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exam 3 Flashcards

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1
Q

asterixis

A

involuntary flapping of the the hands

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2
Q

which lab level being affected causes jaundice?

A

bilirubin-high

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3
Q

direct serum bilirubin

A

0.1-0.4 mg/dL

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4
Q

direct serum bilirubin

A

0.1-0.4 mg/dL

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5
Q

total serum bilirubin

A

0.3-1mg/dL

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6
Q

Serum albumin

A

3.5-5.2g/dL

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7
Q

AST

A

10-40U/mL

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8
Q

ALT

A

8-40U/mL

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9
Q

Ammonia

A

15-45 mcg/dL

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10
Q

Where does the liver get blood from?

A

Nutrient rich but no oxygen from the GI tract via the portal vein and oxygen rich from the hepatic artery

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11
Q

What are the metabolic functions of the liver?

A

Glucose metabolism, ammonia conversion, protein metabolism, fat metabolism, vitamin and iron storage, bile formation, bilirubin excretion, drug metabolism

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12
Q

What can happen if the drugs aren’t being metabolized properly?

A

Toxicity

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13
Q

How much damage has occurred before liver function studies are abnormal?

A

70%

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14
Q

What is the serum aminotransferase test for?

A

Useful in detecting hepatitis; abnormal levels indicate injury to liver cells

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15
Q

Alanine aminotransferase (ALT)

A

levels increase in liver disorders; used to monitor the course of hepatitis, cirrhosis, the effects of treatments that may be toxic to the liver

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16
Q

Aspartate aminotransferase (AST)

A

not specific to liver diseases; however levels of AST may increase with cirrhosis, hepatitis, and liver cancer

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17
Q

Gamma-glutamyl transferase (GGT)

A

levels are associated with cholestasis; alcoholic liver disease

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18
Q

What should the nurse be aware of post liver biopsy?

A

Watch for signs of bleeding as the liver is very vascular-tachycardia, hypotension. Watch for peritonitis

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19
Q

What is peritonitis?

A

Life threatening condition in which the peritoneum (the tissue that lines the inside of the abdomen) becomes inflamed or infected

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20
Q

What are the symptoms associated with liver disease?

A

Hematemesis, bloody stool, easy bruising, anorexia, weight gain d/t fluid, increasing abdominal girth. abdominal pain, jaundice, fatigue, dry/itchy skin, ascites

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21
Q

What are some drugs that can affect the liver?

A

IV drug use, NSAIDs, Anti-fungals (ending with-zole), and valproic acid seizure meds.

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22
Q

What is cirrhosis?

A

Cell destruction and fibrosis (scarring) of hepatic tissue; typically due to alcohol use

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23
Q

How much fluid is considered to be ascites?

A

greater than 500 mL

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24
Q

Symptoms that come with ascites:

A

Abdominal pain, bloating, increased abdominal girth, nausea, SOB

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25
Q

Why is there an increased risk of bleeding for patients with cirrhosis?

A

The liver produces prothrombin which is a clotting factor; the liver is very vascular and damage could cause bleeding; a clinical manifestation for cirrhosis is thrombocytopenia which is decreased platelets

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26
Q

Portal hypertension

A

Obstructed blood flow through the liver results in increased pressure throughout the portal venous system; life-threatening as it can cause esophageal varices

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27
Q

What is hepatic encephalopathy?

A

A brain disorder caused by the buildup of ammonia when the liver does not properly filter it out

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28
Q

What is the Sengstaken-Blakemore tube used for?

A

To apply pressure to esophageal varices and slow bleeding

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29
Q

Diet for cirrhosis patients:

A

Restrict fluid, sodium, and protein.

30
Q

Why should you restrict protein for a patient with hepatic disorders?

A

More protein=more ammonia, which can build up and cause hepatic encephalopathy

31
Q

What medicine can help lower BP in portal hypertension?

A

Beta -blockers(which end in -lol)

32
Q

You can check for edema in:

A

arms, legs, sacrum (bottom of the back)

33
Q

Skin issues seen in cirrhosis:

A

Jaundice, petechiae

34
Q

Which vitamin can be administered to help with clotting?

A

Vitamin K

35
Q

What medicines should patients be educated not to take with cirrhosis?

