Cardiovascular Flashcards

1
Q

start of CV01

how does the heart beating too fast lead to poor performance?

A

impaired filling > impaired output

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2
Q

what is the most likely cardiac cause of poor performance

A

arrhythmias

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3
Q

name 3 ECGs that should be done to investigate arrhythmias

A
  1. resting
  2. exercising
  3. prolonged/Holter
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4
Q

name 2 common sites of pathological block (very rare) leading to poor performance and collapse

A
  1. sinus node
  2. AV node
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5
Q

what HR is considered bradycardia?

A

less than 24 bpm

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6
Q

name 4 treatments for pathological bradycardia

A
  1. glucocorticoids
  2. parasympatholytics
  3. Sympathomimetics
  4. Transvenous pacemaker
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7
Q

name 2 parasympatholytics that can be used to treat pathological bradycardia

A
  1. atropine
  2. hysocine
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8
Q

name 2 sympathomimetics that can be used to treat pathological bradycardia

A
  1. isoproterenol
  2. clenbuterol
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9
Q

these are premature beats;
atrial, ventricular or junctional (AV node) origin;
cause poor performance if frequent or deteriorate into sinister rhythm

A

ectopy

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10
Q

what origin of ectopy is more concerning than others?
more likely to cause sudden death

A

ventricular

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11
Q

this is when an early beat is preceded by P wave;
may see unconducted early P waves;
common over 24h, most cases benign;
risk of triggering atrial fibrilation (AF)

A

premature atrial complexes (PAC)

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12
Q

these re premature beats NOT preceded by P wave;
wide and bizarre morphology

A

premature ventricular complexes (PVC)

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13
Q

this is one of the most common clinically significant arrhythmias;
~20% reduced potential of cardiac output;
irregular rhythm, variable pulse quality

A

atrial fibrillation

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14
Q

name 3 ECG characteristics of atrial fibrillation

A
  1. irregular RR intervals (‘normal’ QRS)
  2. absent P waves
  3. ‘f’ waves
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15
Q

name 2 treatment options for atrial fibrillation

A
  1. quinidine sulphate
  2. transvenous electrocardioversion (TVEC)
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16
Q

what is the dosing for quinidine sulphate for treatment of atrial fibrillation

A

20-22 mg/kg intra-stomach q2h

(max 6 doses)

17
Q

name the 5 most common complications caused by quinidine sulphate

A
  1. dull demeanour
  2. flatulence
  3. colic
  4. diarrhoea
  5. tachycardia
18
Q

start of CV02

name 2 types of physiological cardiac murmurs
(associated with normal blood flow)

A
  1. ejection
  2. filling
19
Q

name 3 types of pathological cardiac murmurs
(associated with abnormal blood flow +/- cardiac abnormalities)

A
  1. regurgitation
  2. stenosis
  3. abnormal communications
20
Q

name 2 common types of Left side SYSTOLE murmurs

A
  1. aortic ejection
  2. mitral regurgitation
21
Q

name 2 common types of Right side SYSTOLE murmurs

A
  1. tricuspid regurgitation
  2. VSD
22
Q

name 2 common types of DIASTOLE murmurs

A
  1. aortic regurgitation
  2. ventricle filling
23
Q

name the type of heart murmur

short;
never reachthe second heart sound;
crescendo decrescendo shape

A

mitral regurgitation

24
Q

name 6 causes of mitral regurgitation

A
  1. athletic horse
  2. thickening/fibrosis
  3. endocarditis
  4. ruptured cordae tendinae
  5. dilation of valve annulus
  6. dysplasia (congenital)
25
Q

name the type of heart murmur

long murmur;
typically holodiastolic;
commonest site for valve pathology;
degenerative lesions;
bacterial endocarditis

A

aortic regurgitation/insufficiency

26
Q

name the type of heart murmur

commonest congenital cardiac defect;
usually membranous portion of the septum (subaortic);
prognosis depends on size and location of defect and maximal velocity of the shunt;
CANNOT tell severity by noise intensity

A

VSD
(ventricular septal defect)