Cardiology Flashcards

1
Q

Murmurs and grades

A

MRASS
MSARD
Grade 1-6: 4 includes a thrill

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2
Q

S3 and S4 sounds along with causes

A

S3: Kentucky and d/t increased fluid volume
S4: Tennessee: Stiff ventricular wall

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3
Q

Systolic vs Diastolic HF

A

Systolic: HFrEF
Diastolic HFpEF

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4
Q

Acute vs chronic HF include s/s

A

Acute: abrupt onset following MI, LVH, Valvular rupture . S/S: S3, mitral regurgitation, pulmonary symptoms
Chronic: Inadequate compensatory mechanisms over time, right sided heart failure. S/S: JVD, Hepatomegaly, dependent edema resulting from increased capillary hydrostatic pressure

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5
Q

Four classifications of HF

A

i. 1: no limitations, normal activity
ii. 2: slight limitations, comfortable at rest
iii. 3: marked limitations, comfortable at rest, 3 pillows to sleep
iv. 4: severe, symptoms at rest

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6
Q

What is the first line pharmacologic therapy for all HF

A

Diuretics

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7
Q

What are the 4 cardiomypoathies, ECHO results, and treatment

A

a. Dilated : Most common, LV dilation, no squeeze, LV thinning, and global dysfunction
ii. Idiopathic, presents like heart failure
b. Hypertrophy: No fill LV thickening >1.5cm
c. Restricted: No fill, LVH, low voltage, speckled myocardium
i. Stress: Takotsuo: ACS with clean arteries
Aldactone for all, avoid CCB with dilated. ACE/ARBs and BB.

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8
Q

Primary vs Secondary HTN

A

a. Classification
i. Primary
1. Most cases
2. Onset >55yrs of age
ii. Secondary
1. Most common is renal artery stenosis

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9
Q

HTN urgency vs emergency and treatment

A

Urgency = don’t overtreat
-BP>180/110, may or may not have headache, anxiety, SOB
-Oral therapy: catapress/clonidine (Alpha agonist)
Emergency
-BP > 180/120, requires immediate BP reduction within 1 hour
-OR if you have end organ damage, encephalopathy, ICH, angina, MI, HF, dissection, eclampsia
-Management: ICU admission, Parenteral administration (nicardipine drip or nipride drip)
Reduce to <140 in 1 hour in high risk patient or less than <120 for dissection
-Reduce no more than 25% or SBP 160 in first hour if unstable

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10
Q

Unstable angina Treatment

A

TX: Elevated give Statin
1. High statin are Atorvastatin and rosuvastatin
2. Can’t handle a statin, you can give questran, fenofibrate, tricor, ezetimide, niacin
h. Aspirin, CCB, Nitrates, BB

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11
Q

EKG interpretation and area of heart that has an MI

A

e. EKG: ST elevation
i. ST elevation in 1 and AVL = Lateral MI
ii. 2,3,AVF = Inferior
iii. V3, V4 = Anterior

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12
Q

ACS treatment

A

k. Treatment: Aspirin, nitro, lasix, morphine, possible BB IV if not contraindicated, possible ACE inhibitor (prevented ventricular remodeling), ?anticoagulation (Lovenox 1mg/kg q12)

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13
Q

TPA contraindications

A

ii. TPA contraindication: Current or prior bleed, neoplasms, ischemic stroke in past 3 months, head or fascial surgery in the past 3 months, severe uncontrolled HTN***

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14
Q

PVD s/s, workup, and treatment

A

i. arterial sclerosis over time
ii. HX smoking, DM, HLD
iii. S/S: Claudication calf pain, shiny hairless skin, dependent rubor, elevation pallor
iv. Ankle Brachial Index***
v. Exercise, stop smoking
vi. Fletal medication
vii. Angioplasty, bypass, amputation

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15
Q

CVI s/s, workup, and treatment

A

i. Destruction of valves/thrombophlebitis, venous hypertension, causing venous stasis
ii. S/S: Aching of lower extremities received by elevation, night pain, edema, dermatitis, periphery is cool to touch
iii. R/O edema d/t HF
iv. R/O Ray turner’s syndrome***
1. Results in LLE DVT
v. Treatment: elevate legs, elastic support stockings, weight reduction, dermatitis (Tap water compress, hydrocolloid dressings)

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16
Q

Pericarditis s/s, workup, and treatment

A

i. Inflammation of outside of heart
ii. Viruses are most common cause*
iii. Classic Presentation: Precordial chest pain worse with coughing/swallowing, SOB, pericardial friction rub, pleural friction rub, fever
iv. Labs: ST elevation is all leads and PR depression
*
v. Needs ECHO
vi. Tx: NSAIDs, Ibuprofen, endomethacin, steroids only if there is total failure of high dose NSAIDS (Can increase viral replication)
1. ABX: only if bacterial
2. Codiene and monitor for tamponade

