Block 7 Flashcards
McKenzie
Peripheralization
Centralization
Postural syndrome - normal tissues
Dysfunction syndrome - mechanically shortened tissue (tx with mvmnt that produces pain)
Derangement syndrome - follows periph/cent principle (tx with cent mvmnt)
Mulligan
Therapist applied accessory mvmnts that are combined with physiological NEVER CAUSE PAIN MWM NAG - natural apop glide SNAG - sustained (with active movement)
Prokaryotes
no nucleus, no circular DNA, cell walls made with petidoglycan
gram positive vs. gram negaitve
Positive = purple, thick peptidoglycan layer Negative = red, thin layer - has outer membrane too though with LPS
Sterilization
Destroys all - even spores Moist heat (autoclaving) Dry heat Radiation Ethylene oxide gas Filtration
Cellulitis
Can’t tell involved vs. uninvolved tissue
Usually GAS or staph
MRSA = resistant to
betalactams (penicillin)
Hospital acquired
Tx with vancomycin
Viruses = need
Host for replication!
HSV
Has a latent period
Tx = acyclovir
HSV 1 vs. HSV 2
Oral = 1 Genital = 2
Varicella zoster
Varicella = chicken pox = primary Zoster = shingles = reactivation
Hep
A = fecal/oral - no long term prob, VACCINE B = BLOOD contact or sex, VACCINE C = BLOOD or sex, no vaccine!!!
HIV/AIDs
Mostly through sex
CD4 < 200 for diagnosis of AIDS
Tinea corporis
ring worm
Typically on trunk but can be anywhere
General targets for antibiotics
Cell wall/membrane
Protein synthesis
DNA/RNA synthesis
Beta lactams include
Penicillin
Cephalosporin
Monobactams
Carbapenems
A lot of bacteria become resistance to beta lactams
Cell wall inhibitors
Penicillin (hypersens) Cephalosporins Monobactams Carbapenams (hypoten) Vancomycin Bacitracin Polymyxin B
Protein synthesis inhibitors
Aminoglycosides (end in mycin - nephron/oto toxicity)
Tetracycline (impair growth, avoid in preg F and kids)
DNA/RNA synthesis inhibitors
Rifamycin
Sulfonamides
Trimethoprin
Fluoroquinolones (tendons)
Levator ani includes
Pubococcygeus
Puborectalis
Iliococcygeus
PFM includes
Levator ani and coccygeus
PFM - fast vs. slow
70% slow twitch and 30% fast
Obturator internus
ER
Palpate with pressure medially towards ischial tub
Piriformis
Above 60 flex is an IR and abd
Otherwise = ER
Sphincters - which is only in males
IUS - involuntary - contracts to prevent retrograde ejaculation
Sphincters
EUS - 70% invol, 30% volun (control with pee)
IAS - involuntary (20% resting tone)
EAS - voluntary - control defecation
WH - general innervations
Somatics = PFM and external sphicnters S2-4 Sympathetics = internal sphinc = STORE - T11 - L2 Parasymp = smooth mm relax (PEE) - S2 - S4
Anorectal angle
Puborecatlis maintains anorectal angle at 90 degrees
Increase angle = poop
Decreased angle = harder to poop
Normal amount of voiding in 24 hours
5-7
Mild = 7 to 9 with min to no urgency Mod = 10+ with urgency
Functional incontinence
Unable to reach restroom due to movement limitations
Preg - Estrogen functions to
Increase uterus/breast size
Relax ligaments
Preg - relaxin peaks
in 1st trimester and stays elevated
Inhibits uterine contraction
Relaxes ligaments
Preg - progesterone
smooth mm relaxation
Hyperventilation
Inc core temp
CV changes with preg
Blood volume inc 50%
HR, SV, CO increase
Inc venous pressure
Aorta partially occluded in supine
Pulmonary changes with preg
Stretching of ribcage out and up Inc secretions Inc hyperventilation Inc work of breathing Dyspnea earlier with exercise
MS changes
Abdominal mm stretched PFM damage Joint hypermobility Postural changes (inc kyphosis, GH IR,, pec tight, weak scap)
STRETCH GASTROCS FOR ALL PREG W
Stages of birthing
Labor
Delivery
Afterbirth
Always make sure to ask how long stage 2 was
Exercise and preg
HUGE FALL RISK!
