brain lab briefing Flashcards

1
Q

describe the layers of the scalp

A

Skin: where hair grows, nutrients for hair follicles are found here

Connective tissue: dense subcutaneous layer of fat and fibrous tissue. contains nerves and vessels of scalp.

epicranial Aponeurosis: tough layer of dense connective tissue which runs from frontalis muscle anteriorly to occipitalis posteriorly

Loose connective tissue: areolar connective tissue, made up of random collagen bundles, separates the upper 3 layers of scalp of pericranium, and allows them to move in realign to pericranium.

Periosteum: provides nutrition to the cranium capacity to repair

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2
Q

if there is an infected scalp wound, how is there potential for it to spread intracranially?

A

via emissary veins into cranial cavity

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3
Q

why is the convex shape of the skull important?

A

it dissipates injury by distributing and minimising the effects fo a blow to the head so the head is able to take more force without fracturing

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4
Q

what important structures are found the base of the skull?

A

cranial nerves and blood supply

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5
Q

what bones make up the base of the skull?

A

orbital plate of frontal bone, sphenoid bone, squamous and petrous temporal bones, occipital bone

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6
Q

describe a depressed skull fracture

A

bone fragments depress inwards with damage/compression of brain

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7
Q

describe a linear skull fracture

A

a break in cranial bone resembling a thin line. it occurs at site of impact but the fracture lines radiate away

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8
Q

what is battle’s sign?

A

bruising over the mastoid process (purplish bruising)

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9
Q

what is periorbital ecchymosis also known as?

A

raccoon or panda eyes

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10
Q

what causes panda eyes?

A

basal skull fracture that ruptures meninges and causes venous sinuses to bleed into arachnoid vill and cranial sinuses.

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11
Q

what clinical features are seen in panda eyes beside bruising around the eyes?

A

blood or csf coming out of the ear or nose which indicates breach of the meninges

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12
Q

what diagnostic test should be carried out if panda eyes are present?

A

CT scan

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13
Q

what is the pterion?

A

region of skull behind the temple, where the frontal, parietal, temporal, and sphenoid bones join together.

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14
Q

why is there a high chance of fracture of the pterion and what consequences would a fracture have?

A

It is translucent and very thin

There are branches of meningeal arteries underneath so fracture could lead to intracranial haematoma.

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15
Q

what are the 2 layers of the dura mater and where are they attached and separated?

A

endosteal and meningeal layers

they are closely attached and only separate to form venous sinuses and at reflections

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16
Q

what are the 2 main dural reflections and what do reflections do?

A

falx cerebri and tentroium cerebellum

They divide the cranial cavity into compartments by forming partitions

17
Q

list 3 types of intracranial haematomas

A

epidural/extradural
subdural
intracerebral

18
Q

describe an epidural haematoma including causes and shape

A

When blood collects between periosteal layer of dura and skull, the blood strips the dura away from the periosteum.

Related to a skull fracture, caused by arterial bleeding usually of middle meningeal artery. It forms a lens shaped swelling because it cannot swell along suture lines

19
Q

describe a subdural haematoma

A

when blood collects between dura and arachnoid

Blood is of venous origin = slower bleeding

20
Q

what features can be seen in an acute extradural/epidural haematoma CT scan?

A

lens shaped haematoma
“white” area = fresh blood in context of head injury
ventricular shift - swelling of brain closes the ventricles and shifts the ventricle away from midline

21
Q

what features can be seen in an acute subdural haematoma CT scan?

A

crescent shaped haematoma as there are no suture limitations so blood can spread all the way around the brain
middling shift of ventricles

22
Q

what re the 3 components of intracranial volume?

A

blood, csf, brain

23
Q

what happens in response to raised cranial pressure due to presence of a mass? what happens I this mass keeps increasing?

A

compensatory mechanisms eg removing venous blood and csf

after a critical point, there is decompensation which causes ICP to rise rapidly

24
Q

what is the equation for cerebral perfusion pressure?

A

CPP = MAP - ICP

25
Q

how does a normal brain auto regulate its blood flow to maintain perfusion pressure?

A

by altering the resistance of cerebral blood vessels

26
Q

if homeostatic mechanisms fail after a head injury, what effect does a rising ICP have?

A

can lead to reduced CPP and ischaemic damage

27
Q

what are signs of increasing ICP?

A

decreasing Glasgow coma scale
diminished pupil response to light
lateralising signs eg weakness on one side

28
Q

if there is uncontrolled rise in ICP, what are the types of herniation that occur? describe them

A

1st: cingulate herniation occurs
cingulate gyrus slips under the flax cerebri and this causes a midline shift which is seen on CT scan

2nd: uncal herniation occurs
uncal of temporal lobe slips under tentorium cerebellum and this ipsilaterally compresses oculomotor nerve (CN3) which affect pupils reflexes = fixed and dilated pupil

if it progresses further: brainstem could be compressed