Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

NUR372 > Endocrine > Flashcards

Endocrine Flashcards

1
Q

Anterior Pituitary Gland

A

thyroid stimulating hormone (TSH), growth hormone (GH), adrenocorticotropic hormone (ACTH), follicle stimulating hormone (FSH), luteinizing hormone (LH), prolactin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Posterior Pituitary Gland

A

antidiuretic hormone (ADH), oxytocin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Thyroid gland

A

works via COMPLEX FEEDBACK; a vascular gland that produces T4, T3, and calcitonin.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

T3 and T4

A

regulate cellular metabolism as well as growth and development, stress response (if body is cold or trauma) these will produce opposite effects

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Calcitonin

A

controls calcium levels in the blood

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

The hypothalamus stimulates the pituitary gland to produce

A

TSH which stimulates T3 and T4

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What is required to synthesize thyroid hormones

A

iodine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Overactive thyroid gland

A

weight loss, anorexia, fast metabolism

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Underactive thyroid gland

A

overweight, obesity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Parathyroid gland

A

secretes PTH which works opposite of calcitonin to regulate serum calcium levels

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

PTH is secreted when

A

serum calcium levels drop

increases osteoclast activity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Adrenal glands

A

medulla

cortex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Cortex is stimulated by

A

ACTH from anterior pituitary

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Mineralocorticoids

A

primary aldosterone (conserves na and water)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Glucocorticoids

A

primary cortisol

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Transsphenoidal removal of the Pituitary gland (Hypophysectomy): after procedure

A

 Don’t blow nose for 2 months
 Avoid bending or stooping (keep intracranial pressure low to avoid loss of CSF through surgical site)
 Fever or drainage must be reported immediately
 Pt will wear a mustache dressing because they will be dripping CBS

Puts at risk for meningitis!!! s/s: nucal rigidity, fever

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Cushings disease

A

caused by adrenal cortext issues

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Cushings syndrome

A

resulting from something other than the gland itself (i.e. medications, steroids); is due to a ACTH-secreting tumor or is induced by corticosteroid drugs (prednisone)

19
Q

What happens to the glucose levels in cushings syndrome?

A

will go up because glucose mirrors cortisol

20
Q

Plasma cortisol levels morning

A

5-23

21
Q

Plasma cortisol levels evening

A

3-13

22
Q

Know what to assess for with SIADH remember this is inappropriate diuretic hormone so think about fluid status and s/s of fluid overload.

A

Hyponatremia

Severe hyponatremia

23
Q

Hyponatremia s/s

A

muscle cramps, weakness, thirst, fatigue, dulled sensorium, low UOP, WEIGHT GAIN

24
Q

Severe hyponatremia s/s

A

vomiting, abdominal cramps, muscle twitching, seizures, cerebral edema, anorexia, confusion, coma

25
Q

Understand why we would either restrict or not restrict fluids for patients experiencing SIADH.

A

restrict fluids

26
Q

Important point about SIADH is that you typically have normal renal function

A

but significant fluid retention

27
Q

Important to treat underlying cause in SIADH

A

tumors, head trauma, drugs

28
Q

SIADH monitor which electrolytes

A

sodium

potassium

29
Q

What happens with hyperthyroid? Speeds everything up remember fight or flight response what manifestations will be seen?

A

graves disease
toxic nodular goiter
CM

30
Q

Graves disease

A

autoimmune may be triggered by insufficient iodine levels, stress, infection

31
Q

Toxic nodular goiter

A

nodules that excrete excessive T3 and T4, independent of TSH stimulation

32
Q

CM

A

goiter, thyroid bruits from increased oxygen supply, exophthalmos, anorexia, weight loss, depression, a fib, nervousness, confusion

33
Q

What are complications of hyperthyroid?

A

Thyrotoxic crisis

Severe tachycardia, heart failure, shock, hyperthermia, agitation, pain, nausea, vomiting

34
Q

Potential trigger of hyperthyroid

A

stress, infection

35
Q

T3

A

70-205

36
Q

T4

A

4-12

37
Q

TSH

A

2-10

38
Q

Hypothyroid

A

Decreased circulating thyroid

39
Q

Most common cause of hypothyroid

A

iodine deficiency

40
Q

Hypothyroid s/s

A

(cold intolerance, hair loss, muscle weakness, weight gain)

Myxedema (fat accumulation)

41
Q

REMOVAL OF THYROID GLAND

A

Lifetime replacement of hormone (Hormone Replacement Therapy HRT)

Assess for s/s of hyper/hypo calcemia due to hormone level changes

42
Q

Hypercalcemia s/s

A

weakness, loss of appetite, constipation, need for sleep, emotional disorders, decreased attention span

43
Q

hypocalcemia s/s

A

looks like tetany, tingling lips, fingertips. Increased muscle tension leading to paresthesias and stiffness.

44
Q

calcium and phosphorus have an antagonistic relationship

A

When one is elevated the other is decreased

NUR372 (13 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions