Fetal Growth Disturbances & Prematurity Flashcards

1
Q
A
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2
Q

What is SGA?

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3
Q

Major differences between preterm and SGA babies

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4
Q

What are the external features assessed on the clinical assessment of maturity - scoring system (New Ballard score, modified from Dubowitz)?

A
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5
Q

Neurological assessment (relative flexor tone of 4 limbs)

[New Ballard score, modified from Dubowitz]

A

Don’t use this scoring for neurologically abnormal baby

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6
Q

What is Intrauterine Growth Restriction?

A

SGA is statistical term

Assessment of foetal growth must be based on statistics from that ethnicity

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7
Q
A

Yellow: IUGR (initially normal, but gets worse = look at the course)
Red: SGA (small from the start)

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8
Q

What is the consequence of reduce growth support?

A
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9
Q

% reduction in organ weight in SGA baby compared to normal counterparts

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10
Q

Pathological factors associated with slow foetal growth

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11
Q

What are fetal and placental factors?

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12
Q

What are embryopathies?

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13
Q

What. areconstitutional and genetic factors causeing slow feotal growth>

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14
Q

Idiopathic causes for SGA

A
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15
Q

History, ultrasound, p/e

A
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16
Q
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17
Q

Special problems of SGA babies

A

Lower reserve

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18
Q

Glucose disturbance in SGA babies

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19
Q

Hypothermia in SGA babies

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20
Q

CBC issue in SGA babies

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21
Q

Cardiorespiratory problems in SGA babies

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22
Q

What are some primary causes of SGA in babies?

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23
Q
A
  • Hepatosplenomegaly
  • Hearing loss
  • Intracranial calficiations
  • Blueberry muffin baby
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24
Q

Management of SGA babies

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25
Q

What must be done during resuscitation of SGA baby?

A

Dextrose gel may be used before mother can breastfeed

Search for underlying cause

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26
Q

Future growth and development of SGA babies
- Catch up growth
- Neurodevelopmental sequalae

A
27
Q

Long-term implications of SGA babies

A

I.e. blood will be diverted to brain, instead of gut, liver, pancreas = in adulthood, more prone to adult disease due to poorer development of these organs

Metabolic syndrome risk is greater

28
Q

Complicatoins of infant of diabetic mother

A
29
Q

What is preterm, very preterm and extreme prematurity?

A
30
Q

What is associated with spontaneous premature birth?

A
31
Q

Foetal and socioeconic factors with spontaneous premature birth

A
32
Q

Problems related to prematurity

A
33
Q

What is RDS?

A

Surfactant is made at around 30-32 weeks
Mother with diabetes

34
Q
A

Widreslread collapsed membrane in alveoli

35
Q

Factors predisposing to RDS: Sex, C-section, maternal, familial

A
36
Q

Factors predisposing to RDS

A
37
Q

What is the usual cord clamping time in labor ward?

A

~ 1 min, unless there are other causes

38
Q

Factors protecting against RDS

A
39
Q

Clinical diagnosis of RDS

A
40
Q
A

Bilateral lung fields are white-out
Cannot see cardiac border
Air bronchograms (atelectasis)
Reduce no. of ribs = lung voluem diminished

41
Q

What is done for prevention of RDS?

A
42
Q

What are other causes of respiratory distress?

A
43
Q

Treatment of RDS

A
44
Q
A

Nasal CPAP or nasal IMV

Prevent lung complications, which may persist into later life

45
Q

What are neurological complications in RDS?

A
46
Q

RF for IVH/PVH

A
47
Q

missing slide

A
48
Q
A

Blood clot inside IVH

49
Q
A
50
Q

Management of PVH / IVH

A
51
Q
A

Enlarged lateral ventricels

52
Q

What is periventricular leucomalcia?

A
53
Q

Neuroreduction in

A
54
Q

Nutritional management in premature infant

A
55
Q

Enteral feeding

A
56
Q

What is NEC?

A
57
Q

RF for NEC

A
58
Q

XXX

A

DISTENS

59
Q

CLINIACL EFATURES OF NEC

A
60
Q
A
61
Q

Prevention of NEC

A

Breast milk = evidence-based

62
Q

Treatment of NEC

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63
Q
A
64
Q

Late complications related to prematurity

A