But ex can overall be continued
Exercise and fitness leads to faster recovery and potentially healthier baby
Exercise and preg guidelines
Reg/mod intensity for 30 min or more dailry
Avoid more than 5 min of supine
Stop when fatigued - never ex to exhaustion
CRH -
ACTH - adrenal cortex - cortisol, aldosterone
Cushings (high)
Addisons (low)
TRH -
TSH - thyroid gland - T3 and T4
Hyperthyroidism (graves)
Hypothyroidism (hashimoto)
TRH also - PRL - breast tissue to increase milk production
GnRH -
FSH and LH - testes and ovaries
GHRH
GHIH
RH - inc GH
IH - dec GH
Gigantism
Acromegaly
Dwarfism
Steroid hormones
derived from cholesterol fat soluble Adrenal (cortisol, aldosterone) Gonadal (test, est, prog) Vit D
Bind to receptors inside the cell
Water soluble hormones
Catecholamines (from adrenal med - Epi, NE)
Bind to receptors on cell membrane
What inhibits prolactin release
Dopamine! (PIH)
Post pit
ADH/Vasopressin
Oxytocin
Thyroid disroders always have ____ effects
CARDIAC
Increase blood glucose levels = secrete
Insulin (from beta cells) to dec blood glucose
Decrease in blood glucose = secretion of
GLucagon (from alpha cells)
Type 1 DM
beta cells can’t produce insulin
Can see ketoacidosis - life threatening
Type 2 DM
Cells become insulin resistant Polyuria because of increased osmotic load in BV Hyperglycemia Plyphagia Tri neuropathy
PTH
activated release when Ca is low
Requires active vit D to promote Ca resorption from bone
Hypocalcemia - hypoPTH - hyperexcitability of the mm and nerves (tetany, CHF, seizures, arrhythmias)
Hypercalcema - hyperPTH - hypoexcitability of the mm and nerves (fatigue, weak, constipation)
Calcitonin
Released by thyroid gland when Ca is too high
Dec activity of PTH and dec bone resorption
Vit D
Have to have it to uptake Ca from the gut
Inc Ca and P concentrations in the plasma
Promotes PTH function of osteoclasts and bone resorption
Leukopenia
Dec WBC
below 1000 = risk for infection
below 500 = life threatening
Acute lymphoblastic leukemia
Most common leukemia in children
ALL
Acute myelogenous leukemia
Most common leukemia in adults
AML
Hodgkins lymphoma - diagnostic characteristic
Reed Sternberg Cells (Owl eyes)!!!
Very survivable , causes pruritis, no CNS effects, pain, night sweats, usually young men
Non hodgkins -
Not as predictable as HL
Might have neuro s/s
Spreads to visceral structures
Sickle cell anemia
Autosomal recessive
Abnormal form of hemoglobin
RBCs change shape as they de oxygenize
Most common anemia
Iron deficiency
Fat soluble vitamisn
A E K D Can be toxic
Cell types of the epidermis
Keratinocytes (primary)
Melanocytes
Langerhan
Merkel
Blister = damage to
Basement membrane
Dermis made of
Papillary and Reticular (deeper)
Epidermis made of (sup to deep)
Cornuem Lucidum (only palms and soles) Granulosum Spinosum Basale
Dermis cell types
Fibroblasts (primary)
Macrophages
Mast
Sweat glands
Apocrine - puberty
Eccrine - activity/temp
Stages of wound healing
Inflammatory (0 to 4 days)
Proliferation/Granulation (4 to 21 days)
Remodeling/Maturation (21 days to 2 months)
What pressure is required to obstruct blood flow in capillary bed
32 mmHg
Venous ulcer
Medial
Hemosiderin
Irregular wound margin
Lipodermatosclerosis
COMPRESSION
Arterial ulcer
Symmetrical margins Pain Dry Pale wound bed Diminished pulses Skin shiny, red, hairless
NO COMPRESSION! CHECK ABI
Non selective debridement
Mechanical (whirlpool, scrubbing, wet to dry, pulsed lavage)
Surgical
Non enzymatic
Take viable and non viable
Selective debridement
Sharp
Autolytic
Enzymatic
Zones with burns
Central zone of coagulation - tissue is dead
Zone of stasis - critical zone - can reverse with tx
Zone of hyperemia - zone will recover
Rule of nines - adult vs. child
Head = 9% Chest and back = EACH 18% Each arm = 9% Each leg = 18% Perineum = 1%
Child Head = 18%
Each leg is 13.5%
Short stretch bandage
Low resting, high acting working pressure
Long stretch bandage
Low acting, high resting
Buoyancy
Archimedes principle
When immersed in water, body will experience an upward thrust equal to the weight of fluid that was displaced
Center of B = T11
Unloading with water depths
C7 - 90% unloaded
Xiphoid - 60%
Umbillicus - 50%
Relative density - body vs. water
Body is less than water so we tend to float
Fat people float more than muscular people
Fat (0.8) Muscle (1.5)
Hydrostatic pressure
Pascals law
When body is immersed in water, there is a pressure exerted by the fluid on the tissues at rest
The deeper the body part, the greater the pressure
Hydrostatic vs. venous pressure
Hydrostatic pressure greter than venous = will decrease edema
Can also make it harder to breathe
Inc SV and CO
Viscostiy
Friction between water molecules that cause resistance to flow
Creates turbulence
Faster you move in water - the more resistance
Contraindications to aquatics
Tubes/lines/trach Open wounds Environmental communicable disease Ejection fraction less than 25% Vital capacity less than 1.5 L Immuno compromised Seizures Renal disease