General surgery Flashcards

1
Q

Small bowel stoma (ileostomy) location

A

right side more likely

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2
Q

Large bowel stoma (colostomy) location

A

left side more likely

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3
Q

Small bowel stoma contents

A

more likely to be more liquid, green, due to things not being properly digested yet and mixtures of enzymes etc

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4
Q

Large bowel stoma contents

A

more likely to look more like faeces, a lot more water has been absorbed

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5
Q

Inspection of small bowel stoma

A

stoma is everted, spouted a bit, so that enzymes don’t touch skin, ( less time for digestion because is more proximal, would break skin down if it was too close to skin)

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6
Q

Inspection of large bowel stoma

A

stoma can be flat on skin, no enzymes

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7
Q

Rutherfor morrison incision

A

Extraperitoneal approach to left or right lower quadrants
renal transplant

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8
Q

Pfannenstiel incision

A

Transverse supra pubic, primarily used to access pelvic organs
c section etc

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9
Q

Lanz incision

A

Incision in right iliac fossa
appendicitis

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10
Q

Kocher’s incision

A

Incision under right subcostal margin
open cholecystectomy

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11
Q

Rooftop incision

A

more unusual , upper GI surgery eg whipple procedure

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12
Q

volvulus

A
  • twisting of a loop of intestine around its mesenteric attachment, resulting in a closed loop bowel obstruction.
  • The affected bowel can become ischaemic due to a compromised blood supply, rapidly leading to bowel necrosis and perforation.
  • Most volvuli occur at the sigmoid colon and are a common cause of large bowel obstruction
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13
Q

Volvulus Ix

A

Bloods - inc electrolytes, Ca2+, and TFTs to exclude any potential pseudo-obstruction
CT imaging - Very dilated sigmoid colon with a ‘whirl sign’, from the twisting mesentery around its base.
AXR- “coffee-bean sign” arising from the left iliac fossa

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14
Q

Volvolus Mx

A

treated conservatively initially with decompression by sigmoidoscope and insertion of a flatus tube.
Surgery indicated if ischaemia/perforation/failed decompression

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15
Q

Caecal volvulus epidemiology

A

bimodal age of onset
10-29 year group - intestinal malformation or excessive exercise as the predisposing cause
60-79 year group- more associated with chronic constipation, distal obstruction, or dementia.

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16
Q

Caecal volvulus Mx

A

always laparotomy and ileocaecal resection

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17
Q

Strangulated bowel obstruction

A

obstruction with compromised intestinal blood flow, resulting in bowel ischemia

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18
Q

Causes of SBO

A

Adhesion
Hernia
Intussusception in children

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19
Q

Causes of LBO

A

Malignancy
Diverticular disease
Volvulus

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20
Q

Bowel obstruction pathophysiology

A
  1. bowel segment occluded,
  2. gross dilatation of the proximal limb of bowel occurs, resulting in an increased peristalsis of the bowel.
  3. This leads to secretion of large volumes of electrolyte-rich fluid into the bowel (often termed ‘third spacing’). Leads to dehydration and hypovolemia.
  4. Compression of intestinal veins and lymphatics leads to bowel wall edema leading to compression of intestinal arterioles and capillaries leading to bowel ischemia.
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21
Q

SBO symptoms

A

early vomiting, pain,l ack of flatulence or passing stool

22
Q

LBO symptoms

A

early distension, pain, constipation, feculent vomiting is a late symptom,lack of flatulence or passing stool

23
Q

Bowel obstruction signs

A

Evidence of the underlying cause eg surgical scars
Dehydration and possible hypovolemia
Percussion may reveal a tympanic sound
auscultation may reveal high pitched ‘tinkling’ bowel sounds when it is early , absent bowel sounds when late

24
Q

Small bowel on AXR

A

Valvulae conniventes are mucosal folds that form lines that extend the full width of the small bowel.

