CVA/AVM Flashcards

1
Q

most common stroke in children

A

hemorrhagic stroke

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2
Q

who are at increased risk for pediatric stroke

A

boys and children of African-American descent

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3
Q

what are different types of stroke

A

arterial ischemic stroke, hemorrhagic stroke

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4
Q

clot that travels to the brain

A

embolus

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5
Q

clot that occurs in the brain

A

thrombus

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6
Q

narrowing or damaged arteries

A

arteriopathies

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7
Q

virus travels to cerebral arteries

A

post varicella arteriopathy

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8
Q

risk factors for arterial ischemic stroke

A

congenital heart disorder, sickle cell disease, head/neck trauma due to damage to blood vessels

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9
Q

abnormal shaped RBCs don’t travel through vessels typically and don’t carry oxygen correctly to the brain

A

sickle cell disease

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10
Q

focal deficits of arterial ischemic stroke and other signs

A
  • Focal: acute hemiparesis, diploplia, dysarthria
  • seizures, altered status, behavioral changes
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11
Q

management for arterial ischemic stroke

A
  • anticoagulation medicine
  • RTCs lacking use of tissue plasminogen activator (tPA) in children
  • supplemental oxygen is hypoxemic
  • hydration
  • monitor for fevers/infectious causes
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12
Q

causes of hemorrhagic stroke

A
  • rupture of blood vessel in brain
  • rupture of malformed vessels (AVM)
  • sickle cell
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13
Q

the risk of hemorrhage stroke is higher in individuals with

A

hemophilia (lack of blood clotting proteins)

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14
Q

signs/sx of hemorrhagic stroke

A
  • altered mental status
  • seizures
  • nausea/vomiting
  • older children: HA
  • abrupt onset of sx with neurologic deterioration
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15
Q

management of hemorrhagic stroke

A
  • ICP monitoring (external ventricular device)
  • prevention of cerebral edema
  • surgical management (hematoma evacuation)
  • watching for signs of hydrocephalus
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16
Q

tangled mess of blood vessels in the brain or spine

A

arteriovenous malformation (AVM)

17
Q

disruption of oxygenated blood to the brain - oxygenated blood in the arteries gets diverted to the veins, bypassing brain tissue

A

arteriovenous malformation

18
Q

what may patients have with AVM even though most go un-diagnosed until rupture

A

history of HA or present with HA/seizures

19
Q

etiology of AVM

A

considered congenital, males more affected than females

20
Q

movement analysis used to assess isolated joint movement vs synergy patterns

A

Brunnstrom stages of recovery

21
Q

no voluntary or reflex activity present

A

I - flaccidity

22
Q

synergy pattern begins to develop, may appear as associated reactions

A

II - spasticity begins to develop

23
Q

movement synergies can be performed voluntarily

A

III - spasticity reaches peak

24
Q

deviation from movement synergy is possible, limited combo of movements

A

IV - spasticity begins to decrease

25
Q

movement synergies less dominant, more complex movement possible

A

V - spasticity continues to reduce

26
Q

isolated and combo movement evident, coordination may be impaired

A

VI - spasticity essentially absent

27
Q

return to fine motor skills

A

VII - return to normal function