final exam med surg 2 Flashcards

1
Q

Hourly urine output

A

30 mL/hr

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2
Q

Sodium

A

135-145

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3
Q

Potassium

A

3.5-5

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4
Q

Magnesium

A

1.6-2.6

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5
Q

Types of isotonic IV solutions

A

Lactated ringers, normal saline

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6
Q

Types of hypotonic solutions

A

1/2 NS

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7
Q

Hypertonic solutions

A

3-5% NS, D5 and D10 NS

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8
Q

What does hypertonic IV solutions cause in the cells?

A

water leaves the cells to try to balance out solutes in the blood (they shrivel)

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9
Q

What does hypotonic IV solutions cause in the cells?

A

Water enters the cell to try to balance the solutes (swells)

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10
Q

What is the sodium-potassium pump?

A

Located on the cell membrane; moves sodium into the cell from ECF and potassium from cell to ECF

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11
Q

Types of sensible (countable) fluid outputs

A

Urine, feces, vomit

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12
Q

What is ADH?

A

antidiuretic hormone; helps body retain fluid-if the body is dehydrated, more ADH will be produced; if the body is in fluid overload, less ADH will be produced

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13
Q

Renin-angiotensin system and what it does

A

Kidneys release renin-liver releases angiotensin. angiotensin and renin= angiotensin 1- to the lungs, which creates angiotensin 2-causes the blood vessels to constrict. Nephrons retain sodium and water and the blood volume is increased.

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14
Q

What is aldosterone?

A

Released by adrenal glands; holds sodium and water-excrete potassium

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15
Q

What is spironolactone?

A

Aldosterone antagonist; saves potassium and rids sodium and water

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16
Q

Where is potassium excreted and commonly lost?

A

excreted by kidneys (kidney disease can cause build up) and commonly lost through GI d/t vomiting or diarrhea

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17
Q

Foods that raise potassium

A

bananas, spinach, potatoes

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18
Q

Calcium

A

8.5-10.5

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19
Q

Where is calcium regulated?

A

By the parathyroid (PTH) and thyroid( calcitonin)

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20
Q

Which vitamin helps with the absorption of calcium?

A

Vitamin D

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21
Q

What does PTH do in terms of calcium?

A

Released when calcium is low; pulls from bone into blood.

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22
Q

How does the thyroid regulate calcium?

A

When calcium levels in the blood are to high, calcitonin is released and the calcium is pushed into the bones

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23
Q

Chloride

A

95-105

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24
Q

Phosphorus

A

2.5-4.5

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25
Q

What is the inverse of calcium?

A

Phosphorus (if one is high, the other is low)

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26
Q

Bicarbonate

A

22-26

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27
Q

Where does bicarb come from and what does it do?

A

Made by kidneys; assists in acid-base balance; alkalotic

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28
Q

CO2

A

35-45

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29
Q

Hypovolemia

A

Dehydration; low bp, high HR, flat veins, tented skin turgor, concentrated urine with high specific gravitiy

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30
Q

Specific gravity

A

1.005-1.020

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31
Q

Hypervolemia

A

Can be caused by excessive salt intake or cardiac, kidney, liver disease; elevated BP, bounding pulse, distended neck veins, weight gain, edema

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32
Q

Severe hypervolemia

A

Crackles in lungs, dyspnea, ascites

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33
Q

Severe hypovolemia (hypovolemic shock)

A

rapid, weak pulse and orthostatic hypotension

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34
Q

Hyponatremia

A

Can be caused by diuretics, adrenal insufficiency (decreased secretion of aldosterone), kidney disease, SIADH. Weakness, lethargy, nausea and vomiting, muscle cramps, seizures

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35
Q

Hypernatremia

A

Caused by excessive sodium intake, excessive water loss, decreased sodium excretion, and hyperaldosteronism (holds onto sodium, so if more is released-more is held). thirst, dry mouth, hallucinations, irritability, lethargy, seizures, altered mental status

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36
Q

Hypokalemia

A

Can be caused by potassium-wasting diuretics, GI loss, hyperaldosteronism (excretes more potassium), anorexia/bulimia, and alkalosis. Can cause dysrhythmias, flat T-weaves, muscle weakness and cramps, and increased sensitivity to digitalis (digoxin)

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37
Q

How is IV potassium given?

