Behavioural addiction and the brain Flashcards
Empirical support for addiction hierarchy
combined substance addictions among treatment seeking gamblers and also potential other behavioural addictions
Strict comparisons between gambling and loot boxes wrong
50% of gamblers aim to win money while 0.9% of loot boxers do
Can’t apply same spending cap policy as house doesn’t try to maintain edge
Strict comparisons between substance and gambling wrong
substance gain reward
gambling potentially no reward
Gage
damage to ventromedial prefrontal cortex
no impact on memory or movement
became impulsive
vmpfc patients
Iowa gambling task
struggle to avoid bad decks
normal showed skin conductance responses early on when considering bad decks before verbal reports
links to somatic marker hypothesis
somatic marker hypothesis
emotional signals inform risky decisions
Support for somatic marker hypothesis
gamblers experiencing harm perform as badly as vmpfc - bad decks bigger wins so excite gamblers which might obstruct other emotional signal regulating behaviour
Soros- back ache pain signal something wrong in portfolio
emotions less helpful in poker
loose due to bad luck often go on tilt by making riskier decisions to get back even (vicious cycle)
Brain disease model
1- Cues and urges
2- Tolerance and higher use
3- Withdrawal
Cues and urges
dopamine releases upon reward- related cues
releases earlier when have expected reward compared to unexpected
Rewarding addictions cause dopamine releases - release prior to reward not after
Pavlovian conditioning
Gambling cues
elicit cravings
self-reported change in urges and activate the insula
front of football shirts and warning about dangers of gambling acts as major cues
Tolerance in substance-based
dopamine release falls with repeated engagements
other activities also see fall in dopamine release
so motivation to engage deeper with addiction
Tolerance in gambling
larger amounts of money to get same feeling of excitement
tolerance unique to gambling - make more exciting by better at longer odds
x- finical harms happen first so ay actually be unable to bet more
Self-regulation becomes harder
changes in pre-frontal cortex occur in addiction
harder to implement long-term plans
Withdrawal in gambling
negative reinforcement
urges and feelings drawing back in
Evidence for dopamine’s role
Parkinson’s disease
disease related to less dopamine
medications targeting lack of dopamine can lead to harmful gambling and impulse control disorders
Drug treatment for harmful gambling
opioid antagonists can be use to reverse opioid overdose and mange alcohol addiction
evidence that it can be effective for harmful gambling too
Critiques of the brain disease model
Brain changes in response to all kinds of things
most recover without treatment known as natural recovery and conflicts with chronic relapsing disease that changes structure and function of the brain
neuroplasticity- behavioural changes can in turn cause changes to the brain (causation both ways)
Benefit of the brain disease model for gamblers
tendency for self-blame - seen as illness instead to avoid blame
X- can also lead to people giving up hope inducing pessimism and helplessness
Other models needed to give gamblers hope
-neuroplasticity
-public health model (continuum and why some people more at risk)
- government action