Cardiopulmonary Diseases Flashcards

1
Q

Cardiopulmonary system

A
  • lungs
  • heart
  • pulmonary artery
  • main function = to regulate blood flow between the heart and the lungs
  • if there is not enough oxygen in the body, literally the organs start to die
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2
Q

Respiratory system

A
  • nose
  • throat
  • trachea
  • bronchial tubes
  • diaphragm
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3
Q

Lungs

A
  • 2 balloon-like organs in the chest cavity
  • spongelike tissue
  • right lung = 3 lobes
  • left lung = 2 lobes
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4
Q

Pleura

A
  • membrane surrounds the lungs
  • separates lungs from the chest wall
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5
Q

How the lungs work

A
  • oxygen is inhaled into the trachea = diaphragm muscle pulls down the chest cavity to create a vacuum for air to enter
  • oxygen goes through the bronchial tubes to alveolar sacs
  • oxygen enters the blood stream to the cells
  • oxygen is exchanged for carbon dioxide
  • blood stream carries carbon dioxide back to the lungs
  • carbon dioxide is exhaled
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6
Q

Respiratory system

A
  • plays an important role in protecting the body from irritants
  • inhaled irritants are expelled through coughing and sneezing mechanisms
  • the epiglottis prevents foreign materials from entering the air passages that lead to the lungs
  • inhaled air is warmed and moistened for optimal humidity within the nose and mouth
  • inhaled air is cleaned by mucus = a sticky substance that lines the airway structures and catches foreign matter like dust, germs, and other irritants
  • cilia = hairlike structures that move like waves within the airway, remove the mucus up and out of the system
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7
Q

Heart

A
  • fist-sized organ made of muscular cardiac tissue that expands and contracts approximately 100,000 times a day, pumping about 2,000 gallons of oxygen and nutrient rich blood throughout the body
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8
Q

Chambers of the heart

A
  • 2 upper chambers (atria) = receive blood from the veins
  • 2 lower chambers (ventricles) = pump blood out of the heart through the arteries
  • the atria and ventricles work in tandem, contracting and relaxing to pump blood out of the heart
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9
Q

Septum (heart)

A
  • muscular wall that separates the left and right sides of the chambers
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10
Q

Valves (heart)

A
  • prevent blood from flowing in the wrong direction
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11
Q

Sinus node (heart)

A
  • nestled in the wall of the right atrium
  • pacemaker
  • sends out an electrical signal causing each chamber to contract in rhythm
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12
Q

One complete heartbeat

A
  • made up of 2 phases
  • systole = ventricles contract and pump blood into the aorta and pulmonary artery
  • diastole = ventricles fill in with blood
  • the systemic pathway transports blood from the heart to all other parts of the body and back again
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13
Q

Pulmonary circulatory system

A
  • loops the heart with the lungs
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14
Q

Path of blood flow through the heart

A

Deoxygenated blood:
- superior and interior vena cava
- right atrium
- tricuspid valve
- right ventricle
- pulmonary valve
- pulmonary arteries
- lungs
Oxygenated blood:
- pulmonary veins
- left atrium
- mitral valve
- left ventricle
- aortic valve
- aorta
- body

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15
Q

Cardiopulmonary vital signs

A
  • the routine of monitoring vital signs by health care professionals is critical when working with clients who have been diagnosed with a cardiopulmonary condition
  • vital signs can tell you info about the patient
  • pulse or heart rate (HR)
  • respiratory rate (RR)
  • blood pressure (BP)
  • oxygen saturation (O2 sat)
  • temperature
  • vital signs can help detect potential medical issues as well as monitor how the body is tolerating activities
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16
Q

Pulse rate

A
  • pulse (heart rate) = the number of times a heart rate beats in one minute (BPM)
  • can vary from person to person
  • normal range (adults) = 60-100 BPM
  • typically lower at rest
  • increases with exercise = more oxygen-rich blood is required with increased activity demand
  • carotid (infant) = between the bicep and tricep
  • radial pulse (adult) = on the underside of the wrist at the base of the thumb, below the wrist creases
  • carotid pulse (adult) = on the neck next to the windpipe
  • arrhythmia
  • tachycardia
  • bradycardia
  • atrial fibrillation (A-fib)
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17
Q