A

NEVER PUSH; on pump and diluted

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38
Q

Hypocalcemia

A

Can be caused by hypoparathyroidism (PTH draws calcium out of the bone into the blood when needed), malabsorption, vitamin D deficiency, increased calcium excretion (kidney disease, diarrhea), and hyperphosphatemia. Diarrhea, numbness/tingling, tetany, cardiac irritability, muscle cramps, convulsions, Chvostek and Trousseu’s

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39
Q

Hyperkalemia

A

Renal failure, potassium-sparing diuretics (spironalactone), hypoaldosteronism (aldosterone causes potassium to be excreted), major trauma/burns. Muscle twitching and late flaccid paralysis, dysrhythmias, elevated T-waves, diarrhea

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40
Q

If a pt’s potassium is low or high, what should be a priority for the nurse?

A

CARDIAC MONITOR

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41
Q

What can be given IV to help with cardiac excitability d/t hyperkalemia?

A

Calcium gluconate

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42
Q

Why is insulin and glucose sometimes given with hyperkalemia?

A

Insulin forces potassium back into cells and glucose to prevent hypoglycemia (REGULAR INSULIN ONLY)

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43
Q

Hypercalcemia

A

Malignant bone disease, hyperparathyroidism, hypothyroidism, prolonged immobilization. Constipations, bradycardia, kidney stones of calcium, muscle weakness, changes in mental status.

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44
Q

Hypomagnesemia

A

Malnutrition, starvation, alcoholics. Hyperactive reflexes, mood changes, disorientation, dysrhythmias.

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45
Q

Hypermagnesemia

A

excess antacids, renal failure. hypoactive reflexes, drowsy, lethargic, depressed respirations, hypotension, bradycardia

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46
Q

Hypophosphatemia

A

acidosis, refeeding after starvation, hyperparathyroidism, high calcium. May experience no symptoms or have joint stiffness, seizures, cardiomyopathy, paresthesia

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47
Q

TPN

A

Should always be started slowly to avoid drops in phosphate; always ask about allergies to eggs d/t fat emulsions

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48
Q

Hyperphosphatemia

A

hyperthyroidism/hypoparathyroidism (would cause calcium to be low so phosphate would go up), chemo, renal failure. Tetany and calcification of soft tissue if imbalance lasted long term.

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49
Q

Intraosseous nursing considerations

A

drill into bones for access; no longer than 24 hours, tibia is the most common location. Complication can be osteomyelitis which may lead to amputation

50
Q

Extravasation at IV site

A

infiltration with caustic fluids that causes tissue damage.

51
Q

CVAD

A

central venous access device; typically inserted into major vein-subclavian or jugular. Risk for sepsis or air embolism

52
Q

How do the lungs work to restore normal pH in acidosis?

A

Respiratory rate increases and depth decreases-get as much CO2 out as quickly as possible

53
Q

How do the lungs works to restore normal pH in alkalosis?

A

Respiratory rate decreases and depth increases-hold on to CO2

54
Q

How do the kidneys work to restore normal pH?

A

In acidosis, bicarb is retained. In alkalosis, bicarb is excreted.

55
Q

How is potassium affected with Acid-base abnormalities?

A

Acidosis-potassium is pushed out of cells by hydrogen ions; in alkalosis, potassium is pushed into cell

56
Q

Atelectasis is:

A

when the alveoli collapse; happens in respiratory acidosis. Can be prevented with turn, cough, deep breath and incentive spirometer

57
Q

What type of respirations are common in metabolic acidosis?

A

Kussmaul; rapid deep breathing. DKA

58
Q

Metabolic alkalosis is commonly caused by

A

excessive vomiting or gastric suctioning causing loss of acid

59
Q

Correct order for abdominal assessment

A

Inspection, auscultation, percussion, and palpation

60
Q

Fecal occult blood test-color and location of bleed

A

Black-upper GI(peptic), Bright red-lower GI (hemorrhoids or polyps).

