Arthroplasty Flashcards
RFs for PJI in THA?
Patient factors: Obesity, DM, steroids, EtOH use, CLD, post-traumatic OA<div>Surgical factors: prolonged OR time, Wound drainage, hematoma, revision surgery</div>
MSIS Criteria for PJI?
Major (need 1)<div>-sinus tract communicating with joint</div><div>-pathogen isolated by culture from 2 separate tissue/fluid samples</div><div><br></br></div><div>Minor (need 4/6)</div><div>-serum: ESR>30 or CRP>10</div><div>-aspirate</div><div> -purulence on aspirate</div><div> -WBC>1,100cells/uL knees, >3k hips</div><div> -Neuts >64% knees, 80% hips</div><div> ->5 Neuts/HPF X5</div><div> -isolation of organism in 1 culture</div><div><br></br></div>
X-ray Findings of FAI?
CAM<div>-pistol grip</div><div>-Head neck offset <8mm</div><div>-Alpha angle>55-60 deg</div><div><br></br></div><div>Pincer</div><div>-Overcoverage: CEA>40, tonnis<0, downsloping sourcil, coxa profunda, acetabulum protrusio</div><div>-Acetab retroversion: cross-over sign, ischial spine sign, PW sign</div>
Coxa profunda vs Acetab Protrusion
Coxa profunda: deep socket - acetabular fossa medial to ilioischial line<div><br></br></div><div>Acetab Protrusion: deep head - femoral head medial to ilioischial line</div>
3rd gen cement techinque?
porosity reduction (<b>vacuum assisted - but not necessary</b>)<div>cement pressurization</div><div>pulsatile lavage for canal prep</div><div>cement plug</div><div>retrograde filling</div>
Advantages of HXPE vs UHMWPE
<b>decreased wear</b><div>decreased fracture</div><div>decreased oxidization</div><div><br></br></div><div><b>increased cost</b></div>
THA Modes of wear?
Mode 1 – primary bearing surface to primary bearing surface (1x1) - <b><u>most common</u></b><div>Mode 2 – primary x secondary (2x1=2)</div><div>Mode 3 – 3rd body</div><div>Mode 4 – Wear between 2 non bearing surfaces, ie. 2 secondary surfaces (such as backside wear or neck socket impingement) (secondary surface x secondary surface – 2x2=4)<br></br></div>
Properties of PMMA
<div><b>strongest in compression (not tension)</b></div>
exothermic to 75 deg<div>does not have adhesive properties (its a grout)</div><div>porosity reduction increases strength by 15%</div>
Tantalum properties?
“-Young’s modulus approximates that of bone compared to other metals<div>- more ingrowth pores</div><div>-greater friction and bone-metal interface</div><div>-<b>similar biocompatibility to other metals</b></div>”
Advantages of high off set stem?
-improved ROM<div>-improved stability</div><div>-improved joint reactive forces-> decrease wear/loosening</div><div>-decrease GT impingement</div><div>-improved abductor tension -> decr tren gait</div>
Complications for mal-positioned cup
Dislocation, instability<div>Increased wear/osteolysis</div><div>Aseptic loosening</div><div>Impingement</div><div>LLD</div><div>Decreased ROM</div>
RFs for hip AVN
Patient factors<div>-SLE, post-transplant</div><div>-hem disorder, (<b>not hemophilia)</b></div><div>-radiation tx</div><div>-dysbarism</div><div>Drugs: EtOH, Steroids</div><div>Trauma: fracture, dislocation, surgery</div>
Treatment options for hip AVN?
Non-op: ?bisphosphonates<div>Core decompression</div><div>Core decompression + augment (fib, tantalum rod)</div><div>Vascularized FG</div><div>Rotational osteotomy</div><div>THA</div>
Technical Considerations for Crowe 4 hip THA?
Acetabulum: anteversion, identifying and restoring true hip centre, anterosuperior bone deficiency<div><br></br></div><div>Femur: increased anteversion, valgus neck shaft, small diameter canal</div><div>-Options: Wagner cone, SROM, cemented polished</div><div><br></br></div><div>Soft-tissue: tensioning of sciatic nerve (subtroch shortening), deficient contracted abductors</div>
Modular neck in THA leads to?
Increased corrosion
RFs for HO post THA?
Male > <b>Female</b><div>Pagets</div><div>Ank Spon</div><div>Cementless implants</div><div><br></br></div><div>OA: post-traumatic, hypertrophic</div><div>DISH</div><div>Extended iliofem>kocher>ilioinguinal</div>
RFs for HO (in general)?
“Arthropathies: DISH, Ank Spon, hypertrophic OA<div>Others: Paget’s<br></br><div>Surgeries: THA, TKA</div><div>Approach: iliofemoral</div><div>Trauma: polytrauma, SCI (20%), TBI (11%), burn</div><div><br></br></div></div>”
THA and hemophilia?
