Cardio10 Flashcards

ECG tracings AV block ANP

1
Q

ECG tracing characteristics of Atrial fibrillation.

A
  • Chaotic, erratic baseling (irregularly irregular)

- No discrete P waves in between irregularly spaced QRS complexes

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2
Q

What can A Fib result in?

A

Atrial stasis –> Stroke

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3
Q

What are the treatments for A fib?

A

Rate control
Anticoagulation
Cardioversion (possibly)

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4
Q

ECG tracing characteristics of Atrial flutter.

A
  • Rapid succession of identical, back-to-back atrial depolarization waves
  • “Sawtooth” appearance
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5
Q

Pharmacologic conversion to:

  • Sinus rhythm
  • Rate control
A
Sinus rhythm:
 - class IA, IC, or III antiarryhythmics

Rate control:

  • beta-blocker
  • Ca++ channel blocker
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6
Q

ECG tracing characteristics of Ventricular fibrillation.

A

Completely erratic rhythm with no identifiable waves

*Fatal w/o immediate CPR & defibrillation

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7
Q

1st degree AV block.

A

PR interval is prolonged (>200 msec)

*Asymptomatic

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8
Q

2nd degree AV block: Mobitz type I (Wenckebach)

A

Progressive lengthening of the PR interval untial a beat is “dropped”
-P wave no follwed by a QRS complex

*Usually asymptomatic

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9
Q

2nd degree AV block: Mobitz type II

A
  • Dropped beats that are NOT preceded by a change in the length of the PR interval (as in type I)
  • Abrupt, nonconducted P waves

*Pathologic condition

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10
Q

3rd degree AV block (complete).

A
  • Atria & Ventricles beat independently of eachother
  • Both P waves and QRS complexes are present
  • P waves bear no relation to the QRS complexes
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11
Q

T/F - The atrial rate is faster than the ventricular rate.

A

True

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12
Q

What is the treatment for 3rd degree AV block?

A

Pacemaker

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13
Q

What condition can result in 3rd degree AV block?

A

Lyme disease

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14
Q

What triggers the release of Atrail Natriuretic Peptide (ANP) from atrial myocytes?

A

INcreased blood volume

INcreased atrial pressures

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15
Q

Function of ANP.

A
  • Generalized vascular relaxation

- DEcreased Na+ reabsorption at the medullary collecting tubules

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16
Q

MOA of ANP.

A
  • Constricts Efferent renal arterioles
  • Dilates Afferent arterioles (cGMP mediated)
  • Promotes diuresis
  • Contributes to the “escape of aldosterone” mechanism