus

A

Presents like mono

Diarrhea, gastroenteritis

23
Q

What is acd

A

Aids dementia complex
Cognitive and motor dysfunction
Only caused by hiv
Impaired attention, concentration, slow mental speed and agility, slow motor speed, apathy

24
Q

Toxoplasmosis

A

Parasite that attacks CNS

Fever headache neuro dysfunction (confusion, lethargy, visual disturbance, seizure)

25
Progressive multimodal leukoencephalopothy
Demyelinating disease of white matter in the brain Progressive limb weakness, sensry problems, difficulty controlling fingers and toes visual disturbance subtle alterations in mental status hemisphere ataxia to floppy seizures
26
Kaposis sarcoma
Malignancy of the endothelial cells | Lesions found in G.I. tract lungs skin and in mouth
27
Non Hodgkin's lymphoma
Fever night sweats weight loss | poor prognosis
28
Noninvasive cervical cancer
Develops more rapidly than women with HIV | less responsive to treatment
29
Wasting syndrome
Aids defining illness | involuntary weight-loss at least 10% of baseline bodyweight in presence of diarrhea or chronic weakness and fever
30
Lipodystrophy
Metabolic disorder changes in body appearance (buffalo hump,wasting of fat from face, increased ab girth) changes and metabolic appearance
31
Non specific gi symptoms
Nausea Vomitting Ab pain
32
Hepatomegaly with normal liver function
Caused by antiretroviral therapy | Can be fatal
33
Psychosocial issues
Complex reactions from others May be indication of hidden lifestyle Feeling of fear guilt isolation etc Previous mental health issues
34
Prevention
Risk free or low risk behaviors Abstinence Correct and consistent condom use Avoid recreational drug
35
Antiviral drugs
Reduce replication of the virus but don't kill it
36
Combo of three Meds
Highly active antiretroviral therapy
37
What is the focus of tx
Minimize the effects of complications such as infections or malignancy
38
Zidovudine therapy
Slows the virus and effects on the body
39
Physical dysfunction
Fatigue sob visual cancer cardiac opportunistic infections pain stroke
40
Mental functions
Depression anxiety | Guilt preoccupied with death angry at disease or God body image issues helpless hopeless
41
Contextual factors
Isolation Physical env challenges Economic impact
42
What to do for intervention
Enhance Competency pain management adaptive equipment energy conservation relaxation stress management
43
What was aids initially called
Grids gay related immunodeficiency syndrome