Book 6, Case 3-Pedi strabismus Flashcards
Succinylcholine in pedi pt if you couldn’t get LMA (pt still stable) This is a strabismus case
I would prefer not to use succinylcholine for this case because it may interfere
with the forced duction test often used during strabismus surgery (it may interfere with the
test for up to 20 minutes), and due to the risk of undiagnosed muscular dystrophy in this
patient < 8 years of age
You give sux and then stiff jaws-what’s your DDx?
- masseter muscle rigidity
- undiagnosed muscular dystrophy
- MH
Why is MMR concerning? and does it typically inhibit ventilation?
b/c it can suggest a susceptibility to MH. It doesn’t usually inhibit ventilation. Due to this risk of developing MH or rhabdo. Rhabdo can sometimes happen independent of MH in MMR
What should yuou do with MMR (assuming the pt is okay and it resolves)
Elective case cancelled and pt admitted to hopsital for 12-24 hours for monitoring of possible moglobinuria, myalgias, or rigidity. Thye should also have a neurology evaluation fro presence of occult myopathy and close monitoring of electrolytes.
You gonna suggest that pt with MMR get Caffeine Halothane Contacture test?
I would recommend it to evaluate susceptibility to MH, but I would relay that I understand that the test would require expenses and that they could also always alert the anesthesiologist
You administering prophylactic dantrolene? Why or why not?
Nah. Dantrolene has side effects of nausea and vomiting-but threshold for tx is low in pts
How do you prepare for MH cases?
In addition to all the vaporizer stuff you know, also ensure that MH cart nearby, Ice nearby
HR falls to 62 during strabismus surgery-what are you thinking? Wyd?
Thining: oculocardiac reflex.
Ask surgeon to stop
Thenensure oxygenation and ventilation, evaluate rhythm and bp, assess volume and anesthetic depth-insufficient depth could lead to cardiac depression
Consider administration of IV atropine if that doesn’t work. Dose: 0.02 mg/kg
What is the oculocardiac reflex?
The afferent limb of the reflex
involves the ciliary nerves and ophthalmic division of the trigeminal nerve, while the efferent
limb is the vagal nerve. Stimulation of this reflex, which may occur under local or general
anesthesia, most commonly results in bradycardia, but can lead to a wide variety of cardiac
dysrhythmias.
PACU calls you because your strabismus kid with MMR now has HR of 118-wyd?
First I would evaluate the patient’s volume status and ensure adequate pain
control, oxygenation, and ventilation, and ensure that pressure is okay.
Next, given the fact that there is an increased
incidence of MH in patients who experience an episode of significant MMR, I would
evaluate the patient for other signs of MH such as generalized rigidity, tachypnea, changes in
blood pressure, arrhythmias, increased temperature, peripheral mottling, rhabdomyolysis (tea
colored urine), sweating, and cyanosis.
If optimization of the patient’s ventilation, volume
status, and pain control did not resolve the tachycardia, I would obtain an arterial blood gas
(decreased P02, metabolic and respiratory acidosis) and check for hyperkalemia,
hypercalcemia, myoglobinemia, and elevated serum creatine kinase.
initial stages of MH reaction may show a blood gas with what? What are you going to do if that blood gas is accompanied by an unlabored pt?
during the initial stages of a MH reaction an arterial blood gas
may show a nearly pure respiratory acidosis. So if this happens in pts who seem like theyre brathing okay:if the patient’s ventilation appeared
adequate, I would order a mixed or peripheral venous blood gas and, if the results indicated a
hypermetabolic state (Pv02 < 40 mmHg despite supplemental oxygen due to increased
oxygen consumption), initiate aggressive treatment.
How will you manage a pt with suspected MH who is extubated and in PACU?
I would: (1) call for assistance; (2) hyperventilate the patient with 100%
oxygen by face mask;(3) administer dantrolene (2.5 mg/kg ofDantrolene every 5-10 minutes
as necessary to control symptomatology); ( 4) maintain urine output with intravenous fluids,
mannitol, and lasix; (5) monitor urinary output, potassium, calcium, and arterial blood
gasses; (6) order serum creatine kinase, liver function tests, and a coagulation profile; and (7)
treat hyperkalemia (dextrose and regular insulin), acidosis (bicarbonate-only if you know what!), hyperthermia (cold
intravenous fluids; ice packs over major arteries of the groin and axilla; gastric, bladder,
rectal, and wound lavage; cold peritoneal dialysis; and even cardiopulmonary bypass),
rhabdomyolysis (mannitol), and dysrhythmias as necessary. Finally, after resolution of the
crisis, I would (8) continue intravenous dantrolene, 1 mg/kg every 6 hours, for 24-48 hours to
prevent relapse; and (9) monitor the patient for up to 72 hours in the ICU for signs of
disseminated intravascular coagulation, myoglobinuric renal failure, and recrudescence
(relapse).
When should active cooling be stopped in MH?
Active cooling should be discontinued when the temperature drops
to 38-38.5 °C to prevent hypothermia.
How long would you continue dantrolene after the patient’s ABGs returned to normal?
I would continue dantrolene, 1 mg/kg every 6 hours,
for 24-48 hours, because up to 25% of patients may experience relapse within the first 24
hours.
After MH episode, pt has urine output of 0.5 mL/kg/hr-you concerned?
Although this is normal, it is on the edge of being abnormal. I wouldattempt to improve urine output
by administering intravenous fluids and mannitol or lasix. In addition, if the patient’s urine
was acidic, I would give bicarbonate to help prevent renal tubule injury (helps prevent cast
formation).