Arrhythmia

A
  • a problem with the rate or rhythm of the heartbeat
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18
Q

Tachycardia

A
  • when the heart beats faster than 100 BPM in adults
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19
Q

Bradycardia

A
  • heart rate below 60 BPM
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20
Q

Atrial fibrillation (A-fib)

A
  • common
  • an irregular heartbeat that causes the heart to beat in an unorganized rhythm
  • caused by a problem in the heart’s electrical system, which stimulates the heart to squeeze and relax
  • when the blood flow slows, it has the potential to pool which can increase the risk for blood clots, stroke, or other heart-related complications, including heart failure
  • cause the risk of ischemia
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21
Q

Respiratory rate

A
  • the number of breaths an individual takes per minute
  • measured by counting the number of times the chest rises upon inhalation
  • most typically observed with the individual at rest
  • an average adult takes 12-20 breaths per minute
  • a respiration rate under 12 = abnormal
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22
Q

Blood pressure

A
  • how much the pressure or the force the heart sends the blood through the arteries
  • as the heart beats, it creates pressure that forces blood through the arteries, veins, and capillaries
  • systolic pressure = blood being pushed out of the heart and into the arteries of the circulatory system (top number of blood pressure)
  • diastolic pressure = when the heart rests in between heart beats (bottom number of blood pressure)
  • average blood pressure varies by age
  • normal range for adults = less than or equal to 110/70 mm Hg
  • typically, more attention is paid to the systolic blood pressure measurement as an indicator of cardiovascular disease, particularly for individuals over 50
  • either an elevated systolic or diastolic reading may be used to make a diagnosis of high blood pressure
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23
Q

Oxygen saturation

A
  • the amount of hemoglobin in the blood that is saturated with oxygen
  • hemoglobin = binds with oxygen and is transported around the bloodstream to the organs, tissues, and cells (each red blood cell contains about 270 million molecules of hemoglobin)
  • dependent on oxygen availability (what an individual breathes in), the concentration of hemoglobin in the blood, and gas exchange in the lungs
  • normal oxygen saturation ranges between 96-100%
  • measured non-invasively using pulse oximetry (Spo2) = clips to a finger and measures the wavelengths of light reflect from the blood (on the finger, ear, or toes for little kids)
  • values under 90% = low (will need supplemental oxygen on)
  • hypoxemia (low blood oxygen) = lower than normal level of oxygen in the blood (can cause problems with gas exchange, such as in cases of asthma, COPD, or lower concentration of hemoglobin like iron deficiency anemia)
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24
Q

Hypertension (HTN)

A
  • higher than 140/90 mm Hg
    Primary factors:
  • no specific identifiable etiology
  • may be age, family history or obesity or lifestyle choices (definitely a genetic factor)
  • smoking, ETOH abuse, limited exercise, and high sodium intake
    Secondary factors:
  • triggered by an underlying condition = kidney, adrenal or thyroid gland, congenital blood vessel defects, and obstructive sleep apnea
  • medication, birth control pills, dietary pills, stimulants, and antidepressants

*sedentary lifestyle leads a to weaker body systems
- known as the “silent killer”
- 108 million adults in U.S. diagnosed each year
- primary cause or contributing factor to almost 1,000 deaths each day (not because of hypertension, but of what it causes like strokes or heart attacks

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25
Q

Signs and symptoms of hypertension

A
  • often asymptomatic until it becomes a problem
  • persistent high blood pressure damages heart, kidneys, and other body structures
  • sometimes can cause nose bleed, headaches, or shortness of breath (SOB) = when it is severe or life threatening
  • can damage other body structures because they have blood vessels
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26
Q

Medical management for hypertension

A

Lifestyle changes:
- dietary changes = decreasing sodium intake
- increasing physical activity
- smoking cessation
- limiting alcohol (ETOH) consumption
Medications
- diuretics = (makes you pee which helps get rid of the fluid in your body) support kidney function and reduce blood volume (your heart pumps through your body, goes to your kidneys to produce urine)
- angiotensin-converting enzyme (ACE) inhibitors = prevent blood vessels from narrowing, stays open
- calcium channel blocker = prevents calcium from entering cardiac muscle calls (calcium helps muscle to contract) and allows blood vessels to relax
- slow heart rate and inhibit force to decrease cardiac workload, lowers blood pressure

