CH 11 Lecture Flashcards
Tidal Volume
Dead space
Air that doesn’t make it into your lungs
Exhalation
Passive. Diaphragm and intercostal muscles relax, and smaller thorax compresses air into the lungs
Hypoxia
Not enough oxygen
Regulation of ventilation - mechanism
PH changes in the blood and cerebrospinal fluid
Hypoxic drive
Breathing based on low oxygen. Typically seen in patients with end-stage COPD
Oxygenation
Process of loading oxygen molecules onto hemoglobin molecules in the bloodstream
Respiration
Actual exchange of oxygen and carbon dioxide in the alveoli and tissues
Internal Respiration
Exchange of oxygen and carbon dioxide between . . .
External respiration
Ventilation/Perfusion ratio and mismatch
Air and blood flow must be directed to same place at same time
Ventilation and perfusion must be matched
Failure to match can cause gas exchange not to take place -> lack of O2 in blood stream, CO2 is recirculated in bloodstream, hypoxia occurs
Intrinsic vs Extrinsic factors
External vs internal factors
Examples: internal: pneumonia, COPD. External: partial pressure of oxygen in atmosphere
pneumothorax
Open pneumothorax
Hemothorax
Hemopneumothrax
Hypovolemic Shock
Vasodilatory shock
AGPs
Aerosol Generating Procedures. Include CPR, Nebulizer treatments (Nebs), Endotracheal intubation, continuous positive airway pressure
Recognizing adequate breathing
12-20 breaths/min
Regular pattern of inhalation and exhalation
Regular, equal chest rise and fall
Adequate depth (tidal volume)
Clear and equal lung sounds
Abnormal breathing signs
Less than 12 breaths/min
more than 20 breaths/min
Irregular rhythm
Diminished, absent, or noisy auscultated breath sounds
Reduced flow of expired air at nose and mouth
Unequal or inadequate chest expansion
Increased effort of breathing
Shallow depth
Skin that is pale, cyanotic, cool, or moist
Skin pulling in around ribs or above clavicles during inspiration–this indicates use of accessory muscles
Cheyne-Stokes breathing
irregular respirations followed by a period of apnea. Often seen in patients with head injuries
Agonal Gasps
Appearance of breathing after heart is stopped
Ataxic respirations
Irregular or unidentifiable pattern
Often associated with head injuries
Kussmaul respirations
Deep, rapid respirations
Common in patients with metabolic acidosis
Measuring maximal CO2 at the end of an exhaled breath: Low CO2 level
Hyperventilation
Decreased CO2 return to the lungs
Reduced CO2 production at the cellular level
Measuring maximal CO2 at the end of an exhaled breath: High CO2 level
Ventilatory inadequacy
Apnea