Electrocardiogram Flashcards

1
Q

P wave

A
  • atrial depolarization
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2
Q

PR interval

A
  • time for atrial depolarization and conduction from the SA node to the AV node
  • normal duration is 0.12 to 0.20 seconds
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3
Q

QRS complex

A

ventricular depolarization and atrial repolarization

-normal duration is 0.06 to 0.10 seconds

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4
Q

QT interval

A
  • time for both ventricular depolarization and repolarization
  • normally ranges from 0.20 to 0.40 seconds depending on HR
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5
Q

ST segment

A
  • isoelectric period following QRS when the ventricles are depolarized
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6
Q

Twave:

A

-ventricular repolarization

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7
Q

normal sinus rhythm

A
  • atrial repolarization begins in the SA node and spreads normally throughout the electrical conduction system with a HR between 60 and 100 bpm
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8
Q

sinus bradycardia

A
  • sinus rhythm with a heart rate less than 60 bpm
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9
Q

sinus tachycardia

A

-sinus rhythm with a HR more than 100 bpm

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10
Q

sinus arrhythmia

A
  • sinus rhythm but with quickening and slowing of impulse formation in the SA node resulting in a slight beat-to-beat variation of the rate
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11
Q

sinus arrest

A

sinus rhythm, except with intermittent failure of either SA node impulse formation or AV node conduction that results in the occcasional complete absence of P or QRS waves

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12
Q

Premature atrial contractions PAC

A
  • occurs when an ectopic focus in the atrium initiates an impulse before the SA node
  • the P wave is premature with abnormal configuration
  • PACs are very common and generally benign, but may progress to atrial flutter tachycardia or fibrillation
  • may occur with a normal heart and any type of heart disease
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13
Q

Atrial Flutter

A
  • an ectopic very rapid atrial tachycardia
  • atrial rate of 250-350 bpm; ventricular rate dependent upon AV node conduction
  • saw tooth shaped P wave are characteristic
  • occurs with valvular disease, ischemic heart disease, cadriomyopathy, HTN, acute myocardial infarction, chronic obstructive lung disease and pulmonary emboli
  • S/S include palpitations lightheadedness and angina due to a rapid rate
  • stagnation of blood may predispose to thrombi in the atria
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14
Q

Atril Fibrillation

A
  • common arrhythmia where the atria are depolarized between 350 and 600 times/min
  • ECG shows characteristically irregular undulations of ECG baseline without discrete Pwaves
  • occurs in healthy hearts and in patients with coronary arteyr disease, HTN and valvular disease
  • S/S may include palpitations, fatigue, dyspnea, lightheadedness, syncope, and chest pain
  • stagnation of blood may predispose to thrombi in the atria
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15
Q

1dt degree Atrioventricular Block

A
  • PR interval is longer than 0.2 seconds but relatively constant from beat to beat
  • no symptoms or significant change in cardiac function
  • PR interval may become prolonged fro many reasons including medications that supress AV conduction
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16
Q

2nd degree Atrioventricular block

A
  • AV conduction disturbance in which impulses between teh atria and ventricles fail intermittently
  • two major types Mobitz type I and Mobitz type II block
17
Q

Mobitz I

A
  • progressive prolongation of PR interval until one impulse is not conducted
18
Q

Mobitz Type II

A
  • conductive PR intervals are the same and normal followed by non-conduction of one or more impulses. if heart rate is slow, CO will decr with the blocked impulse
  • also 2nd degree AV block may progress to 3rd degree AV block
19
Q

3rd degree atrioventricular block

A
  • all impulses are blocked at the AV node and none are transmitted to the ventricles
  • the atria and ventricles are paced independently; atrial rate> ventricular rate
  • Considered a medical emergency requiring a pacemaker
  • if the ventricular rate is too slow, the CO drops and the patient may faint
  • common causes include degenerative changes of the conduction systems, digitails, heart surgery, and acute MI
20
Q

Premature ventricular complex PVC

A
  • premature depolarization arising in the ventricles due to an ectopic focus
  • on ECG, the P wave is usually absent and QRS complex has a wide and aberrant shape
  • common arrhythmia that occurs in healthy and diseased hearts
  • Pt may be asymptomatic or have palpitations
  • Common causes include anxiety, caffeine, stress, smoking and all forms of heart disease
21
Q

unifocal PVC

A
  • unifocal PVCs arise from the same ectopic focus and have the same configuration
22
Q

multifocal PVC

A
  • arise from different ectopic foci and have different configurations
23
Q

bigeminy

A
  • normal sinus impulse is followed by a PVC
24
Q

trgeminy

A
  • PVC occurs after every two normal sinus impulses
25
Q

Ventricular Tachycardia V-tach

A
  • 3 or more consecutive PVCs at a ventricular rate of >150 bpm
  • P wave are absent andQRS complexes are wide and aberrant in appearance
  • V-tach longer than 30 sec is a life threatennig arrhythmia and requires immediate medical intervention
  • Pt ar enot able to maintain an adequate BP and eventually become hypotensive
  • V-tach may degenerate into venticular fibrillation causing Cardiac arrest
  • common causes include: MI, cardiomyopathy and valvular disease
26
Q

Ventricular fibrillation V-fib

A
  • ventricles do not beat in a coordinated fashion, but fibrillate or quiver asynchronous and ineffectively
  • no cardiac output; pt becomes unconscious
  • ECG shows characteristic fibrilatory waves with an irregular pattern that is either coarse or fine
  • a lethal tachyarrhythmia requires immediate defibrillation
  • additional measures include medications to support teh circulation and intravenous antiarrhythmic agents
  • common causes include heart disease of any type, MI and cocaine use
27
Q

ventricular asystole

A
  • ventricular standstill with no rhythm
  • ECG records a straight-line pattern
  • Requires immediate CPR and medications to stimulate cardiac activity
  • common causes include acute MI, ventricular rupture, cocaine use, lightning strikes and electrical shock
28
Q

ST segment depression

A
  • a depressed ST segment is a sign of subendocardial ischemia, but also can be due to digitalis toxicity or hypokalemia
  • the segment is evaluated relative to isoelectric baseline at 0.08 seconds after the Jpoint
  • deviations from the isoelectric baseline are expressed at ST segment depression of 1 mm. 2 mm etc
29
Q

ST segment elevation

A
  • earliest sign of acute transmural infarction
  • can also indicate a benign early repolarization pattern in normal heart
  • deviations from the isoelectric baseline are expressed as ST segment elevation of 1 mm, 2 mm etc
30
Q

Q wave

A
  • characteristic marker of infarction; signifies teh loss of positive electrical voltages due to necrosis
  • significant or abnormal Qwave is longer than 0.04 msec and larger than 1/3 teh amplitude of teh R wave
31
Q

T wave inversion

A
  • occurs hours or days after an MI as the result of a delay in repolarization produced by the injury
  • may also occur with right and left bundle branch blocks, after a CVA, and as a normal juvenile T wave pattern in children and some adults