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27
Q

Coronary artery disease (CAD)

A
  • one of the most common cardiac-related disorders affecting millions of individuals worldwide
  • coronary arteries = the blood vessels that supply the heart with blood, oxygen, and nutrients
  • coronary arteries become damaged over time
  • atherosclerosis = a buildup of fatty, fibrous plaque that can progressively narrow the vessels, can occlude blood supply to the heart muscle
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28
Q

Etiology of CAD

A
  • decreased blood flow over time
  • weakness the heart muscle
  • increase risk for angina, myocardial infarction (MI), heart failure, and arrhythmias
  • genetics
  • high cholesterol levels
  • diabetes
    Lifestyle factors:
  • limited physical activities
  • smoking
  • excessive alcohol (ETOH) consumption
  • stress
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29
Q

Signs and symptoms of CAD

A
  • angina (heart/chest pain) = pressure, aching, or squeezing in the left shoulder, arm, neck, back, or jaw (the blood is not getting to the heart for it to work, so the heart is having to work harder
  • shortness of breath (SOB)
  • nausea
  • rapid or irregular heartbeat
  • diaphoresis = excessive sweating (because you’re working hard)
  • feeling similar to indigestion
  • complete blockage of artery = heart attack
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30
Q

Diagnosis of CAD

A
  • medical history
  • medical exam
  • echocardiogram (can see oxygenated and deoxygenated blood going through the heart and the chambers of the heart) = assess heart’s effectiveness
  • stress test (makes you walk on treadmill for 3 minutes and monitor your vitals and heartbeat) = determine the heart’s response to increased activity demand
  • cardiac catheterization or angiogram = camera on a tube, inserted through femoral artery into the heart to what’s happening
31
Q

Course and prognosis of CAD

A
  • progresses slowly
  • goes unnoticed until the narrowing is bad
  • medication and health lifestyle choices leads to a good prognosis
32
Q

Medical/surgical management of CAD

A

Management:
- controls high blood pressure, cholesterol levels, and diabetes
- address diet and weight and activity levels
- decrease stress
- stop smoking
Medication:
- Aspirin or blood thinner = anti-coagulants, help prevent the formation of dangerous blood clots in your arteries, which could block the blood flow (if your blood is too thin, it can lead to excessive bleeding and bruising)
- beta-blockers = slow heart rate and decrease blood pressure
- nitroglycerin tablets = opens coronary arteries

33
Q

Angioplasty

A
  • with or without stent placement
  • deflated balloon is passed through a catheter that is in the blocked artery
  • the balloon is inflated to open the artery and improve blood flow to the heart
  • if needed a stent, a small mesh tube is inserted to maintain the changes
34
Q

Coronary artery bypass graft (CABG)

A
  • procedure for severe cases of CAD (if angioplasty didn’t work out, then you do this procedure)
  • circumventing the blocked artery
  • takes a harvest vessel (usually saphenous vein in the leg) and reroutes the blood around the CAD artery
  • single, double, triple, or quadruple CABG
  • sternal precautions = no pushing, pulling, or lifting anything more than 5-10 pounds
  • cardiac rehabilitation = medically supervised program for exercise (EKG) and education
35
Q

Congestive heart failure (CHF)

A
  • decrease in cardiac efficiency and output affecting the body’s ability to circulate blood (cannot continue to beat really well)
  • chronic, progressive condition
  • impacts the heart’s ability to pump blood to the body and provide organs and tissues with oxygen and vital nutrients
  • impaired ventricle structure/function = often effects both sides, but could just be one
    Left sided dysfunction:
  • more so the heart and lungs
  • increased fatigue
  • shortness of breath
  • pulmonary edema (too much fluid in the lungs)
    Right sided dysfunction:
  • can’t clear the fluid in the body, not just the lungs
  • peripheral edema (fluid in the legs)
36
Q