A

Tylenol and herbal supplements

36
Q

Palmar erythema

A

redness of the palm

37
Q

Hepatocellular jaundice:

A

Caused by liver cells being damaged; symptoms include lack of appetite, nausea, weight loss, malaise, fatigue, weakness

38
Q

Obstructive Jaundice:

A

Caused by obstruction in bile duct from liver= increased levels of bilirubin not being able to be excreted; clay-colored stools, jaundiced urine, dyspepsia, intolerance of fats, pruritus

39
Q

Most common diuretic for patient with ascites

A

spironolactone-potassium sparing

40
Q

Fetor hepaticus

A

bad breath (sweet and fecal) d/t accumulation of toxins

41
Q

Manifestations of hepatic encephalitis

A

EEG can show progressive slowing, changes in LOC, potential seizures, fetor hepaticus

42
Q

Medical management of hepatic encephalopathy

A

Lactulose- which will cause patient to excrete excess ammonia through feces, IV glucose to minimize protein catabolism, protein restriction, reduction of ammonia from GI by gastric suction, enemas, and oral antibiotics. DISCONTINUE USE OF: sedatives, analgesics, and tranquilizers as liver can not metabolize

43
Q

Manifestations of esophageal varices:

A

MEDICAL EMERGENCY. Hematemesis, melena, general deterioration, and shock-hypotension and tachycardia

44
Q

How often should a cirrhosis patient have an endoscopy to check for varices?

A

Every 2 to 3 years

45
Q

Treatment for bleeding varices:

A

Treat shock-administer oxygen and IV fluids (NS only in case blood is needed )through large gauge. Vasopressin or octreotide which can decrease pressure in portal vein. Octreotide inhibits release of vasoactive peptides that widen vessels. Somatostatin constricts vessels. Beta blockers to decrease BP

46
Q

Nursing management of varices:

A

Administer prescribed treatments and monitor for complications. Encourage deep breathing and sitting up to prevent aspiration

47
Q

Hepatitis A and E

A

spread by fecal-oral route; E also by contaminated water; A by poor hand hygiene

48
Q

Hepatitis B and C

A

Bloodborne

49
Q

Hepatitis D

A

Only people with Hepatitis B are at risk

50
Q

Nonviral hepatitis is caused by:

A

hepatic toxins or drug induced

51
Q

Pancreas-endocrine

A

insulin, glucagon, somatostatin

52
Q

How does the pancreas lower glucose?

A

By allowing glucose into liver cells

53
Q

Choledocholithiasis

A

Gallstones

54
Q

What is murphy’s sign?

A

Deep breath causes increase in pain while examiner palpates; positive with cholecystitis

55
Q

Why should morphine not be used with cholecystitis?

A

Can cause spasm in sphincter of oddi

56
Q

Complications of laparoscopic or open cholecystectomy

A

Infection, bleeding, peritonitis. Risks increase with gall bladder perforation, multiple abdominal surgeries, and morbid obesisty

57
Q

What diet started and when after cholecystectomy?

A

Soft diet after bowel sounds return

58
Q

What simple nursing intervention can help with pain post-cholecystectomy?

A

Splinting or holding a pillow to the site

59
Q

Nursing management post cholecystectomy

A

turn, cough, deep breathe q1h; watch for washboard abdomen; monitor vitals -fever, hypotension, tachycardia; soft diet-low in fat and high in carbs and protein to aid in healing.

60
Q

How long should a patient be on a restricted fat diet post-cholecystectomy?

A

4-6 weeks

61
Q

Acute pancreatitis

A

Inflammation of the pancreas; caused by the release of pancreatic enzymes that “autodigest’ the pancreas

62
Q

Signs of peritonitis:

A

Steatorrhea (fatty stools), elevated BP, nausea and vomiting, elevated temp, severe epigastric pain

63
Q

What is Cullen’s sign?

A

edema and bruising around the umbilicus; can indicate internal bleeding: hemorrhage d/t necrotizing pancreatitis

64
Q

What is Turner’s sign?

A

Bruising of the flanks or lower back that indicates internal bleeding; hemorrhage d/t necrotizing pancreatitis

65
Q

What electrolyte is affected by acute pancreatitis?

A

Calcium- can be low. Monitor closely for Trousseu’s and Chvostek’s

66
Q

Why does hypocalcemia happen in acute pancreatitis?

A

Chelation of calcium salts by increased fatty acids; Causes calcium to bind to the chelating agent and lower the level in circulation

67
Q

What medications to be used with acute pancreatitis and why?

A

Opioid narcotics-treat pain; anticholinergic-decrease intestinal motility and pancreatic enzyme release; spasmolytics-relaxes smooth muscle and sphincter of oddi; PPI-decreases gastric acid secretions

68
Q

What are Histamine 2 Antagonists such as cimetidine used for in the treatment of acute pancreatitis?

A

Reducing pancreatic secretions

69
Q

Which labs should be reviewed for chronic pancreatitis?

A

Amylase, lipase, bilirubin, alkaline phosphatase, and elevated blood glucose

70
Q

Normal amylase

A

25-120 units per liter

71
Q

Normal lipase

A

0-160 units per liter

72
Q

After a Whipple procedure or pancreaticoduodenectomy:

A

NGTs are maintained to decompress the stomach, prevent bloating, and remove gastric acid secretions to minimize pressure on surgical site. DO NOT MANIPULATE NGT

nursing (14 decks)

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