61
Q

Pt education for fecal occult blood test

A

No red meats, aspirin, or NSAIDs for 72 hours prior-possibility of a false positive. Vitamin C ingestion the morning of can cause false negatives. No laxatives, enemas, or suppositories for 3 days before testing.

62
Q

Pt education for H. Pylori breath test

A

No antibiotics or bismuth for 1 month prior to test, no PPIs for 2 weeks prior, no antacids for 24 hours prior. H. Pylori is a bacteria that infects your stomach or duodenum and can cause a peptic ulcer if not treated.

63
Q

Pt education for after a barium swallow

A

Increase fluids to help evacuate the barium-if it is not excreted, it will harden like concrete. Return immediately if you don’t pass the barium or have bleeding.

64
Q

Prior to CT scan

A

Patient’s renal function should be checked (creatinine and BUN) and allergies to shellfish or iodine should be assessed

65
Q

Creatinine

A

0.8-1.2 for males

66
Q

BUN

A

8-20

67
Q

Post endoscopy, pt should be NPO until

A

gag reflex returns

68
Q

What position should the pt be placed in for colonoscopy?

A

Left side with legs drawn to chest

69
Q

Potential complications for a pt receiving enteral nutrition

A

Diarrhea, vomiting, stomach discomfort, tube displacement, clogging, infection at tube site, refeeding syndrome (drop in phosphate-can be life threatening), aspiration, high blood sugar

70
Q

What position should the bed be in when a pt is receiving enteral feeding?

A

At least 30 degrees

71
Q

What should the graft of a patient with neck resection with a radial forearm free flap look like?

A

Should be pink, if it is blue/mottle there may be venous congestion-immediately alert dr. Look for chyme leakage and immediately report if seen

72
Q

What position should a pt be in post neck dissection?

A

Fowler’s-promotes comfort and spontaneous breathing

73
Q

What to know about suction on a pt with a neck dissection

A

Avoid suctioning near suture line as it can destroy the graft

74
Q

What can be given IV to a pt with an esophageal obstruction from eating?

A

Glucagon-relaxes the esophagus and allows pt to swallow.

75
Q

What is given to treat peptic ulcers?

A

Antibiotics and a PPI; kills the bacteria and stops acid production to promote healing

76
Q

How does H. Pylori cause an infection?

A

Through contaminated food or water

77
Q

What is washboard abdomen a sign of?

A

Perforation/Bleeding (emergency)

78
Q

What is the duration of treatment for a patient with peptic ulcer disease?

A

4-8 weeks

79
Q

How long to listen for bowel sounds before charting absent?

A

5 minutes

80
Q

Meds that can be given for hyperkalemia

A

lokelma and kaexalate, regular IV insulin and glucose

81
Q

Universal donor

A

O-

82
Q

Universal recipient

A

AB

83
Q

TPN risk factors

A

SOB, egg allergy, and IV dextrose

84
Q

What to watch for with blood transfusion

A

Flank pain, SOB- immediately stop transfusion

85
Q

Priority nursing action when caring for a pt with an NG tube post-Whipple procedure

A

DO NOT MANIPULATE

86
Q

Steatorrhea

A

Fat in stool

87
Q

What is stool going to look like after barium swallow?

A

White, chalky

88
Q

S/S for a pt with appendecitis

A

belly button pain, fever and chills, nausea and vomiting, rebound tenderness, bloating, constipation or diarrhea

89
Q

Nursing priority for pt with GI bleed

A

Watch for S/S of shock-low bp, high and weak pulse, diaphoretic; watch for fluid-electrolyte imbalances

90
Q

Dietary measures for pts with Crohn’s

A

increase oral fluids; low residue, high protein, high calorie diet with supplemental vitamins and iron. Avoid foods that could exacerbate symptoms-stuff that can cause bloating and diarrhea

91
Q

Assessment for acute diverticulitis

A

typically dx by colonoscopy; abdominal CT with contrast to confirm diverticulitis; cramping in left lower quadrant, change in bowel habits, bloating, nausea, anorexia, abdominal distention

92
Q

Assessment for stoma 3 days postop

A

Stoma is beefy, red and moist; some serous drainage is normal.