Etiology of CHF

A

Systolic CHF (systolic heart failure)
- CAD
- narrow vessels limit the flow of oxygenated blood to the heart
- causes the cardiac muscle to weaken and have trouble contracting
- heart can’t pump and its walls are stretched and thin
Diastolic CHF (diastolic heart failure)
- hypertension
- heart pumps with increasing force over time
- walls of the heart thicken and ventricles atrophy
- oxygenating the heart becomes increasingly difficult and causes ischemic damage
- heart can’t fill and its walls are stiff and thick

  • arrhythmias, infection of the heart, diabetes, thyroid problems, obesity, and unhealthy lifestyle choices
37
Q

Signs and symptoms of CHF

A
  • dyspnea = shortness of breath (SOB in right and left sided CHF)
    • ex: taking a shower can be exhausting
  • depression
  • decreased alertness
  • cognitive decline = impaired memory, recall, decreased attention, and decreased ability for new learning
38
Q

Signs and symptoms of left sided CHF

A
  • dyspnea initially only noted with increased exertion
  • as disease progresses, fluid accumulates in the lungs (can happen while at rest or lying down = orthopnea)
  • wheezing and bronchospasm = causing chest tightness and coughing
  • increased fatigue
  • weakness
39
Q

Signs and symptoms of right sided CHF

A
  • peripheral edema
  • legs, liver, and abdomen
  • decline in appetite and nausea
  • cardiac cachexia = a condition that causes severe, unintentional weight loss and muscle loss in people with heart failure, typically in the end stages
  • weight loss associated with CHF
40
Q

Diagnosis of CHF

A
  • medical history
  • physical exam
  • ECG
41
Q

ECG

A
  • determines heart irregularities
  • determines presence of structural changes in the ventricles
  • determines valve function issues
  • assess the thickness and stiffness of the heart
  • determines ejection fraction (EF) = the amount of blood the ventricles pump out with each contraction
  • normal EF = 60% of the blood goes out of the heart into the body
  • in CHF where the heart doesn’t work effectively, the heart isn’t doing its job so there is more left over (residual) in the heart than usual
42
Q

Ejection fraction

A
  • high function = more than 70%
  • normal function = 55-70%
  • low function - 40-55%
  • possible heart failure = less than 40%
  • hospice = 15%
43
Q

Prognosis of CHF

A
  • progressive
  • over time, it slowly deprives the body of oxygen
  • may eventually lead to death
  • CDC says 1/2 of the individuals who develop CHF die within 5 years
  • early detection and treatment can prolong life
44
Q

4 stages of congestive heart failure

A
  • stage 1 = breathless or tiredness (with brisk walk, a jog or taking flights of stairs)
  • stage 2 = comfortable when resting (heart races or breathless when walking a block or taking the stairs)
  • stage 3 = palpitation or tiredness with simple tasks like getting up from the sofa and walking over to the kitchen
  • stage 4 = heart and breath go faster even at rest; tiredness even while sitting; anxiety and palpitations almost all the time
45
Q

Medical/surgical management of CHF

A

Management long term:
- medical care = may need oxygen
Medication:
- ACE inhibitors
- beta-blockers
- diuretics = manage fluid retention
Lifestyle changes:
- diet modification
- regular physical activity
- smoking cessation
- reducing alcohol (ETOH) intake
- minimizing stress
- daily weight monitoring for edema (to see if the fluid is building up or not)

46
Q

Chronic obstructive pulmonary disease (COPD)

A
  • impairment with oxygenated and deoxygenated blood being exchanged
  • an umbrella term for emphysema and chronic bronchitis
  • describes progressive lung disorders
  • irreversible flow obstruction causing dyspnea and limited reserve lung capacity
  • risk for hypoxemia = not enough oxygen
  • progressive and life threatening
  • a chronic inflammatory lung diseases that causes obstructed airflow from the lungs, obstructed by either mucus (chronic bronchitis) or breakdown of alveolar walls (emphysema)
47
Q

Emphysema

A
  • little bit more prevalent then chronic bronchitis
  • walls between alveoli are damaged (can’t exchange oxygen and carbon dioxide)
  • you’re not getting the oxygen you need and you’re never getting rid of carbon dioxide
  • due to smoking
  • limiting gas exchanges
48
Q

