GI Flashcards
what is the most important part of a GI workup
history: ask about diet and presenting complaint
dysphagia is due to
oral cavity/laryngeal/upper esophageal disease
dysphagia C/S, ddx
trouble eating/grabbing food, difficult swallowing, exaggerated head movements, coughing/aspiration
ddx- severe dental disease, oral masses, neuro disease
vomiting vs regurg
vomiting- active process, nausea, GI material from stomach, indicates GI disease
regurg- passive, no nausea, GI material esophagus, indicates esophageal problem
sm vs lg bowel d+
SI- lg volume, melena, can have fat, v+, weight loss
LI- sm volume, tenesmus, mucous, fresh blood
ddx for anorexia in cats
pretty much anything, hepatic lipidosis, IBD
acute vs chronic GI
acute- self limiting, less diagnostics (unless GI urgency markers)
chronic- lasting more than 2-3wks, defined therapeutic trial/more diagnostics
differentials for GI
dysphagia/regurg- separate
anorexia, v+/d+, weight loss
primary vs secondary GI
therapeutic vs diagnostic tests
therapeutic trial- v+/d+ w no urgency markers, chronic w no progression or weight loss
diagnostics- chronic w/ progresion, dysphagia or regurg
GI urgency markers
unstable patients, weight loss, painful abdomen, low TP, effusion, prolonged anorexia, intractable vomiting
steps to therapeutic trial
- initial problem and urgency
- eliminate simple disease first (dewormer)
- eliminate dietary factors- diet trial
- treat symptoms
- hydration
- further workup if no resolution
GI tx (no urgency markers)
acute v+- MPO trial, GI diet, probiotics, antiemetic, deworm
acute d+- probiotics, GI diet +/- deworm, abx
chronic v+- elimination diet, deworm, probiotics
chronic d+- elimination diet, probiotics, fiber for lg bowel, abx
gi diet vs elimination diets
GI- highly digestible, low fat 1wk diet w transition
elimination- hypo diet to eliminate rxns 6-8wks
abx therapeutic trials
metro 8-12mg/kg q12 3-5d
tylosin 10-15mg/kg q12
fenben- 50mg/kg q24 5d
NPO for acute v+
NPO for 12h-> sm amount water q2h (if v+ do diagnostics)-> sm amount food 2-3d-> reintroduce diet 25% for 1-2d
other therapeutic trial meds
prebiotic, probiotics, anti-emetic, antacid, deworm, sucralfate (antacid and sucralfate probably don’t work)
when to use SQ or IV fluids
<5% under 25kg-> SQ
>5% or shocky-> IVF
normal swallowing stages
oral - prehension
pharyngeal- moving towards esophagus
cricopharyngeal- relaxation of upper esophageal sphincter
gastroesophageal reflux
reflux of gastric acid into esophagus. LES (striated and smooth muscle in dogs, smooth only in cats)
causes: LES incompetence, motility disorder, FB, v+, GERD
prolonged fasting >24h, anesthetic
esophagitis C/S, dx
anorexia, regurg, drooling, coughing, loud vocalization after eating, many are subclinical
dx- hx, C/S, TXR, can do contrast rads/scope
esophagitis tx
PPI- omeprazole 1mg/kg PO q12 for 2d then q24 7-10d, panto if IV
+/- cisapride, sucralfate
prognosis good
esophageal FB signalment, C/S
common in dogs, near LES< base of heart-> mucosal damage, ulcers, perf
acute regurg, gagging, hypersalivation, pain
esophageal FB dx, tx
dx- rads, scope
tx- scope to remove, push into stomach-> gastrotomy?
prognosis good, risk stricture
esophageal strictures
secondary to esophagitis (FB, reflux)
dx- esophagram w contrast, scope
tx- balloon dilation (3tx) can perforate
hiatal hernias
repeated protrusion of abd contents into thorax-> reduced LES tone
congenital- bulldogs, brachys
trauma induced
C/S- regurg, v+, repeated aspiration pneumonia
dx- rads, scope
hiatal hernia tx
medical- PPI, low fat diet, prokinetic
surgical- hernia reduction
PRAA
embryonic R aortic arch becomes aorta
GSD, greyhounds
regurg solid food, underdeveloped puppy, aspiration pneumonia
PRAA dx
contrast rads, fluoroscopy
tx- surgical correction
prognosis fair to poor
megaesophagus causes
congenital- schnauzer, GSD, great dane
acquired- idiopathic, chronic esophagitis, myasthenia gravis
megaesophagus C/S, dx
regurg, weight loss, cough/fever if acute
dx- TXR