93
Q

Medication that a pt with glaucoma should not take for N/V

A

Phenergan

94
Q

The goal of care in an IBS pt

A

That they will be able to make the lifestyle/diet changes necessary to get the flares under control

95
Q

Nutritional education for a pt with ascites

A

Low sodium to help avoid fluid retention

96
Q

S/S of hepatic encephalopathy

A

Increased serum ammonia, mental status changes, motor disturbances-pt looks confused and unkempt and has alterations in mood and sleep patterns- asterixis (liver flap), apraxia (not being able to remember how to do something), fetor hepaticus

97
Q

What is asterixis and what causes it?

A

Asterixis is the involuntary flapping of the hands when the pt holds their arm out and it is d/t hepatic encephalopathy

98
Q

Nursing priorities for pt with esophageal varices

A

treat for shock, administer oxygen, IV fluids and electrolytes, Vasopressin SOmatostatin Octreotide to decrease bleeding, Beta blockers (lols) to decrease portal pressure

99
Q

Nursing interventions for pts with ascites and portal hypertension

A

daily weights and abdominal girth, administer diuretics as prescribed, low sodium diet

100
Q

What is spironolactone and why would a pt be prescribed it?

A

Spironolactone is an aldosterone antagonist; it is a potassium sparing diuretic so a pt with low potassium but built up fluid would be prescribed this

101
Q

C-diff diarrhea can:

A

cause you to lose a lot of potassium leading to arrhythmias

102
Q

Cullen’s sign is

A

bruising around the umbilicus-pointing to hemorrhagic pancreatitis

103
Q

S/S of chronic pancreatitis

A

severe upper abdominal and back pain, vomiting, weight loss***, steatorrhea (fat in the stool), pale clay colored stool

104
Q

What to watch for after gallbladder surgery

A

Bleeding, peritonitis, sepsis; if the pt complains of any or if it s/s are noticed by nurse-report to dr immediately; rigidity of abdomen, bowel sounds, hypotension and tachycardia

105
Q

Nutrition for pts with gallbladder disease

A

avoid fatty foods, eat low residue diet

106
Q

What to watch for after Endoscopic retrograde cholangiopancreatography (ERCP)?

A

Signs of bleeding and perforation(rigid abdomen, signs of shock) and lozenges can be used for sore throat once gag reflex returns

107
Q

Rapid Acting Insulin

A

Lispro, aspart, glulisine; 15 min onset, 1 hour peak, <5 hour duration; DO NOT GIVE UNTIL PT IS EATING

108
Q

Short-acting/Regular Insulin

A

These will have names like Humulin-R; 30-60 minute onset, 2-3 hour peak, 4-6 hour duration; typically given 15 min before meal

109
Q

Intermediate/NPH insulin

A

these will have names like Humulin-N; 1-1.5 hour onset, 4-12 hour peak, up to 24 hour duration; food given around onset and peak

110
Q

Long-Acting Insulin

A

Glargine, Detemir; 3-6 hour onset, no peak, 24 hour duration; this will be the patient’s basal dose

111
Q

Addisonian Crisis

A

Shock, hypotension, rapid and weak pulse, pallor, extreme weakness; loss of fluid and cortisol drops dramatically

112
Q

Sick day rules for diabetes

A

Take insulin as prescribed, adding more if needed. Alert provider of hyperglycemia and dehydration

113
Q

1st thing to do for pts in DKA

A

Address fluid and electrolyte imbalances

114
Q

What disease typically causes cola-colored urine?

A

Glomerulonephritis

115
Q

Normal BUN

A

8-20

116
Q

Normal creatinine (males)

A

0.6-1.2

117
Q

BPH is treated by

A

-zosins or -osins; Tamsulosin, terazosin, doxazosin

118
Q

Which hormone regulates calcium?

A

Parathyroid hormone causes calcium to leave the bones and enter the blood; calcitonin causes serum calcium to enter the bones

119
Q

What insulin can be given IV?

A

Regular ONLY

120
Q

What is the most common symptom of Addison’s?

A

Muscle weakness

121
Q

What are the functions of the kidneys?

A

filtering blood, producing urine, acid-base balance, regulating fluids