Cards Flashcards

1
Q

Origin of ascending aorta and pulmonary trunk

A

truncus arteriosus

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2
Q

What does truncus arteriosis become

A

ascending aortia and pulmonary trunk

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3
Q

What does bulbus cordis become?

A

smooth parts (outflow tract) of LV and RV

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4
Q

Origin of RV and LV outflow tract (smooth parts)

A

bulbus cordis (“your bulbs are smooth, baby”)

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5
Q

Origin of atrial septum, membranous interventricular septum, AV and semilunar valves (VALVES AND SEPTUMS)

A

endocardial cushions

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6
Q

What do endocardial cushions become

A

atrial septum, membranous intraventricular septum, all the valves

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7
Q

What is the origin of trabeculated part of left and right atria

A

primitive atrium

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8
Q

What does the primitive atrium become

A

trabeculated part of left and right atria

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9
Q

What does the primitive ventricle become?

A

trabeculated part of left and right ventricles

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10
Q

Origin of trabeculated part of left and right ventricles

A

primitive ventricle

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11
Q

What is the origin of the smooth part of left atrium

A

primitive pulmonary vein

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12
Q

Primitive pulmonary vein becomes….

A

smooth part of left atrium

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13
Q

origin of coronary sinus

A

left horn of sinus venosus

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14
Q

what does left horn of sinus venosus become

A

coronary sinus

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15
Q

what does right horn of sinus venosus become

A

smooth part of right atrium

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16
Q

origin of smooth part of right atrium (sinus venarum)

A

right horn of sinus venosus

17
Q

origin of SVC

A

right common cardinal vein and right anterior cardinal vein

18
Q

What do the right common cardinal vein and right anterior cardinal vein become?

A

svc

19
Q

allantois->urachus becomes

A

medIAN umbilical ligament

20
Q

ductus arteriosus becomes

A

ligamentum arteriosum

21
Q

ductus venosum becoems

A

ligamentum venosum

22
Q

foramen ovale becomes

A

fossa ovalis

23
Q

notochord becomes

A

nucleus pulposus

24
Q

umbilical arteries becomes

A

mediAL ubilical ligaments

25
Q

umbilical vein becomes

A

ligamentum teres hepatis (round ligament) contained in falciform ligament

26
Q

murmur that gets louder with inspiration

A

tricuspid regurg

27
Q

murmur that gets louder with valsalva and standing up

A

HOCM and MVP (valsalva and standing decreases intensity of most murmurs)

28
Q

murmur that gets better (less loud) with hand grip

A

HOCM and AS (increases the intensity of most murmurs because it increases afterload)

29
Q

murmurs that get worse with rapid squatting

A

As, MR, VSD

30
Q

murmur that gets less intense with rapid squatting

A

rapid squatting increases afterload and preload so

HOCM murmur gets better with this while most murmurs get louder

31
Q

Asian male with ECG pattern of pseudo heart block with ST elevations in V1-V3…

A
brugada syndrome (AD)
increased risk for ventricular tachyarrhthmia and SCD) treat with implantable ICD
32
Q

Drugs that cuase long QT syndrome

A
"Anti-ABCDE"
Antiarrhythmics - class IA and III
antiBiotics - macrolides
antiCychotis - haloperidol
antiDepressants - TCAs
antEmetics - ondansetron

al of these increase risk for torsades

33
Q

saw tooth appearance on ECG

A

atrial flutter

34
Q

progressively increasing PR interval until dropped beat

A

second degree heart block type 1 mobitz (usually asymptomatic)

35
Q

stable PR interval until dropped beat

A

second degree heart blck type 2 (may progress to 3rd degree heat block so treat with pacemaker)

36
Q

atria and ventriicles beat independently of one another (atrial rate > venricular rate)

A

third degree heart block (treat with pacemaker, can be caused by lyme dx)

37
Q

MOA atrial natriuertic peptide

A

responds to increases in blood volume, and acts via cGMP.
causes vasodilation and decreased Na reabsorption in collecting tubule….dilates afferent renal ateriorle and constricts efferent arteriold promoting diuresis (“aldosterone” escape mechanism)

38
Q

differentiate BNP and atrial (ANP)

A

BNP is secreted by ventricular myocytes while ANP is from atria
BNP has longer half life making it good diagnostic tool for HF