Common Pathologies LL 1 Flashcards
When are avulsion injuries most common
Adolescence as tendons are stronger than apophyses
Avascular necrosis
Loss of blood supply to bone
Bone death then collapse
Hip stiffness, pain, limp, groin, buttock and or thigh pain
Hip dysplasia
Normal centre edge angle is 25-40°
< 25-30 borderline dysplasia
< 20 dysplasia
<16 certain osteoarthritis
CAM type FAI hip impingement
Irregular osseous prominence of proximal femoral neck
Symptomatic in physically active young males
Bony protrusion located at the anteriosuperior aspect of femoral head neck junction
Pincer type FAI hip impingement
Result of excess acetabular coverage of femoral neck
Over coverage= global (coxa profunda) or focal anteriorly (acetabular retroversion)
Rim presses up against labrum
Symptom summary of FAI hip impingement
Pain/difficulty crossing legs Difficulty putting socks/shoes on Unable to sit for long periods Limp Adductor problems Painful walking long distances Pain after sport activities Buttock/lower back pain
Greater trochanter pain syndrome (GTPS)
Common cause lateral hip pain
Tendopinopathy of gluteus medius/minimus/bursal pathology
Compressive forces cause impingement of these into greater trochanter by ITB
Typical patients of GTPS
Female 40-60
Post menopause
Lower femoral neck shaft angle
Increased BMI
Snapping hip syndrome- coxa saltons
Internal- iliopsoas over iliopectineal eminence, paralabral cyst
External- ITB snapping over trochanter
Proximal hamstring tendon rolling over Ischial tuberosity, TFL, Gluteus maximus over greater trochanter
Osteochondral defect
Focal area of damage to articular cartilage and piece of underlying bone
Osteochondral defect acute/develop chronically
Separation of osteochondral fragment casued by acute traumatic injury or unstable fragment in osteochondritis dissecans
Acute- impaction of bone with resultant contour deformity
Collapses of subchondral bone in subchondral insufficiency fracture (SIF) or avascular necrosis (AVN) or bone collapse uncovering lsrge subchondral cyst
Subchondral cyst
Synovial fluid intrusion theory: articular surface defects and increased intra Articular pressure allow intrusion of synovial fluid into bone forming cavities
Bone contusion theory: non communicating cysts arise from subchondral foci of bone necrosis, results in opposing articular surfaces coming in contact with one another