ch5-fats Flashcards

1
Q

What does level of saturation refer to

A

Saturation refers to how many hydrogen atoms surround each carbon

Saturated fatty acids can pack tightly together

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2
Q

Monosaturated vs polyunsaturated fatty acids

A

mono- Lacks hydrogen atoms in one region (have one double bond)

Poly- Lack hydrogen atoms in multiple locatons, have two or more double bonds

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3
Q

what is hyrogenation

A

the addition of hydrogen atoms to unsaturated fatty acids

-converts liquid fats into semisolid/solid forms

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4
Q

What are phosoliped composed of

A

Glyerol backbone
2 fas
Phosphate

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5
Q

where does digestion of fats begin

A

in small intestine

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6
Q

what enzyme in mouth begins breakdown of fats in mouth

A

Lingual lipase

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7
Q

What is the effect of the gallbladder on fat digestion (what stims it and what does it release)

A

CCk and secretin are released from mucosal cells and stim gallbladder to contract and release bile

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8
Q

what is the effect of bile and pancreatic enzymes on fate

A
  • Bile disperses fat into smaller fat dropleta

- Pancreatic enzymes break triglycerides into 2 seperate fatty acids and a monoglyceride

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9
Q

What does fat enter the mucosal cell as

A

micelle

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10
Q

What happens in the intestinal mucosal cell to fats

A

FAs are reattached to monoglyceride to reform triglycerides

-Small amount of pro added to form chlyomicron

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11
Q

Why are short and medium chain fatty acids absorbed more quickly

A

becuase they are not arranged into chylomicrons

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12
Q

what does lipopro lipase do

A

Once chylomicron gets to cell in the body it is disassembled into 2 fas and monoglyceride before they enter. after entering they reassemble

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13
Q

What are the fat soluble vits

A

ADEK

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14
Q

What are the 2 essential fas

A

Linolic Acid (omega 6)- converted to arachidonic acid

Alpha linoleic acid (omega 3)- converted to EPA and DHA which are important regulators of inflamation/blood clotting/BP

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15
Q

AMDR of fat

A

20-35%

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16
Q

Modifiable risk factors for cardiovascular disease

A
  • being overweight
  • Physical inactivity
  • smoking
  • diabetes
  • inflamation
17
Q

Monosaturated vs polyunsaturated fatty acids

A

mono- Lacks hydrogen atoms in one region (have one double bond)

Poly- Lack hydrogen atoms in multiple locatons, have two or more double bonds

18
Q

what is hyrogenation

A

the addition of hydrogen atoms to unsaturated fatty acids

-converts liquid fats into semisolid/solid forms

19
Q

What are phosoliped composed of

A

Glyerol backbone
2 fas
Phosphate

20
Q

where does digestion of fats begin

A

in small intestine

21
Q

what enzyme in mouth begins breakdown of fats in mouth

A

Lingual lipase

22
Q

What is the effect of the gallbladder on fat digestion (what stims it and what does it release)

A

CCk and secretin are released from mucosal cells and stim gallbladder to contract and release bile

23
Q

what is the effect of bile and pancreatic enzymes on fate

A
  • Bile disperses fat into smaller fat dropleta

- Pancreatic enzymes break triglycerides into 2 seperate fatty acids and a monoglyceride

24
Q

What does fat enter the mucosal cell as

A

micelle

25
Q

What happens in the intestinal mucosal cell to fats

A

FAs are reattached to monoglyceride to reform triglycerides

-Small amount of pro added to form chlyomicron

26
Q

Why are short and medium chain fatty acids absorbed more quickly

A

becuase they are not arranged into chylomicrons

27
Q

what does lipopro lipase do

A

Once chylomicron gets to cell in the body it is disassembled into 2 fas and monoglyceride before they enter. after entering they reassemble

28
Q

What are the fat soluble vits

A

ADEK

29
Q

What are the 2 essential fas

A

Linolic Acid (omega 6)- converted to arachidonic acid

Alpha linoleic acid (omega 3)- converted to EPA and DHA which are important regulators of inflamation/blood clotting/BP

30
Q

AMDR of fat

A

20-35%

31
Q

Modifiable risk factors for cardiovascular disease

A
  • being overweight
  • Physical inactivity
  • smoking
  • diabetes
  • inflamation