Chronic bronchitis

A
  • inflammation in the bronchioles
  • increasing mucus production and thickening and narrowing of the airways
  • if the bronchioles are clogged, the air can’t get through
  • tips of fingers and lip turn blue because not getting enough oxygen in the system
49
Q

Causes of COPD

A
  • long-term exposure to irritants such as cigarette smoke, air pollution, chemical fumes, and dust
50
Q

Symptoms of COPD

A
  • shortness of breath
  • wheezing
  • chest tightness
  • chronic cough with or without mucus production
  • frequent respiratory infections
51
Q

Diagnosis of COPD

A
  • pulmonary function tests (PFT)
  • chest X-rays
  • CT scans
52
Q

Treatments for COPD

A
  • smoking cessation
  • bronchodilators
  • steroids
  • oxygen therapy
  • pulmonary rehabilitation
  • in severe cases, surgery
53
Q

Key differences between COPD, emphysema, and chronic bronchitis

A

Underlying structure affected:
- COPD = involves both small airway disease and parenchymal destruction
- emphysema = specifically affects the alveoli
- chronic bronchitis = affects the bronchial tubes
Primary symptom:
- COPD = combination of emphysema and bronchitis symptoms
- emphysema = shortness of breath due to damaged alveoli
- chronic bronchitis = persistent mucus-producing cough

54
Q

Signs and symptoms of COPD

A
  • dyspnea with minimal exertion
  • persistent cough = increased mucus production, wheezing, and fatigue
  • most symptoms don’t occur until the lung is already damaged (doesn’t notice the severity of the symptoms until it is too late)
    Severe cases:
  • lower extremity edema
  • weight loss
  • cyanosis = bluish color to lips and fingernails and decreased oxygen levels
55
Q

Course and prognosis of COPD

A
  • as mucus production increases, it further narrows the airways that causes accumulation of carbon dioxide (at risk for hypo ventilation)
    Can have exacerbations (the worsening of a disease or an increase in its symptoms) of symptoms:
  • exposure to irritants
  • respiratory infection
  • need immediate medical intervention = lung failure
56
Q

Medical/surgical management of COPD

A
  • bronchodilators = relax the muscles around the airways to relieve coughing and dyspnea
  • lifestyle changes = smoking cessation
  • steroids at times
  • pulmonary rehab program
57
Q

Myocardial infarction (MI)

A
  • heart attack
  • blood flow that brings oxygen to the heart muscle is severely reduced or completely occulted (a stroke in the heart)
  • prolonged lack of oxygen to cardiac tissue
  • irreversible damage
  • if blood flow is not restored (ischemic) = cardiac muscle dies and can be fatal
  • atherosclerosis = plaque within the arterial walls, leading cause of cardiac blockage, and can rupture and lead to thrombus formation that obstructs the blood flow (hemorrhage)
  • artery dissection = a tear in the arterial wall
  • different than the cardiac arrest
  • cardiac arrest is the electrical system to the heart, SA, and VA nodes, the electrical input that comes to your heart doesn’t work
  • abrupt loss of heart function
  • typically triggered by a malfunction in the electrical system
  • due to irregular heart rhythms
  • heart attack is the issue with the arteries of not getting enough oxygen or a tear in the arteries
58
Q

Etiology of MI

A
  • hypertension
  • smoking
  • high cholesterol levels
  • diabetes
  • obesity
  • poor diet
  • sedentary lifestyle
  • excessive alcohol consumption

*the leading cause of death in both men and women in the U.S.

59
Q

Prevention of MI

A
  • stop smoking
  • choosing good nutrition
  • being physically active every day
  • aiming for a healthy weight
  • managing diabetes
  • reducing stress
  • limiting alcohol consumption
  • making sure you get enough sleep
  • lowering high blood pressure
60
Q

Signs and symptoms of MI

A
  • chest pain = pressure, aching, fullness, squeezing sensation that may radiate to the jaw, shoulders, left arm and/or upper back
  • dyspnea
  • diaphoresis
  • gastric discomfort = nausea or vomiting
  • syncope = fainting or passing out
  • impaired cognition
    Women:
  • extreme weakness
  • fatigue
  • restlessness
61
Q