“<b>infection rate higher than normal pop’n</b><div>coxa valga</div><div>should replace factors to 100% 2 hrs pre-op</div><div>HO decreased</div>”
RFs for Fem Neck # with BHR
Patient: BMI, female, low BMD, inflamm arthropathy<div><br></br></div><div>Surgical:</div><div>varus implant <130 deg</div><div>femoral neck notching</div><div>femoral neck cysts</div><div>improper implant seading</div><div><br></br></div>
Indications/Contraind for BHR?
Indications: male, <65, active, large fem head<div><br></br></div><div>Contra-indications</div><div>-Absoulte: CKD, women of childbearing age</div><div>-Relative: female, >65 years, low demand, Obese, AVN, Dysplasia, LLD</div>
Poor Px factors for PAO in Adult Dysplasia?
Patient: advanced age, morbid obesity<div>X-ray factors</div><div>-Tonnis Grade 2/3 OA (sclerosis/cysts)</div><div>-CEA<0</div><div>-Os acetabuli</div><div>-incongruent van rosen (Ab IR) view</div>
Signs of loosening femoral stem?
<div><ul> <li>Cemented (Harris Criteria)</li> <ul> <li>Definite --> subsidence, fractured stem, fractured cement mantle</li> <li>Probably --> continuous radiolucent line at interface, >2mm space at any point</li> <li>Possible --> any radiolucent line that is >50% of interface</li> </ul> </ul> <ul> <li>Uncemented:</li> <ul> <li>Subsidence, reactive line around implant, pedestal formation</li> <li>**stem geometry/Corail</li> </ul></ul></div>
<div><br></br></div>
<div>Cemented (Harris Criteria)<br></br></div>
<div>-subsidence</div>
<div>-fractured stem</div>
<div>-fractured cement mantle</div>
<div>-continuous radiolucent line</div>
<div><br></br></div>
<div>Uncemented</div>
<div>-subsidence</div>
<div>-reactive line around implant</div>
<div>-pedestal formation</div>
Components of Paprosky Acetabular Bone Loss?
location of hip centre (ie migrated)<div>ischial osteolysis</div><div>integrity of Kohlers (ilioschial) line</div><div>teardrop destruction</div>
Paprosky components of Femoral Bone Loss?
Proximal Metaphysis - intact or basent<div>Diaphysis -</div><div>-intact: extensively poorous coated</div><div>->4cm vs <4cm isthmus: modular <u>tapered</u> stem</div><div>-absent: allograft prosthetic composite</div>
Components of Vancouver classification
Fracture location, implant stability, bone stock<div><br></br></div><div>A - GT/LT</div><div>B1 - stem, stable -> ORIF</div><div>B2 - unstable, good bone stock -> long stem revision</div><div>B3 - unstable, poor stock –> APC, PFR</div><div>C - below -> ORIF</div>
RFs for instability post THA
Patient: Female, neuro impairment (dementia, cant comply with restrictrions, NM disease), no pre-op stiffness (AVN, fracture), high ASA, obesity<div><br></br></div><div>Surgical</div><div>-Approach: PL</div><div>-Soft tissue: Abductor integrity/tensioning</div><div>-Component position (Acetab 40 abd, 15 ante)</div><div>-Components Used:smaller head</div>
Surgical tx options for recurrent THA instability
-component positioning!<div>-increase offset, trochanteric advancement</div><div>-constrained liner</div><div>-bipolar prosthesis</div><div>-resection arthroplasty</div>
Releases to balance a varus knee?
medial osteophytes<div>joing capsule</div><div>POL</div><div>sMCL</div><div>semiM</div><div>pes anserine</div><div>PCL</div><div>MHG</div><div>deep MCL</div><div><br></br></div><div>lateral tightening</div>
Releases to balance Valgus knee?
Osteophytes<div>Posterolateral capsule</div><div>Popliteus (flex)</div><div>ITB (extension)</div><div>LHG</div><div>PCL</div><div>LCL</div><div><br></br></div>
Causes of post-op TKA stiffness?
“Patient factors<div>-poor pre-op ROM</div><div>-preop baja</div><div>-poor compliance with rehab</div><div>-poor pain control</div><div>-HO</div><div>-infection</div><div>-hematoma</div><div><br></br></div><div>Surgical factors</div><div>-component malalignment, malposition</div><div>-gap imbalance</div><div>-PF overstuffing</div><div>-cement extravasation posteriorly</div><div>-joint line elevation (patella baja)</div><div><br></br></div><div>Factors that don’t affect ROM</div><div>-obesity</div><div>-previous OR</div><div>-keloid scar</div><div>-age</div><div>-sex</div><div>-bilateral TKA</div><div>-multiple joint involvement</div><div><br></br></div>”
Cyclops lesion?
anterior arthrofibrosis -><b>block to extension (not flexion)</b><div><b><br></br></b></div>
hemophiliac TKA postop complaint?
stiffness, <b>not laxity</b>
Methods to improve flexion gap during TKA?
<div>Bony: downsize femoral component, increase tibial slope</div>
resect PCL<div>posterior capsule</div><div>gastrocs</div><div>posterior corners (popliteus)</div><div>sMCL</div><div>pes anserine</div>
Methods to prevent PF maltracking?
ER and Lateralization of tibial and femoral components<div>Medialization of Patellar component</div>