Common Pathologies Of UL 2 Flashcards

1
Q

Adhesive capsulitis

A

Formation of excessive scar tissue or adhesions across GHJ
Leads to pain, stiffness and dysfunction
Primary (idiopathic)- spontaneous
Secondary - post trauma

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2
Q

Risk factors for adhesive capsulitis

A
Females>males
>40 
Trauma 
Diabetes
Hyperthyroidism 
Cerebrovascular disease
Coronary artery disease 
HLA P-B27 positive
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3
Q

Stages of adhesive capulitis

A

1) shoulder pain mainly at night
Arthroscopically- synovitis no adhesions
Inflammatory cell infiltration of synovium
2) stiffness development
Arthroscopically- synovitis, loss of axillary fold
Synovial proliferation
3) loss of ROM, pain at EOM
Arthroscopically- resolved synovitis, adhesions, axillary fold obliterated.
Dense collagenous tissue within capsule
4) chronic stage- persistent stiffness, minimal pain.
Resolved synovitis, advanced adhesions

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4
Q

Clinical presentation of adhesive capsulitis

A

Pain

Gradual loss of ROM

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5
Q

Management of adhesive capsulitis

A
Physio- early mobilisation
Education
Corticosteroid injections
Hydrodilation
NSAIDS
Surgery-MUA and Capsular release
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6
Q

Types of fractures

A
Transverse
Linear
Oblique non-displaced
Oblique displaced
Spiral
Greenstick
Communited
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7
Q

Clavicle fractures

A

Result from fall
Neuro/vascular structures nearby
Mid-shaft>lateral>medial

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8
Q

Clavicle fracture management

A

Conservative- physio and sling

Surgical

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9
Q

Proximal humeral fractures and management

A

Classified on number of fragments displaced

Collar and cuff 2-3/52
Progressive active management
Surgery

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10
Q

Distal radius fracture

A

Colles fracture- extra articular, dorsally displaced
Smiths fracture- anteriorly displaced
Bartons fracture- intra articular, associated dislocation of RCJ

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11
Q

Distal radius fracture management

A

Physio- immobilisation

Splints, cast, k wires, MUA

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12
Q

Scaphoid fracture

A

FOOSH
waist>proximal pole>medial pole
Risk of AVN and non union
Pain over anatomical snuff box

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13
Q

Scaphoid fracture management

A

Cast, surgery, physio after immobilisation

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14
Q

Osteoarthritis

A

Can develop in any synovial joint

Common in knees, hips and small joints of the hand

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15
Q

Osteoarthritis management

A

Physio
Corticosteroid injection
Surgery (joint replacement, debridement)

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16
Q

Rheumatoid arthritis

A

Systematic autoimmune disease- inflammatory with extra articular involvement
Synovium infiltrated by immune cells
Fibroblasts and inflammatory cells lead to osteoblast generation resulting in bone and loss of joint integrity

17
Q

Epidemiology of osteoarthritis

A

F>M
Increases with age
Paediatric- juvenile idiopathic arthritis

18
Q

Risk factors of rheumatoid arthritis

A
Genetics
Smoking
Air pollution
Obesity
Low vitamin D
19
Q

Rheumatoid arthritis clinical presentation

A

Polyarthritis small joint in hand- PIP, MCP, RCJ
Insidious onset
Joint stiffness in morning, fatigue, deformity, pain, weakness, restricted mobility
Cervical spine- instability between C1-2

20
Q

Management of rheumatoid arthritis

A

Symptom management
Pharmacological- DMARDS
nutrition
Physio

21
Q

Shoulder dislocation

A

Anterior- arm in apprehension position (abd and ext)
Humeral head displaces antero- inferiorly
Can result in Hills Sachs lesions or Bankart lesion
Concurrent RC injuries can occur

Posterior- blow to ant shoulder, can occur in seizure
Concurrent injuries to RC (subscap) and post labrum

22
Q

Shoulder instability

A

Disruption of static and dynamic GHJ stabilisers
Static- articular conformity, negative intra art pressure, labrum, GH ligs
Dynamic- RC and scapula stabilisers
Traumatic or atraumatic
Ant,post, inf, multidirectional

23
Q

Shoulder instability clinical presentation

A
Clicking/pain
Sub acromial signs
RC signs
Positive apprehension tests/Relocation test
Increased accessory motion at GHJ
postive sulcus signs
24
Q

Shoulder instability management

A
Physio- education 
Motor control
Strength training
Proprioceptive training
Surgery- depends of structural impairments
25
Q

Dupuytrens disease

A

Nodular hypertrophy and contracture of superficial palmer fascia
Flexion contracture of MCP and PIP joints
Slowly, progressive
Thickening of skin, bands of fibrotic tissue form in palmer area
Occurs bilaterally

26
Q

Aetiology of dupuytrens disease

A

M>F
Genetics
Environmental factors: smoking, alcohol, manual labour, low bmi, anticoagulants
Associated with diabetes, epilepsy, HIV, adhesive capsulitis, cancer

27
Q

Surgical management of dupuytrens disease

A

Simple fasciotomy- contract cord cut
Fasciotomy- partial/complete removal of diseased palmer fascis and cord
Dermofasciotomy- removes underlying skin and fat, full thickness skin graft to cover surgical site
Amputation of digits

28
Q

dupuytrens disease physio treatment

A
Post operative
Splinting
Exercise- passive stretching, active exercises, function
Education and advice
Oedema and scar tissue management