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Dermatology (also gross but true) > Eczematous and Papulosquamous Disorders > Flashcards

Eczematous and Papulosquamous Disorders Flashcards

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Dermatology Board Prep flashcards
1
Q

Hallmark of disease is pruritus. Scratching leads to eczamatous change and lichenification. Lesions may ooze, crust, become purulent

A

atopic dermatitis (eczema)

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2
Q

Two very common locations for atopic dermatitis

A

antecubital and popliteal flexure areas

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3
Q

Antihistamines used to treat pruritus of atopic dermatitis

A

doxepin or vistaril

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4
Q

Solution used to dry up oozing lesions

A

Burows (Domeboro)

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5
Q

Corticosteroid treatment for mild to moderate cases of atopic dermatitis

A

desonide 0.05% or hydrocortisone 2.5%

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6
Q

High potency corticosteroid treatment for moderate atopic dermatitis

A

fluocinolone 0.025%, triamcinolone 0.1%, betamethasone dipropionate 0.05%

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7
Q

Used for patients who require therapy to the face or skin folds for more than three weeks

A

Topical calcineurin inhibitors

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8
Q

Name the Topical calcineurin inhibitors

A

tacrolimus (protopic) and pimecrolimus (Elidel)

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9
Q

Black box warning of Topical calcineurin inhibitors

A

increase the incidence of skin cancer and lymphoma with long term use

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10
Q

Coin shaped lesions that usually occur on the trunk and lower extremities. Tx is the same as for atopic dermatitis

A

nummular eczema

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11
Q

occurs as a result of chronic eczematous changes and scratching. Circumscribed plaque of thickened skin with increased markings with some scaling

A

lichen simplex chronicus

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12
Q

Vesicular eruption on the skin of the hands and feet marked by intense itching (vesicles are deep). Scaling, fissures and lichenification may follow

A

dyshydrotic eczema

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13
Q

Caused by direct exposure to a substance as far back as 2 weeks ago or may develop rxn to products that have been used for yrs. Intense pruritis, rash, papular erythematous lesions

A

contact dermatitis

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14
Q

Common plant substance in north america found in poison ivy/oak/sumac, mango skin, or gingko fruit, that is responsible for contact dermatitis

A

oleoresin urushiol

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15
Q

Systemic corticosteroids that can be used for plant based contact dermatitis that covers a large area (>10% BSA), face, or genitals

A

prednisone 2-3 wks

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16
Q

Topical corticosteroids for contact dermatitis

A

clobetasol 0.05% crm

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17
Q

Helpful for treating pruritus of allergic contact dermatitis but not plant based

A

antihistamines

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18
Q

Caused by a combination of wetness, pH elevation and friction. Erythema in diaper area

A

diaper dermatitis

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19
Q

When should you suspect candidal infection secondary to diaper dermatitis?

A

predominately in creases and there are satellite lesions

20
Q

Treatment for diaper dermatitis without secondary yeast infection

A

frequent diaper changes, vaseline or desitin

21
Q

Treatment for diaper dermatitis with secondary yeast infection

A

topical antifungal (nystatin, miconazole, clotrimazole), vaseline. No steroids

22
Q

Treatment of candidiasis if failure of topical therapy

A

oral fluconazole (diflucan)

23
Q

1-2 mm clustered erythematous papules, paulovesicles or paulopustules with or without scale. Occurs most often around the mouth sparing the vermillion border

A

perioral dermatitis

24
Q

What is perioral dermatitis strongly correlated with?

A

topical steroid use

25
Q

First line treatment for perioral dermatitis

A

topical calcineurin inhibitors***, topical erythromycin, or topical metronidazole

26
Q

What diseases may have widespread seborrheic dermatitis?

A

HIV and Parkinson’s

27
Q

Erythematous scaling patches develop in areas of sebaceous glands (scalp, face, trunk)

A

seborrheic dermatitis

28
Q

What is thought to be the pathogen responsible for seborrheic dermatitis?

A

saphrophyte Malessezia (pityrosporum ovale)

29
Q

In addition to erythema, some pruritus, and distribution involving nasolabial folds and central face, what characterizes clinical manifestation of seborrheic dermatitis

A

swollen and greasy appearance

30
Q

Treatment of seborrheic dermatitis if located on the scalp

A

T-gel, selenium sulfide, zinc pyrithione, or ketoconazole shampoo

31
Q

Treatment of non-scalp seborrheic dermatitis

A

ketoconazole, ciclopirox, tacrolimus, or pimecrolimus crm.

32
Q

What can be added on to treatment of non-scalp seborrheic dermatitis in severe cases?

A

low potency topical corticosteroid for 1-2 weeks

33
Q

Medications that may cause lichen planus

A

ACE inhibitors and beta blockers

34
Q

Shiny, flat, polygonal, violaceous papules or plaques with white lacy pattern that is a clinical manifestation of lichen planus

A

Wickham’s striae

35
Q

How is lichen planus diagnosed?

A

biopsy

36
Q

In addition to Wickhan’s striae, symptoms include White reticulate lesions occur on mucosal surfaces that are intensely pruritic

A

lichen planus

37
Q

Initial tx of localized cutaneous lichen planus on trunk or extremities

A

high potency corticosteroids

38
Q

Treatment for patients with widespread lichen planus

A

phototherapy, acitretin, systemic glucocorticoids

39
Q

Occurs from blood pooling due to chronic venous insufficiency. Hemosiderin from the blood cells stain the skin

A

stasis dermatitis

40
Q

Treatment of stasis dermatitis

A

compression stockings and elevation of legs

41
Q

What is the first sign of pityriasis rosea?

A

herald patch

42
Q

Salmon colored lesions that are often oval with long axis paralleling the lines of skin stress (christmas tree distribution). Resolves in 6-10wks.

A

pityriasis rosea

43
Q

Treatment for severe cases of pityriasis rosea

A

acyclovir or phototherapy

44
Q

manifestation of the mildest form of seborrheic dermatitis

A

dandruff

45
Q

What is the likely etiology for a lesion that is small, starts to grow with steroids and has central clearing?

A

fungal

46
Q

What combination of treatments for skin conditions should be avoided?

A

steroid and antifungal

47
Q

Difference between tinea versicolor and vitaligo

A

tinea versicolor has scaling

Dermatology (also gross but true) (12 decks)

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