ENT Flashcards
which is more common syndromic or non syndromic hearing loss
non syndromic is 70% of congenital hearing loss cases. Its due to genetic mutations of connexin genes found on ch13 (SNHL)
timing of hearing loss
antenatal, perinatal, postnatal
is congenital hearing loss more genetic or not
genetic
syndromic HL is associated with 5 syndromes
- usher syndrome (+retinitis pigmentosa)
- pendred syndrome (+goiter)
- alport syndrome (+nephropathy/keratoconus)
- treacher collins (+misformed features)
- jervell and lange neilson syndrome (+cardiac arrythmia-long QT)
speaking ages
- preverbal (<1yr)
- periverbal (1-3yr)
- postverbal (>3yr)
SNHL is related to what genes
AR-CONNEXIN 26 (GJB2)
GJB6-CONNEXIN 30
AD-CONNEXIN 31
severe HL and deep HL
severe>75-95 dB
deep >95 dB
when do adenoids start to decrease in size
beyond 7 years
TM athelectasis is
retraction of TM
complications of AOM
mastoiditis, meningitis, petrositis, VII defect, cerebral abscess, sigmoid sinus thrombosis
contrast viral and bacterial acute pharyngotonsillitis
viral (65% of cases): tonsil inflammation+low fever(<38) for a few days+non productive cough
bacterial (30% of cases): tonsil inflammation+high fever for >4 days+exudate+uvulitis+no cough
symptoms of rhinosinusitis
chronic cough for more than 4 weeks+rhinorrhea+nasal congestion+post nasal drip+facial pain
otoacoustic emission tests for
cochlear functioning
when does the E tube acquire adult conformation
after 7 years old
frontal and sphenoidal sinuses achieve pneumatization when?
9-13 years old
conductive hearing loss levels
30-40 dB
bugs causing AOM
m. catarrhalis
S. pneumonia
H.influenza
S. pyogenes
treatment of pharyngotonsillitis
first symptomatic treatment. If fever lasts more then 4 dyas start antibiotics (amoxicillin)
neck masses in kids
- HL or NHL
- branchial cyst infection
- thyroglossal duct cysts
- ectopic thyroid
- plunging ranula
otohematoma is
cauliflower ear
ramsay hunt syndrome
reactivation of herpes zoster oticus along VII/VIII near geniculate ganglion.
exostosis
bilateral multiple bony growths in EAC. can be due to periostitis due to cold water
osteoma
benign unilateral neoplasm of bony EAC. Has narrow base of implant
differentiate a transverse from a medial temporal fracture
medial (longitudinal) has secretion in EAC.
transverse can affect TMJ and mastoid
furunculosis is? Therapy is?
a localised pustular folliculitis due to staph aureus. Treat with antibiotics
otomycosis is most often due to
aspergillus and candida
external ear imaging
CT no contrast
MRI contrast
EAC bony vs fibrocartilagenous. E. tube bony vs cartilage?
EAC: cartilage 1/3+bony 2/3
E tube: cartilage 2/3+bony 1/3
cough reflex
stimulation of auricular branch of vagus
cholesteatoma is
s an abnormal skin growth that can develop in the middle ear. It usually begins as a collection of dead skin cells and develops into a cyst-like pocket behind the eardrum. It can significantly impair a person’s hearing and balance, as well as the function of their facial muscles.
the middle ear is divided into 3 parts
epitympanim, mesotympanum, hypotympanum
jacobson nerve
is in hypotympanum, is IX, eventually enters otic ganglion
bullous myringitis
small fluid filled blisters form on the eardrum. Are painful
stages of AOM
hyperemia, exudation, suppuration, resolution