Diagnosis of MI

A

ECG (can be used interchangeably with EKG):
- records heart electrical activity and the location of any damaged cardiac muscle
- unstable angina
- non-ST-Segment Elevation MI (NSTEMI)
- ST-Segment Elevation MI (STEMI) = a sense of presence of ST Segment Elevation or Q waves
Blood test:
- presence of cardiac markers (substances in the blood that indicate heart damage or stress) in the blood
- indicated myocardial cell injury
- lack of cardiac muscle proteins
- remain high within 6 hours to a few days afterwards

  • angioplasty = sending up the camera through the left
  • MI severity depends on the extent and duration of the arterial obstruction
62
Q

Course and prognosis of MI

A

*thinning the blood helps give extra oxygen by forcing oxygen in the system to bypass until they can get the heart to deliver oxygen
Delay in medical care:
- decrease the risk of survival
- limit options for treatment
Early intervention:
- use of antiplatelet medication (like aspirin)
- supplemental oxygen if its oxygen saturation is less than 90%
- nitroglycerin for active chest pain
When blood flow is restored:
- hospitalized until medically stable and have treatment of lifestyle modification
- at risk for developing another MI, heart failure, angina, or a stroke

63
Q

Medical management of MI

A
  • restoration of coronary blood flow and recovery of myocardium
    Medication:
  • blood thinners
  • beta blockers to relax cardiac muscles
  • ACE inhibitors to lower blood pressure
    Lifestyle management:
  • diet modification
  • smoking cessation
  • increasing physical activity
64
Q

Surgical management of MI

A
  • PCI (percutaneous coronary intervention) or angioplasty
  • stunting
  • CABG
65
Q

Impact on ADLs with occupational performance

A
  • ADLS are compromised by shortness of breath and decreased endurance
  • anxiety for fear of falling
  • decreased strength and endurance
  • CABG with sternal precautions
  • bending causes increase in shortness of breath
  • may need oxygen (always attached to you)
66
Q

Impact on IADLs with occupational performance

A
  • IADLs compromised by limited strength, decreased functional activity tolerance, and shortness of breath, especially with this increased activity demand
  • loss of independence
  • dust ration
  • anxiety
  • depression
  • supplemental oxygen = difficulty with community mobility and travel
67
Q

Supplemental oxygen

A
  • oxygen tank and oxygen concentrator
68
Q

Pros and cons of oxygen tank

A

Pros:
- available in different sizes to make transporting them easier
- does not require power
- purchasing them is less expensive up front
- a conserver device, may be used to deliver oxygen in “pulses” (bursts) when you inhale
Cons:
- holds a finite amount of oxygen, meaning you run the risk of running out of oxygen altogether
- must be refilled or replaced when the oxygen inside is used up
- they are awkward, heavy, and difficult to carry and travel with
- may be more expensive over time with ongoing costs of tank refills and replacements
- prohibited aboard for use on airplanes
- liquid oxygen requires special handling to prevent frostbite or burns
- delivers continuous flow oxygen, so oxygen is dispensed when when you exhale

69
Q

Pros and cons of oxygen concentrator

A

Pros:
- can provide unlimited supply of oxygen as long as it has power
- uses the surrounding air to produce supplemental oxygen, so it never needs to be refilled
- they are compact and portable, especially when using a portable oxygen concentrator model, which often weigh less than 5 pounds
- once purchased, the only additional costs are the ocasional replacement part for your oxygen concentrator
- FAA-approved and can be used on board airplanes
- available for both continuous and pulse-dosing, allowing you to get the oxygen dosing you need
Cons:
- requires power via a charged battery or access to a power outlet to function
- does make a quiet sound when in use
- cost more upfront

70
Q

Impact on health management with occupational performance

A
  • usually a delay in recognizing symptoms
  • stress of anxiety
  • struggle to successfully implement lifestyle changes
71
Q

Impact on rest and sleep with occupational performance

A
  • breathing difficulties while laying down
  • stress
  • increased fatigue
72
Q

Impact on education with occupational performance

A
  • can have cognitive changes due to lack of oxygen
73
Q

Impact on work with occupational performance

A
  • struggles to continue working
74
Q

Impact on social participation with occupational performance

A
  • may be difficult to maintain it