Chapter 2 Flashcards
Tetracycline staining
Intrinsically permanently stained - incorporated by ameloblasts
Turner’s Hypoplasia
Periapical inflammatory disease of overlying deciduous
Enamel defects of the permanent teeth
Permanent Bicuspids
Dental Fluorosis
Excess amounts of fluoride result in significant enamel defects
During tooth development (2nd and 3rd years), don’t use fluoridated toothpaste
Optimum fluoridation of drinking water –> .7ppm
Congenital Syphillis
Rare
Hutchinsons incisors
Mulberry molars
Mulberry Molars
*Congenital Syphillis*
Hutchinson’s Incisors
*Congenital Syphillis*
Hutchinson’s Triad
Hutchinson’s Teeth
Interstitial Keratitis (corneal scarring)
8th nerve deafness
Saddle nose (may manifest in leprosy as well)
Abrasion
Erosion
Abfraction
Impacted Teeth
Mandibular 3rd molars > Maxillary 3rd molars > Maxillary Canines
Ankylosis
No PDL
Fuses to the bone – WILL NOT ERUPT
Failure of the tooth to reach the occlusal plane
Primary or permanent
Anodontia
Lack of tooth development
Occur in the presence of Hereditary hpohidrotic ectodermal dysplasia (absence of ectodermal tissue development)
Hypodontia
Lack of one ore more teeth
Abscence of deciduous tooth –> Likely to miss associated permanent tooth (develop from the same dental lamina)
Ectodermal Dsyplasia –> Conical shaped teeth
Oligodontia
Lack of 6+ teeth
Hyperdontia
Supernumerary teeth
Mesiodens
supernumerary tooth that develops in the MAXILLARY incisal region between centrals
Dental Transposition
Teeth flip positions and grow not in the normal location
Rege Fede Disease
Traumatic ulcerations of the adjacent soft tissue from neonatal teeth that may occur during breast feeding
Gemination
Single enlarged tooth or joined teeth in which the tooth count is normal when the anomalous toot is counted as one
single pulp chamber
Fusion
Single enlarged tooth or joined teeth in which the tooth count reveals a missiong tooth when the anomalous tooth is counted as one
Two different pulp chambers
Concrescence
Union of two adjacent teeth by cementum alone without confluence of underlying dentin
Dilaceration
A bend in the tooth – usually distally
Mandibular 3rd molars
Dens Evaginatus
External growth of the tooth
PREMOLAR teeth
100% prevalence in NATIVE AMERICANS
May have a pulp horn
Dens - in Dente (Dens invaginatus)
Deep surface invagination of the crown or rooot that is line by enamel
Permanent LATERAL incisors
Taurodontium
Elargement of the body and pulp chamber of a multi-rooted tooth
Apical displacement of the pulpal floor
Isolated or Syndromic
Syndromes:
* Kleinfelter’s syndrome (XXY)
* Amelogenesis imperfecta
* Trich-dento-osseous syndrome
Hypercementosis
Non neoplastic depositio of excessive cementum - continuous with radicular cementum
Threat –> for extraction of tooth
Local factors: Occlusal trauma, Adjacent inflammation
Systemic Factors: Paget’s Disease of bone
Paget’s Disease of bone
Associated with generalized hypercementosis
Amelogenesis Imperfecta
AD inherited
Developmental alterations of the structure of enamel
No systemic disorder
Permanent and Primary teeth affected
Hypoplastic Amelogenesis Imperfecta
Inadequate deposition of the enamel matrix
Pinpoint pitting on tooth surface
Hypomaturation amelogenesis imperfecta
Enamel matrix is laid down
Defect in maturation of the enamel’s crystals
Snow-capped white opaque enamel on incisal or occlusal 1/3
Hypocalcified amelogenesis imperfecta
Matrix is laid down and teeth are normal shape
Enamel is soft ant is easily lost
Enamel is yellow-brown or orange - rapid calculus apposition
Dentinogenesis Imperfecta
Mutation of the DSPP - dentin sialophosphoprotein gene
Primary or permanent.
Blue to brown discoloration with distinctive translucence
Xray: Bulbous crown, cervical constriction, thin roots, early obliteration of pulp chambers
Shell teeth –> normal thickness enamel, enlarge pulp chamber
Dentinogenesis imperfecta
Shell tooth
Dentinogenesis imperfecta
Dentin Dysplasia
Type I and Type II
Autosomal dominant
Dentin Dysplasia Type I
Rootless Teeth
Clinically normal – usually mobile
Enamel and coronal dentin are normal
Radicular dentin looses organization and is shortened dramatically
Dentin Dysplasia Type 2
Blue-Amber Brown translucence
Root length is normal
Altered pulp anatomy – thistle tube shaped teeth, pulp stones
Regoinal Odontodysplasia
Localized, NON HEREDITARY abnormality of teeth
None of tooth tissues form correctly
“GHOST TEETH”
Usually one quadrant
Clinically –> NO TEETH PRESENT
Aminacycline Staining
Causes blue roots or dark crown
Typically found in 3rd molars
(medication taken for acne)
Perimolysis
Gastric Secretions
Related to erosion
