Ch13: Shock Flashcards
what is shock
inadequate cellular perfusion
perfusion
the circulation of blood to tissues to meet the cells’ needs and remove waste
perfusion triangle
heart
vessels
blood
how does the body compensate for hypoperfusion?
it redirects blood flow from organs that can withstand more time without oxygen such as skin and intestines
how to calculate pulse pressure
systolic – diastolic pressure
what does a wide pulse pressure tell us?
the heart is working harder, arteries have become less flexible or both. very high pulse pressures may indicate heart disease
what are sphincters in the capillary bed controlled by?
autonomic nervous system
factors affecting adequate perfusion
1) adequate blood flow
2) adequate gas exchange and oxygenation of the blood
3) adequate glucose level
4) adequate removal of waste
(think ab aerobic respiration)
which part of the nervous system controls the flight or flight response?
sympathetic (part of autonomic peripheral NS)
3 causes of shock (hint: perfusion triangle)
- pump failure - cardiogenic shock, and obstructive shock (like pulmonary embolism, cardiac tamponade, tension pneumothorax)
- poor vessel function + excessive vasodilation - distributive shock like sepsis, anaphylactic shock, neurogenic shock, psychogenic shock)
- low fluid volume - hypovolemic shock (including hemorrhagic and non-hemorrhagic shock)
4 types of shock
cardiogenic
obstructive
distributive (sepsis, anaphylactic, neurogenic, psychogenic)
hypovolemic (hemorrhagic and non-hemorrhagic)
how does cardiogenic shock –> pulmonary edema
cardiogenic shock damages heart muscle so it does not pump as well. because of the less efficient pumping, blood is backed into the pulmonary vessels. this pressure pushes fluid into the alveoli, causing pulmonary edema and inefficient oxygenation (tachypnea, crackles, rales).
*note: edema = excessive fluid build up
myocardial contractibility
ability of the heart to contract
preload
the pressure increase as blood fills the heart during diastole
blood enter the heart in diastole –> stretch walls + preload –> ? myocardial contractibility
increases myocardial contractibility
afterload
the pressure/force of the heart when pumping out the blood (systole)
consequences of increased afterload
heart overworks –> heart failure in the long term (similar to the idea of wide pulse pressure)
factors that lead to cardiogenic shock
low cardiac output (stroke volume x HR)
high preload
low preload
poor myocardial contractility
pericardial effusion
fluid going into pericardial sac
pericardial tamponade / cardial tamponad
excessive build up of fluid in pericardial sac, preventing blood from filling up ventricles
signs and symptoms of cardiac tamponade
BECK TRIAD
1) jugular vein distention
2) muffled heart soudns
3) narrowing pulse pressure (why? because heart cannot pump properly in systole and and there is A LOT of preload pressure during diastole because of resistance from fluid in pericardial sac when blood tries to flow into the ventricles)
pulmonary embolism
fluid bulid up
signs of neurogenic shock
no sweat below injury site
hypothermia
hypotension
skin pink, warm and normal