ECGs Flashcards

1
Q

how to work out rate

A

300/big sqaures

or no. of big complex x6

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2
Q

how to look at rhythm

A

p waves present?
every p followed by QRS?
regular RR?

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3
Q

axis of ECG

A

reaching in 1 and 3 = right

leaving in 1 and 3 = left

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4
Q

what makes a regular rhythm

A

p wave followed by a QRS wave

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5
Q

causes of regularly irregularly rhythm

A
sinus arrhythmia (ectopics)
wenckebach/motibz 1 = progressively lengthening PR until one dropped
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6
Q

causes of irregularly irregular rhythm

A

AF

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7
Q

causes of absent p waves

A

AF

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8
Q

causes of dissociated p waves

A

complete heart block

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9
Q

causes of p mitrale

A

(bifid p waves) LAH = AS, MR, MS

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10
Q

causes of p pulmonale

A

(peaked p waves) RAH = pulmonary HTN and COPD

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11
Q

causes of prolonged PR

A

heart block

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12
Q

what is the normal PR

A

0.12-0.2

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13
Q

causes of shorted PR

A

WPW, HOCM

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14
Q

causes of depressed PR

A

pericarditis

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15
Q

what is the normal QRS

A

0.12

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16
Q

cause of wide QRS

A

BBB

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17
Q

cause of narrow QRS

A

SVT

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18
Q

cause of pathological q waves

A

full thickness MI

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19
Q

cause of an elevated ST

A

> 1mm
STEMI
prizmentals angina
pericarditis (saddle shape)

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20
Q

cause of depressed ST

A

> 0.5mm
ischaemia (flat)
digoxin (down sloping)
MI

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21
Q

cause of tall T waves

A

hyperkalaemia

MI

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22
Q

cause of flat/inverted T waves

A

hypokalaemia
MI
BBB
anxiety, smoking, tachycardia, haemorrhage, shock

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23
Q

when can inverted T wave be normal

A

V1, V2, aVR

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24
Q

what does RR measure

A

ventricular rate

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25
causes of short QT
digoxin, BB, phenytoin
26
cause of long QT
``` TIMME toxins (macrolides, TCAs, antiarrhytmias) inherited ischaemia myocarditis/MVP electrolytes = low Mg, K, Ca hypothermia ```
27
causes of osborne/j waves
hypothermia SAH hypercalcaemia
28
what is the trio criteria for acute coronary syndrome
chest pain ecg change cardiac markers
29
ecg change in acute coronary syndrome
ST elevation >1mm in 2 contiguous leads with reciprocal ST depression and new LBBB
30
what is the p wave
atrial depolarisation
31
what is the PR interval
time it takes for electrical activity to move from atria to ventricles
32
what is the QRS
depolarisation of ventricles
33
what is the QT
time for the ventricle depolarisation and repolarisation
34
what is T wave
repolarisation of ventricles
35
what is the ST
time between depolarisation of ventricles and depolarisation of ventricles
36
artery involved for lateral
left circumflex
37
artery involved in anterior
left anterior descending
38
artery involved in inferior
right coronary artery
39
ECG in AF
tachycardia irregularly irregular absent p waves
40
causes of LBBB
CAD, HTN, HF, LVH
41
causes of RBBB
RVH, IHD, rheumatic heart disease, Fallots
42
what is 1st degree heart block
PR >200 but always followed by QRS
43
treatment of 1st degree heart block
benign - its just a delayed impulse
44
what is 2nd degree heart block mobitz 1 wenckebach
Progressively increasing PR until one p wave is not conducted
45
treatment of 2nd degree HB mobitz 1
only treat if Sx if Sx - use atropine pacemaking is rare
46
what is 2nd degree heart block mobitz 2
Constant PR then non conducted p wave | Usually 2:1 or 3:1 block
47
treatment of 2 degree heart block mobitz 2
pacemaker as more likely to be haemodynamically compromised
48
what is 3rd degree heart block
P waves and QRS waves independent of each other
49
treatment of 3rd degree heart block
urgent cardiac monitoring and pacemaker insertion
50
ECG in LBBB
Wide QRS >120 Top notched M in v5/6 T wave inversion in lateral leads V1 – dominant s wave
51
treatment of LBBB
thrombolysis
52
ECG in RBBB
Wide QRS RSR pattern in v1– up first wide slurred s in lateral leads
53
what is a bifascicular block
RBBB+LAD
54
what is trifascicular block
RBBB+LAFB+1st degree AV block
55
treatment of RBBB
invasive coronary angioplasty, treatment of HTN and Dmx
56
causes of AF
``` PIRATES PE ischaemia respiratory atrial enlargement (VSD, MS, MR) as alters muscle and causes defective atrial contraction thyroid ethanol sepsis ```
57
ECG in VF
shapeless, rapid oscillations, no organised complexes
58
ECG in flutter
tachycardia saw tooth normal QRS
59
ECG in WPW
delta wave wide QRS short PR
60
ECG in VT
monomorphic
61
ECG in torsades de pointes
polymorphic, similar to VT
62
ECG in Brugada
coved ST elevation v1-v3 | RBBB
63
ECG in digoxin toxicity
reverse tick down sloping ST depression T wave inversion
64
ECG in PE
tachycardia AF S1Q3T3 - deep s wave in lead 1 , q wave in lead 3, t wave inversion in lead 3
65
ECG in hyperkalaemia
tall tented t waves wide QRS absent p waves
66
ECG in hypokalaemia
flat t wave St depression prolonged QT u wave
67
ECG in bradycardia
<60bpm
68
what is a capture beat
SAN transiently captures ventricles to produce normal QRS