Acute care Flashcards
Glasgow Coma Score (GCS)
eyes
E 4 Eyes open, spontaneous
E 3 Eyes open to voice/speech
E 2 Eyes open to pain
E 1 Eyes do not open
Glasgow Coma Score (GCS) voice
V 5 Orientated
V 4 Confused
V 3 Inappropriate Words/speech
V 2 Incomprehensible sounds
V 1 No sound
Glasgow Coma Score (GCS)
motor
M 6 Obeys commands
M 5 Localises to pain
M 4 withdrawal from pain
M 3 Abnormal flexion to pain (decorticate)
M 2 Extends to pain (decerebrate)
M 1 No response
patient is unable to maintain their own airway adequately at GCS
8 or below
Parkland formula can be used to calculate the amount of fluids required for an individual according to their weight.
4 x weight (kg) x %burn = ml fluid required in the first 24 hours from the time of the burn
Half of the total fluid calculated should be given in the first eight hours following the burn. The second half should be given in the subsequent 16 hours (depending on urine output) .
Referral to a burns unit should be made if
All burns ≥ 2% TBSA in children or ≥ 3% TBSA in adults
All full-thickness burns
All circumferential burns
Any burn not healed in 2 weeks
Any burn with suspicion of non-accidental injury (should be referred to burn unit / centre for assessment within 24 hours)
Red flags for sepsis
new deterioration in GCS/AVPU
Systolic BP <90, OR 40 mmHg below normal
Heart rate >130 p min
RR >25 p min
Need oxygen to keep SPO2 92%, 88% in COPD
Non blanching rash or mottled/ashen/cyanotic skin
Not passed urine in last 18h
Urine output less than 0.5 ml/kg/her if catheterised
recent chemo within last 6 weeks
Sepsis six
Give oxygen
Take blood cultures
Give iv antibiotics
Give iv fluids
Take lactate
Take/ monitor hourly urine output
Anaphylaxis adult adrenaline dose
Adult adrenaline dose: 500micrograms IM (0.5mL) 1:1000
Children 6-12 years anaphylaxis adrenaline dose
300 microgramsIM (0.3mL) 1:1000
Children less than 6 years anaphylaxis adrenaline dose
150 micrograms im (0.15 ml) 1:1000
Canadian C-spine Rules
high risk (age >65, dangerous mechanism or paranesthesia),
no c spine imobilisation if:
low risk (delayed neck pain, simple rear-end RTA, sitting position in ED, nil midline tenderness of C-spine, ambulatory at any time) and able to rotate neck (45degrees L and R)
Cushing’s triad refers to a set of signs that are indicative of increased intracranial pressure (ICP)
bradycardia, hypertension, and irregular breathing.
Cauda equina key features
lower motor neurone signs and symptoms
reduced lower limb sensation (often bilateral)
bladder or bowel dysfunction ( constipation/retention or incontinence)
lower limb motor weakness
severe back pain
impotence
saddle anaesthesia
Cord compression key features
surgical emergency with a similar pathophysiology to CES, however is characterised by upper motor neurone signs
Difference between metastatic spinal cord compression and cauda equina
MSCC is where a cancer or metastasis presses on and subsequently compresses the spinal cord.
Compression can also occur due to compromise of spinal stability due to vertebral metastases and compression from associated fractures.
Cauda equina syndrome is the same process but occurring at or below the level of the cauda equina (typically at the level of L1)
Both usually presents with pain and weakness and should be treated the same way
Emergency initial treatment for STEMI/NSTEMI
MMONAC
Morphine
Metoclopramide
Oxygen (if sats <94%)
Nitrates (GTN spray)
Aspirin 300 mg
Clopidogrel 300 mg (Although, in practice, other similar drugs such as Ticagrelor are increasingly being used)
Contraindications to thrombolysis in MI
Aortic Dissection
GI bleed
Allergic reaction
Iatrogenic: recent surgery
Neurological disease: recent stroke (within 3 months), malignancy
Severe HTN (>200/120)
Trauma, including recent CPR
Hypovolaemic shock
Inadequate circulating volume secondary to fluid loss
Causes:
Haemorrhagic ( eg trauma, GI bleeding, obstetric haemorrhage, ruptured AAA)
D & V
DKA
Burns
Distributive shock
Inadequate perfusion secondary to maldistribution
Causes:
Sepsis
Neurogenic shock
Anaphylactic
Obstructive shock
Inadequate cardiac output as a result of mechanical obstruction
Causes:
PE
Tension pneumothorax
Cardiac tamponade (prevents heart from filling adequately)
Acute IVC or SVC obstruction
Cardiogenic shock
Inadequate cardiac output as a result of cardiac failure
Causes:
MI
Myocardial contusion
Myocarditis
Late sepsis
Overdose ( beta blockers)
Complete heart block
Clinical signs hypovolaemic shock
peripheral vasoconstriction an increase in diastolic pressure, increased capillary refill time,
Hypotension and tachycardia
Weak thready pulse with cool pale moist skin