Body systems chapter 32 Flashcards
regulator/ conformers for internal environment
regulators- use internal mechanisms to control internal change when there is external fluctuation.
conformers-allows internal body condition to change in accordance with external changes
* animals can be regulators for certain internal conditions while allowing others to conform to the environment
thermoregulation
physiological mechanism that allow an organism to maintain INTERNAL temperature within a normal body range.
-endotherms- generate heat metabolically
-ectotherms- gain heat from environment
types of heat- radiation- how organisms lose most of their heat, radiate and absorb heat from the sun- evaporation- cooling effect when liquid evaporates into a gas the heat is lost. -convection-how organisms lose heat the fastest- molecules in contact with each other transfer heat– you can’t add cold, only remove heat
temperature adaptations
- insulation- reduce heat flow from body to environment- hair-feathers- adipose(blubber) -tissure
- circulatory adaptations- reduction of blood flow near the body surface traps heat within the body core– vasodilation and visoconstriction in blood vessels. plus countercurrent exchange of heat between closely associated blood vessels minimizes heat loss
* * countercurrent heat exchange- warm blood in arteries in close contact with veins carrying cold blood- this facilitates heat transfer from the arteries to the veins - as blood in the veins approaches the body centre it is almost as warm as the body core. - seasonal acclimatization- how organisms change throughout the seasons- shed fur in summer, ectotherms do thermoregulation by using enzymes to change the amount of saturated and unsaturated lipids to keep the membrane fluid– antifreeze and arctic fish
endocrine and nervous control
- control and coordinate an animals response to external stimuli.
endocrine- hormones that travel all thru body to target cells with specific receptors that respond- the effect is slow but long lasting.
nervous- neutrons send impulses - fast but target cells must have specialized junctions that allow transmission of impulses fast but short effect
hormone regulation
hormones released directly into blood stream by endocrine glands (there are no ducts they are secreted) exocrine glands are the ones that dump their secretions into ducts like the salivary glands liver and pancreas.– hormones recognized by specific tissue which is the target and when stimulated will either increase or decrease in activity.
** hormones control-metabolism, homeostasis and growth and reproduction
types of hormones
peptide- hydrophilic activate receptors on the target cell surface (cannot diffuse through due to polarity) – fast because they activate enzymes present already in the target cell ex; insulin glucagon oxytocin acth ( effect not long)
-peptide hormone is the first messenger and it binds to receptor in plasma membrane activating signal induction which activates an enzyme
steroid- hydrophobic and can diffuse through membrane- pass through membrane of target cell **they are made of lipids - precursor is cholesterol cause target cell to go through protein synthesis. this effect is slow but long lasting. ex; cortisol, estrogen, testosterone.
- steroid binds to hormone receptor steroid- receptor complex enter nucleus and bind to DNA regulatory site ( promoter) then a protein is transcribed and translated.– endocrine glands release hormones when needed – THIS IS A NEGATIVE FEEDBACK LOOP- secretions are shut off when no longer needed.
negative feedback loop
internal body conditions change beyond normal parameters for function body initiates response to NEGATE the problem ex; chime leaves pyloric sphincter and enters duodenum the pH of the chime is low and the acidic contents must be neutralized so the pancreas releases bicarbonate
Same hormone different response
hormones with different functions in certain cells
ex-ephinephrin hormone produces different responses in different target cells - target cells with the same receptor but different single transduction pathways or effector proteins have different responses– different receptors also makes different responses ex; prolactin stimulates the production of milk by mammary glands and different roles in different organisms ( regulates fat and reproduction in birds- delays metamorphosis in amphibians and regulates salt/ water balance in fish
temperature
body temp too warm– hypothalamus detects and sends impulses —-vasodilation, flat hair, sweat.
body temp too cold- hypothalamus detects–vasoconstriction, hair on end, shivering
glucose
high blood glucose levels– pancreas detects– insulin released from Beta cells of the islets of langerhans– which convert glucose into glycogen and cells increase glucose uptake
low blood glucose levels–alpha cells in pancreas detect and secrete glucagon —- cells decrease glucose uptake and the liver converts glycogen to glucose
blood pressure
- ***ADH- antidiuretic hormone which is a major player in blood pressure.
- low blood pressure- low blood volume- low amount of H2O——osmoreceptors in hypothalamus detect— impulses sent to post pit to release ADH—- collecting duct of nephron (kidney) reabsorbs water and excretes K ions.
- high blood pressure, high blood volume, large amount of H2O—- osmoreceptors in hypothalamus detect—- inhibit the release of ADH from the post pit—- collecting duct in nephron no longer permeable to water and there is no reabsorption, excess water excreted
oxytocin
*** positive feedback loops
uterine stretching— receptors send impulses to post pit to release oxytocin—uterine smooth muscle contractions + feedback loop and this PROMOTES uterine stretching for child birth
infant feeding– post pit releases oxytocin- let down (release )of breastmilk also positive feedback loop
sodium in blood
low sodium concentration in blood—– kidney releases and enzyme known as renin— angiotensin 1 is converted by the enzyme renin to angiotensin 2(vasoconstrictor) —- angio tennis 2 causes the adrenal cortex to release aldosterone–nephron collecting duct reabsorbs sodium and secretes potassium
high sodium concentration in the blood— kidney stops secreting renin—- angiotensin 1 does not get converted into angiotensin 2— aldosterone is not released from the adrenal cortex– nephron collecting duct allows sodium to diffuse out of the blood and potassium ions are kept
*** high sodium concentration in blood means that the solute concentration is high, and therefore the blood has a large amount of osmotic pressure which is the ability to draw water in. that is exactly what happens, the blood volume goes up due to the water being pulled in and with a high blood volume comes high blood pressure.
but… blood pressure is always known for ADH , use aldosterone when regarding sodium.
thyroxine
low levels of thyroxine—-chemoreceptors in hypothalamus detect and secrete TSHRF-thyroid stimulating hormone receptor factor—–ant pit releases TSH-Thyroid stimulating hormone—– TSH stimulates thyroid in releasing thyroxine
tropic hormones-causes another gland to release their hormone– tshrf also known as trh(thyroid releasing hormone)
adrenalin
stress— sympathetic nervous system detects (emergency times) send impulses to the medulla of adrenal gland—–adrenal gland releases epinephrine —- fight/ flight increase heart rate, increase blood pressure blood volume and decrease in digestion
describe hypothalamus structure
-ADH and oxytocin made in hypothalamus and stored in post pit.
posterior pituitary -extension of hypothalamus and contains neurosecretory cells that extend from hypothalamus to the end of the post pit. the anterior pituitary is on the back and it is known as the master gland