CP Flashcards
atelectasis shifts heart
toward same side
pneumothorax shifts heart
away
Left coronary artery divides into
Anterior descending
Left circumflex artery
What carries deoxygenated blood to lungs
Pulmonary arteries
Left coronary artery - anterior descending - supplies what
anterior portion of interventricular septum
Left coronary artery - circumflex - supplies
Left atrium
Post and lat walls of LV
An and inf wall of LV
Right coronary artery divides into
Sinus node artery
Right marginal artery
Posterior descending artery
Right coronary artery - sinus node supplies
Right atrium
Right coronary artery - right marginal artery - supplies
Right ventricle
Right coronary artery - posterior descending artery supplies
Inf walls of both ventricles
Inf portion of the interventricular septum
Extrinsic heart regulation
Vagus (dec HR - decrease conduction at AV) Upper thoracic (inc HR - accelerates d/c from SA to AV)
SA node
pacemaker
Inate (without vagal influence) - is 100-110 bpm
But vagal influence - 60bpm
AV node fires at
40-60 bpm
Ventricles fire at
bundle of his - bundle branches - purkinje fibers
20-40 bpm
P wave
Atrial depolarization
0.08 - 0.10
PR
time to pass through AV junction
Norm is 0.12 - 0.20
QRS
Depolarization of ventricles
0.04 - 0.10
T wave
repolarization of ventricles
QT
total time for deplarization and repolarization of ventricles
Less than or equal to 0.44
Lead 2 (most common) shows what
Depolarization from R to L heart in diagonal
Procedure for ECG eval
Evaluate P (atria, SA node) Evaluate PR (AV node) Evaluate QRS complex (ventricles) Evaluate QRS interval (ventricles) Evaluate T wave R-R interval (rate)
Heart blocks
1st = really long PR int
2nd type 1 = prog lengthened PR, drops every 4th
2nd type 2 = fixed long PR, drops every 2nd, 3rd, 4th
3rd = separate firing of atria and ventricle
Bundle branch blocks
Right - Rs go below isoelectric line
Left - Rs do not go below isoelectric line (mountain looking one)
Ischemia will show
ST segment depression
Myocardial injury will show
ST segment elevation
Box drawing for heart sounds
T between RA and RV, M between LA and LV
P after RV, A after LV
S1 at T and M
S2 at P and A
Dias above S1, Sys between, Dias below S2
S1 split
Heard at the bottom of the heart
Commonly due to RBBB
Tricuspid closing later than mitral
S2 split
Heard at the top of the heart
Can be normal in children
Pulmonary closing later than aortic
S3 occurs when
AFTER S2
Early ventricular filling after AV valves open
Due to diastolic distention and vibration of the ventricular walls
CHF
Heard best with bell
S4 occurs when
Right BEFORE S1
Rapid ventricular filling after the atrial kick
Ischemic heart disease, HTN
Pulm restriction
Trouble getting air in
Pulm obstruction
Trouble getting air out
Crackles
Low pitched on INSPIRATION
Ronchi
Low pitched on INSPIRATION and EXPIRATION
obstructive process
Wheezes
High pitched
bronchospasms
Pleural friction rub
Typically louder with inhale
Preload
The amount of stretch in the LV at the end of diastole
Afterload
Systemic vascular resistance
Amount of resistance the heart has to overcome to open the aortic valve and push blood volume into systemic circulation
Nitro will
Decrease preload and afterload by VD
Will also redistribute coronary blood flow to dec O2 demand of the heart
Sublingual in supine (used for angina)
Side effects - HA, dizzy, Ortho hypo
Ca channel blocker
Dec contractility, Dec HR, Dec BP
VD
Ace inhibitor
Prevents conversion of angiotensin 1 to angiotensin 2
Prevents Na and H20 retention - so dec afterload
Dec BP by dec arteriole constriction
“ril”
Dec release of aldosterone and ADH
Beta blockers
“lol”
Dec HR and contractility which dec O2 demand of heart
Dec CO
Will block beta 2 in rest of body too so BronchoCONstirction and dec BP with VC
Side effects = Bradycardia, chest pain, hypotension
Troponin elevates ___ and stays for ___
Troponin elevates in 1 to 2 hours after MI and stays up for 10 days
Myoglobin elevates quickly and then drops quickly after MI so not as good to look at as troponin
Congestive heart failure - tx
Fluid and Na restrictions
Diuretics (to dec preload)
Ace inhibitors, beta blockers, Na/fluid restrictions, VDs, Positive ionotropes (inc contractility)