Equine feeding issues Flashcards

1
Q

feeding management issues (3)

A

competition, vices and dental problems

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2
Q

competition

A

herd feeding issue, fat vs. skinny, no separation during feeding

feed in compatible groups, spread out piles, can cause trauma from fighting over food, esophogeal obstruction - “choke” bolting feed to eat quick to go get more food

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3
Q

esophageal obstruction:
clinical signs
most at risk type of food
TX and management

A

acute onset: watery feed tinged nasal discharge, gagging, repeated attempts to swallow, stretching of the neck, anxious

pelleted feed, hay cubes, beet pulp

TX: sedation, nasogastric tube – lavage to dissolve obstruction - watch out for aspiration
-once it happens once, most likely will happen to same horse again, slow down eating, separate the “bully” and soak feed

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4
Q

Vices
abnormal oral vs. locomotor
all lead to what?

A

abnormal oral: cribbing, wind sucking, wood chewing
locomotor: weaving, stall walking, head shaking

all lead to weight loss
associated with being bored, low roughage diet, prolonged confinement

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5
Q

cribbing

-what is it
-concerns

A

put upper incisors on something, only observed in domesticated or captive horses
GIVE MORE FIBER (ROUGHAGE)

concerns: poor performance, weight loss, incisor wear, colic, more prone to epiploic foramen entrapment, destruction of property, annoying
cribbing collars to prevent
surgery of cutting muscles, laser method, antidepressants

TO FIX: FEED MORE ROUGHAGE

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6
Q

wind sucking

A

start with this and then lead to cribbing
confused with cribbing

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7
Q

wood chewing

A

just chewing, no air sucking
destructive, at risk of splinters/FB

need to feed more hay, bored because they are not grazing

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8
Q

stall walking
weaving

A

bored and have excess energy

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9
Q

dental issues
clinical signs
enamel points
molar hooks

A

clinical signs: weight loss, dropping feed, difficulty eating, quidding (cant grind up enough food, store in cheek to keep chewing and then spit it out)
enamel points: leads to ulceration of cheek, buccal surface on maxilla, lingual surface on mandible
molar hooks also lead to ulceration
TX: dental float - file down sharp points

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10
Q

roughage issues (3 types of roughage)

A

alfalfa, clover and fescue

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11
Q

alfalfa good for ?
problem?
clinical signs with problem?

A

E dense, highly fermentable
problem: blister beetles are attracted by the flower, come in swarms and toxic to horses even if the beetle is dead
swarm of beetles is the problem, if horse eats one thats okay, not in big numbers
Clinical signs: colic, anorexia, depression, frequent urination (hematuria), colitis, oral ulcerations
management: inspect every flake! no specific TX, supportive care

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12
Q

fescue
-problem and effect?
-what group of horses are we concerned about?
CS?

A

fescue contaminated with endophyte –> dopaminergic inhibition of prolactin from ant. pit. = decrease prolactin and progesterone (because of fungus on hay)
-concerned about pregnant mares

CS: prolonged gestation, premature separation of placenta, dystocia, retained placenta

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13
Q

fescue
-problem and effect?
-what group of horses are we concerned about?
CS?

Fescue toxicosis: prevention

A

fescue contaminated with endophyte –> dopaminergic inhibition of prolactin from ant. pit. = decrease prolactin and progesterone (because of fungus on hay)
-concerned about pregnant mares

CS: prolonged gestation, premature separation of placenta, dystocia, retained placenta

toxicosis: due to fungus that grows on plant when plant has gone to seed = mature
PREVENT: cut grass or move mare, dopamine antagonist, fungus free fescue (not as hardy), re seed (alot of $$)

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14
Q

clover
-problem?
-CS?
-fix

A

clover infected with mold aka slobber toxin
-increased salivation, no significant problem
-dehydration in severe cases, but very unlikely
-remove horse from clover and will fix overnight
-more worry about owners

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15
Q

3 concentrate issues

A

grain overload, ionophore toxicity, moldy corn poisoning

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16
Q

grain overload

A

colic, colitis (inflammation of LI), gastric rupture, laminitis

17
Q

colic
CS
TX

A

due to increase in NSC
-rapid fermentation in the hind gut = gas distention
CS: looking at flank, pawing, rolling
NSAIDs, rectal exam, NG intubation (reflux, mineral oil - binds endo toxins to feces and prevents from being absorbed)

18
Q

laminitis
sequence
prevention
TX

A

NSC –> rapid fermentation in LI –> increase endotoxins –> endotox absorption –> laminitis
-affects front limbs first because 60% of weight is in front limbs
prevent: ice slurry - 50% ice, 50% water, change every 2-3 hours for up to 48 hours, iced horses 10x less chance of developing laminitis
TX: NSAIDs, shoeing, sole support, diet (NO CARBS)

19
Q

ionophore toxicity
CS
TX

A

ionophore ABX added in coccidiostats to improve weight gain in ruminants, swine and poultry
-horses are 200x more sensitive to it
-most common: monensin

CS: cardiotoxic - trembling, sweating, cardiac arrhythmias, death

TX: support, lavage GI if acute, mineral oil, activated charcoal - once see clinical signs = too late

20
Q

moldy corn poisoning causes ?
CS
prevention

A

leukoencephalomalacia - melts white matter
any feed containing corn is at risk
-mycotoxin growth with a wide temperate range, pink to red/brown on kernels
-when moisture is greater than 15%
CS: rapid progression- incoordination, depression, blindness, sweating, head pressing, seizures, coma, death
prevent: dont feed moldy corn!!