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17
Q

Endocarditis s/s, workup, and treatment

A

i. Must be excluded in all patients with heart murmur and fever of unknown origin
ii. Typically, bacterial causes
iii. Risk: Rheumatic heart disease, valve prolapse, dental surgery, IVDA, congenital heart disease
iv. S/S: Fever, malaise, night sweats, and weight loss, murmur (not always), Oslars nodes (distal phalanges), splinter hemorrhages, splenomegaly, Janaway lesions (small non painful macula on palms and soles)
v. Night Sweat causes
1. TB, Endocarditis, menopause, HIV/AIDS, heme/onc leukemia
vi. Labs: ALWAYS left shift with band formation, positive BCx, elevated ESR
vii. Acute endocarditis typically due to staph, MRSA, and MSSA
viii. Empiric vanco until results are back

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18
Q

Currigens pulse and quinkes pulse and what do they represent

A

Aortic Dissection
C: Forecful carotid pulse
Q: Finger nails change from pink to white with heartbeat

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19
Q

A fib managment

A
  1. Rate control
    a. Typically a BB
  2. Anticoagulation
    a. First time, young, no risk factors: Aspirin
    b. Elderly, comorbidities, paroxysmal: Coumadin
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20
Q

What valves are open and closed during S1 vs S2

A

S1: Aortic and pulmonic open
S2: Mitral and tricuspid open

21
Q

EF of HFrEF vs HFpEF contractility vs fill

A

HFrEF: < 40%, contractility
HFpEF: >50, fill

22
Q

HFrEF Treatment

A
  1. Diuretics
  2. ACE, ARB, or ARNI
  3. SGLT2
  4. BB (Metoprolol, carvedilol)
  5. MRAs (spironolactone, Aldactone)
23
Q

HFpEF treatment

A
  1. Diuretics
  2. SGLT2
  3. ARB. ARNI, MRA
    DO NOT USE ACE
24
Q

How to manage inpatient acute pulmonary edema

A

O2 1-2 l
Semi Fowler s
Morphine and lasix
Low CI add Dobutamine or dopamine

25
Q

Normal BP, elevated BP, HTN stg 1, HTN stg 2

A

Normal: <120/80
Elevated: 120-129/<80
HTN 1: 130-139/80-89 OR
HTN 2: >140/ >90 OR

26
Q

HTN treatment for non-African, African, DM, and Adults >18 with CKD

A

Non African: Thiazide, ACEi, ARB, CCB
African: Thiazide, possible CCB
DM: ACEi or ARB
CKD: ACEi

27
Q

EKG results that show angina

A

ST depression

28
Q

What is Vasospastic/variant/ Prinzmetal’s angina.

A

Typically occurs at rest; may be present at night or earl in the morning, severe pain due to sudden influx of Ca
-Tx CCB

29
Q

What is Levite’s sign

A

Clenched fist to chest, sign of angina

30
Q

Desired total cholesterol, VLDLs, LDLs, HDLs

A

Total cholesterol: <200
VLDLs: <150
LDLs: <100
HDLs: >60

31
Q

LDL, HDL, and Triglyercide goals for diabetics

A

LDL: <70
HDL: >40
TG: <150

32
Q

Normal INR and therapeutic post MI values

A

Normal: 0.8-1.2
Post MI: 2.5-3.5 x normal

33
Q

Warfarin MOA and reversal

A

Depletes vitamin K stores to impair clotting
Tx: Vitamin K, FFP, Kcentra

34
Q

Lovenox and heparin MOA and reversal

A

MOA: inhibits factor Xa and IIa
Protamine

35
Q

Eliquis MOA and reversal

A

Inhibits factor Xa
Andexxa
Kcentra

36
Q

CHA2DS2-VASc scoring

A

CHF, HTN, age >75, diabetes, stroke, vascular disease, age 65-74, sex (female)
Determines need for Anticoagulation

37
Q

Treatment for A fib

A

Anticoagulation and cardiovert x2 then ablate

38
Q

Indications for pharmacological revascularization

A

Unrelieved chest pain WITH ST elevation

39
Q

Door to needle time in ACS

A

30 mins

40
Q

Which heart valve abnormality causes a S 3 heart sound and beings at S1 causing a blowing/musical high pitched sound at the apex

A

Mitral regurgitation

41
Q

Which valvular abnormality causes an S1 low crescendo rumble

A

Mitral stenosis

42
Q

What does Niacin do?

A

Increase HDL and lower LDL

43
Q

What valve malformation causes S3 systolic murmur

A

Mitral regurgitation

44
Q

What does ACE not do

A

Lower HR

45
Q

What are signs of a successful TPA administration in an MI

A

ST return to normal
V fib/V tach
Decrease in chest pain

46
Q

How is CO affected by aging

A

It isn’t

47
Q

When is troponin peaked or most likely to show positive

A

4-8 hours after symptoms

48
Q

Pericardial effusion treatment

A

Initially NSAIDs and Ibuprofen