25
Large bowel on AXR
Haustra are indents that go Halfway
26
Bowel obstruction on AXR
Upper limits of normal are: 3 cm small bowel, 6 cm colon, 9 cm caecum. Distended loops of bowel proximal to obstruction Dilated bowel values are 5, 8, 10 In SBO, The dilated loops are predominantly central. In LBO (esp. distal LBO): The dilated loops are predominantly peripheral. CT gold standard though
27
Blood Ix for obstruction
FBC (Looking for leukocytosis or anaemia) CRP U&Es (Organ dysfunction or signs of hypovolaemia) LFTs Amylase Group and Save (G&S) VBG to evaluate the signs of ischaemia (high lactate) or for the immediate assessment of any metabolic derangement (secondary to dehydration or excessive vomiting)
28
Bowel obstruction Mx
“drip and suck” Nil by mouth, bowel rest, NG tube on free drainage IV fluids, correction of electrolyte imbalance Urinary catheter and fluid balance IV analgesics ( avoid opioids if possible) Antiemetics Broad spectrum antibiotics given preoperatively Patients with closed loop bowel obstruction or evidence of ischaemia need urgent surgery
29
SBO due to adhesions Mx
1. treated conservatively i 2. water soluble contrast study should be performed in cases that do not resolve within 24 hours 3. surgery
30
Anastomotic leak
contents of a hollow organ that were joined surgically, leaks through a fault in the join
31
Anastimotic leak clinical presentation
typically occur 5-7 days post surgery failure to thrive, a persistent low grade fever, prolonged ileus, abdominal pain, delirium, tachycardic, and / or with signs of peritonism. check for any faeculent / purulent material or bile in any drains.
32
Anastomotic leak Ix
ix for sepsis, lactate for tissue ischaemia A CT abdomen and pelvis with rectal contrast fluoroscopic water-soluble enema.
33
Anastomotic leak Mx
small leak can be managed conservatively with bowel rest, IV fluids and antibiotics larger leaks require emergency laparascopic exploration with potential further surgery.
34
Abdominal wound dehiscence Mx
Coverage of the wound with saline impregnated gauze (on the ward) IV broad-spectrum antibiotics eg IV ceftriaxone and metronidazole Analgesia IV fluids Arrangements made for a return to theatre
35
Clinical presentation of bowel ischemia
sudden onset, severe pain Hematochezia/melaena Nausea, Diarrhoea fever
36
Bowel ischaemia signs
Abdominal tenderness Weight loss in chronic mesenteric ischemia Abdominal bruit
37
Bowel ischaemia risk factors
Cardiovascular disease risk factors Hypercoagulable states Atrial fibrillation Myocardial infarction Structural heart defects Vasculitis Constipation Shock + congestive HF ( exacerbates low flow state) Long term laxative use Colonic carcinoma Use of vasopressors, digitalis, cocaine
38
Bowel ischaemia Ix
FBC- leukocytosis, anaemia UE - renal dysfunction and dehydration Amylase - rises late LFTs ABG /lactate low bicarbonate points to a metabolic acidosis coag ECG - arrhythmias or acute infarction that may be the aetiology
39
Bowel ischaemia imaging
CXR - perforation AXR- air-fluid levels or bowel dilation,thumb-printing sign CT gold standard first line for acute ischaemia Sigmoidoscopy or colonoscopy - diagnosis of colonic ischaemia
40
Bowel ischaemia Mx
Resuscitation and supportive measures ( O2, fluids, NBM,NG, correct electrolytes etc) Abx presence of infarction, perforation, or peritonitis warrants urgent exploratory laparotomy or laparoscopy
41
Ischaemic colitis
describes an acute but transient compromise in the blood flow to the large bowel. This may lead to inflammation, ulceration and haemorrhage
42
Superior mesenteric artery supplies
distal duodenum, jejunum,ileum and the right colon from the cecum to the splenic flexure
43
Inferior mesenteric artery supplies
the left colon from the splenic flexure to the rectum
44
Ischemic colitis likely location
more likely to occur in 'watershed' areas such as the splenic flexure
45
Aetiology of ischaemic colitis
Usually caused by transient hypoperfusion Thromboembolism Hypotension, hypovolaemia ( eg sepsis, dehydration, hemorrhage) Cardiovascular surgery ( especially aortic repairs or cardiac bypass) Vasoconstrictive drugs Thrombophilias ( eg antiphospholipid syndrome) Colonic obstruction from eg tumours, adhesions
46
acute mesenteric ischaemia
cute inadequate blood flow to the small intestine (arterial or venous) that can result in bowel infarction
47
Acute mesenteric ischaemia dx
CT scan with IV contrast
48
Acute mesenteric ischaemia Mx
surgical emergency IV fluids, a catheter inserted, and a fluid balance chart started broad-spectrum antibiotics Excision of necrotic or non-viable bowel/Revascularization of the bowel, involving removal of any thrombus or embolism via radiological intervention
49
Chronic mesenteric ischemia
caused by a reduced blood supply to the bowel which gradually deteriorates over time as a result of atherosclerosis in the coeliac trunk, superior mesenteric artery (SMA), and/or inferior mesenteric artery (IMA).
50
Chronic mesenteric ischemia clinical presentation
Postprandial pain - 10mins-4hrs after eating , Dull, epigastric Weight loss – a combination of decreased calorie intake and malabsorption Concurrent vascular co-morbidities Change in bowel habit
51
Chronic mesenteric ischemia diagnostic test
CT angiography is the diagnostic test of choice
52
Chronic mesenteric ischemia Mx
conservative - modify risk factors, especially smoking cessation, and commencing antiplatelet and statin therapy to minimise disease progression. Surgery- both endovascular and open procedures are options